Mirit Ben Nun: Shortness of breath

'Shortness of breath' is not only a sign of physical weakness, it is a metaphor for a mental state of strong desire that knows no repletion; more and more, an unbearable glut, without repose. Mirit Ben Nun's type of work on the other hand requires an abundance of patience. This is a Sisyphean work (requiring hard labor) of marking lines and dots, filling every empty millimeter with brilliant blots. Therefore we are facing a paradox or a logical conflict. A patient and effortful work that stems from an urgent need to cover and fill, to adorn and coat. Her craft of layering reaches a state of a continuous ceremonial ritual.

This ritual digests every object into itself - useful or discarded -- available and ordinary or rare and exceptional -- they submit and devote to the overlay work. Mirit BN gathers scrap off the streets -- cardboard rolls of fabric, assortments of wooden boards and pieces, plates and planks -- and constructs a new link, her own syntax, which she alone is fully responsible for. The new combination -- a type of a sculptural construction -- goes through a process of patching by the act of painting.

In fact Mirit regards her three dimensional objects as a platform for painting, with a uniform continuity, even if it has obstacles, mounds and valleys. These objects beg her to paint, to lay down colors, to set in motion an intricate weave of abstract patterns that at times finds itself wandering the contours of human images and sometimes -- not. In those cases what is left is the monotonous activity of running the patterns, inch by inch, till their absolute coverage, till a short and passing instant of respite and than on again to a new onset.

Next to this assembly of garbage and it's recycling into 'painted sculptures' Mirit offers a surprising reunion between her illustrated objects and so called cheap African sculpture; popular artifacts or articles that are classified in the standard culture as 'primitive'.

This combination emphasizes the difference between her individualistic performance and the collective creation which is translated into cultural clichés. The wood carved image creates a moment of peace within the crowded bustle; an introverted image, without repetitiveness and reverberation. This meeting of strangers testifies that Mirit' work could not be labeled under the ´outsiders art´ category. She is a one woman school who is compelled to do the art work she picked out to perform. Therefore she isn't creating ´an image´ such as the carved wooden statues, but she produces breathless ´emotional jam' whose highest values are color, motion, beauty and plenitude. May it never lack, neither diluted, nor dull for even an instant

 

Tali Tamir

August 2010

A slight difference to a shot I took previously. Bit more focus on his face

Capture from SW Fourth Avenue in the Pioneer District of Portland, Oregon.

'The Smiths' formed six years after the start of the British punk rock scene, and perhaps projected into their way-of-being three elements from this cultural dynamic: a sense of make-do or DIY, a sense of commentating from outside of society (unemployment culture included), and a sense of opinion generated from polemical example.

 

Punk rock is today perhaps famous for its blanket nihilisms: “Anarchy in the UK”, “White riot”, “No future” - all with varied forms of the Vivian Westwood dress-sense. Aside this ultra-vivid self-created stigma, the late 70s musical/cultural movement of UK Punk had created lyrical and musical vignettes that stayed on record players and transferred to C60 chrome mix-tapes: “Easy germ free adolescence” ('X-Ray Spex' 78) plotted a closed 'existential' in the life of an individual; 'Lost in a supermarket' ('The Clash' 79) was another vignette, but this time describing a poetic angle into a modern life that many could relate to, and, as a third example - “Love comes in spurts” ('The Voidoids' 76) - featuring a lyric that uses 'childish' shock word-play and active double-meanings to register and parody existing lyrical convention, whilst describing certain truisms. It can be said that depictions of highly specific sentiments; unusual but collective views on the modern world and the shock use of words and their dynamic meanings became the terrain of the 'post punk', 'DIY' Manchester band - 'The Smiths'.

 

With the lyrical whit of their singer Morrissey, 'The Smiths' kept a punkist sense of polemical charge (“The Queen is Dead”; “Hang the DJ” and “Meat is Murder”) whilst systematically developing individual and social themes. If musical skills were often missing (or denied) during UK Punk, then the Post Punk years saw an overt return of musical virtuosity (hand in hand with groups higher on idea and form than musical technicity). One nest of guitar skills hovered around the New York City band 'The Voidoids' with Tom Verlain, Robert Quine and Richard Lloyd all important Post Punk guitar stylists (even if US 'Post Punk' arrived before UK Punk - for example 'Pere Ubu' 1975). Around 1980 in Manchester England, a man who could match 'Nick Drake' for reserve produced a low-key, but much listened to record titled “The return of the Durutti Column” (a Spanish civil war reference in the name and title) www.youtube.com/watch?v=pddox1SYHko. 'The Durutti Column's Vini Reilly's meticulous detailing of guitar melody was perhaps matched across the city of Manchester by the extraordinary guitar style of Johnny Marr – the principle co-songwriter to 'The Smiths', whose style became clearly visible as early as 'The Smith's' second single. Marr's sense of shimmering detailing is also clearly apparent in the example of this moving lens test.

 

Fireworks are of so many colours and effects, and the post punk DIY musical alternatives were varied. Despite their differences, there are a few bands that help to put 'The Smiths' into an early context. 'Vic Godard's Subway Sect' had perhaps started out as a British version of 'The Voidoids' ('Double negative' 1978 www.youtube.com/watch?v=8fsYYG_Qf0Q) and had evolved by 1981 into melodic anti pop www.youtube.com/watch?v=3cgzwQBtFaA an approach perhaps akin to the relationship between 'New Wave cinema' and traditional cinema. Again from 1981, the group 'Orange Juice' were inhabiting melody and lyric without the desire to provide 'expected' popularist results: www.youtube.com/watch?v=RmpNSpzx2wI. 'The Smiths' projected aside many groups, Dandyisms, deconstructions and observations and, with some of the lyrical landscapes of Morrissey, were one of the groups that held on longest to the polemical side of punk rock. With the guitar of Johnny Marr, they were also one of the groups that pushed furthest from initial DIY ethics into artistic, holistic and highly developed forms. By the release of 'The Queen is Dead' in 1986 they had refined their idiosyncrasies to such an extent that they had produced a record that could be compared against the very “classic” albums they were initially reflecting against.

 

Prolific songwriters, 'The Smiths' took subject to swathes of British youth, and were another input of inspiration for a new generation of musicians. Postcard, Pop Aural and Rough Trade layered under Creation, Domino and Sarah, and new bands evolved without a close proximity to Punk rock. Contemporary journalists (including ones associated with Manchester's Guardian), muse, relay and decree that it is now time to stop listening to the work of Morressey. The Smiths split in 1987, and in the decades since, Morrissey has remained loyal to his polemical approach to social subjects. I certainly never wanted to hang a DJ, but played the song. One of the tracks that we should apparently be boycotting is a recently released duet with Thelma Houston: www.youtube.com/watch?v=2cB93OUF_sA

 

The track “Some girls are bigger than others' comes from the 1986 record and is a construction with the most minimal idea of verse, and thus arrives almost straight into the chorus, with a slightly detached and minimal refrain. The lyric takes a line that can be associated with both male machismo and the observational naivety of a small child: playing the idea straight as the thought of a young male adult faced by the enormity of mankind's diversity. By referring to the ice age, the lyric also points out that these differences come from the depths of the human race and are not a modern celebrity surface.

 

"From the ice-age to the dole-age, there is but one concern. I have just discovered; some girls are bigger than others... "

 

For the images in the lens test I replaced the 'girls' of the song with the animate 'bodies' of 'mother earth', as reflected by her waterfalls. The shots involve a variety of lenses and locations either side of the Pyrénées.

 

AJM 13.03.20

 

Press play and then 'L' and even f11. Escape and f11 a second time to return.

  

Such a huge contrast between the G and the I.

Dalbeattie , Dumfries & Galloway

My [Wreck This Journal] - DRIP SOMETHING HERE. CLOSE THE BOOK TO MAKE A PRINT -

Spot the difference!

What a difference a day makes; last Friday I was at a press call for the Moscow State Circus and the on Saturday I photographed a massive Orange Lodge march through Johnstone involving over 40 bands (it took at least 40 minutes to pass my viewpoint). While most people probably think that such marches are only held in Northern Ireland they are very much part of life here in Scotland as well as in parts of England. The weather was kind for once, if not a little too sunny given the length of their march, marching all round the town while playing their instruments and carrying their various banners is no mean feat!!

SKILLZ BRUV

 

"Have you ever had a dream, that you were so sure was real? What if you were unable to wake from that dream? How would you know the difference between the dream world and the real world?"

#jonboam #drawing #illustration

Coronavirus disease 2019 (COVID-19) is an infectious disease caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2).[8] The disease was first identified in December 2019 in Wuhan, the capital of China's Hubei province, and has since spread globally, resulting in the ongoing 2019–20 coronavirus pandemic.[9][10] As of 26 April 2020, more than 2.89 million cases have been reported across 185 countries and territories, resulting in more than 203,000 deaths. More than 822,000 people have recovered.[7]

 

Common symptoms include fever, cough, fatigue, shortness of breath and loss of smell.[5][11][12] While the majority of cases result in mild symptoms, some progress to viral pneumonia, multi-organ failure, or cytokine storm.[13][9][14] More concerning symptoms include difficulty breathing, persistent chest pain, confusion, difficulty waking, and bluish skin.[5] The time from exposure to onset of symptoms is typically around five days but may range from two to fourteen days.[5][15]

 

The virus is primarily spread between people during close contact,[a] often via small droplets produced by coughing,[b] sneezing, or talking.[6][16][18] The droplets usually fall to the ground or onto surfaces rather than remaining in the air over long distances.[6][19][20] People may also become infected by touching a contaminated surface and then touching their face.[6][16] In experimental settings, the virus may survive on surfaces for up to 72 hours.[21][22][23] It is most contagious during the first three days after the onset of symptoms, although spread may be possible before symptoms appear and in later stages of the disease.[24] The standard method of diagnosis is by real-time reverse transcription polymerase chain reaction (rRT-PCR) from a nasopharyngeal swab.[25] Chest CT imaging may also be helpful for diagnosis in individuals where there is a high suspicion of infection based on symptoms and risk factors; however, guidelines do not recommend using it for routine screening.[26][27]

 

Recommended measures to prevent infection include frequent hand washing, maintaining physical distance from others (especially from those with symptoms), covering coughs, and keeping unwashed hands away from the face.[28][29] In addition, the use of a face covering is recommended for those who suspect they have the virus and their caregivers.[30][31] Recommendations for face covering use by the general public vary, with some authorities recommending against their use, some recommending their use, and others requiring their use.[32][31][33] Currently, there is not enough evidence for or against the use of masks (medical or other) in healthy individuals in the wider community.[6] Also masks purchased by the public may impact availability for health care providers.

 

Currently, there is no vaccine or specific antiviral treatment for COVID-19.[6] Management involves the treatment of symptoms, supportive care, isolation, and experimental measures.[34] The World Health Organization (WHO) declared the 2019–20 coronavirus outbreak a Public Health Emergency of International Concern (PHEIC)[35][36] on 30 January 2020 and a pandemic on 11 March 2020.[10] Local transmission of the disease has occurred in most countries across all six WHO regions.[37]

 

File:En.Wikipedia-VideoWiki-Coronavirus disease 2019.webm

Video summary (script)

 

Contents

1Signs and symptoms

2Cause

2.1Transmission

2.2Virology

3Pathophysiology

3.1Immunopathology

4Diagnosis

4.1Pathology

5Prevention

6Management

6.1Medications

6.2Protective equipment

6.3Mechanical ventilation

6.4Acute respiratory distress syndrome

6.5Experimental treatment

6.6Information technology

6.7Psychological support

7Prognosis

7.1Reinfection

8History

9Epidemiology

9.1Infection fatality rate

9.2Sex differences

10Society and culture

10.1Name

10.2Misinformation

10.3Protests

11Other animals

12Research

12.1Vaccine

12.2Medications

12.3Anti-cytokine storm

12.4Passive antibodies

13See also

14Notes

15References

16External links

16.1Health agencies

16.2Directories

16.3Medical journals

Signs and symptoms

Symptom[4]Range

Fever83–99%

Cough59–82%

Loss of Appetite40–84%

Fatigue44–70%

Shortness of breath31–40%

Coughing up sputum28–33%

Loss of smell15[38] to 30%[12][39]

Muscle aches and pains11–35%

Fever is the most common symptom, although some older people and those with other health problems experience fever later in the disease.[4][40] In one study, 44% of people had fever when they presented to the hospital, while 89% went on to develop fever at some point during their hospitalization.[4][41]

 

Other common symptoms include cough, loss of appetite, fatigue, shortness of breath, sputum production, and muscle and joint pains.[4][5][42][43] Symptoms such as nausea, vomiting and diarrhoea have been observed in varying percentages.[44][45][46] Less common symptoms include sneezing, runny nose, or sore throat.[47]

 

More serious symptoms include difficulty breathing, persistent chest pain or pressure, confusion, difficulty waking, and bluish face or lips. Immediate medical attention is advised if these symptoms are present.[5][48]

 

In some, the disease may progress to pneumonia, multi-organ failure, and death.[9][14] In those who develop severe symptoms, time from symptom onset to needing mechanical ventilation is typically eight days.[4] Some cases in China initially presented with only chest tightness and palpitations.[49]

 

Loss of smell was identified as a common symptom of COVID‑19 in March 2020,[12][39] although perhaps not as common as initially reported.[38] A decreased sense of smell and/or disturbances in taste have also been reported.[50] Estimates for loss of smell range from 15%[38] to 30%.[12][39]

 

As is common with infections, there is a delay between the moment a person is first infected and the time he or she develops symptoms. This is called the incubation period. The incubation period for COVID‑19 is typically five to six days but may range from two to 14 days,[51][52] although 97.5% of people who develop symptoms will do so within 11.5 days of infection.[53]

 

A minority of cases do not develop noticeable symptoms at any point in time.[54][55] These asymptomatic carriers tend not to get tested, and their role in transmission is not yet fully known.[56][57] However, preliminary evidence suggests they may contribute to the spread of the disease.[58][59] In March 2020, the Korea Centers for Disease Control and Prevention (KCDC) reported that 20% of confirmed cases remained asymptomatic during their hospital stay.[59][60]

 

A number of neurological symptoms has been reported including seizures, stroke, encephalitis and Guillain-Barre syndrome.[61] Cardiovascular related complications may include heart failure, irregular electrical activity, blood clots, and heart inflammation.[62]

 

Cause

See also: Severe acute respiratory syndrome coronavirus 2

Transmission

Cough/sneeze droplets visualised in dark background using Tyndall scattering

Respiratory droplets produced when a man is sneezing visualised using Tyndall scattering

File:COVID19 in numbers- R0, the case fatality rate and why we need to flatten the curve.webm

A video discussing the basic reproduction number and case fatality rate in the context of the pandemic

Some details about how the disease is spread are still being determined.[16][18] The WHO and the U.S. Centers for Disease Control and Prevention (CDC) say it is primarily spread during close contact and by small droplets produced when people cough, sneeze or talk;[6][16] with close contact being within approximately 1–2 m (3–7 ft).[6][63] Both sputum and saliva can carry large viral loads.[64] Loud talking releases more droplets than normal talking.[65] A study in Singapore found that an uncovered cough can lead to droplets travelling up to 4.5 metres (15 feet).[66] An article published in March 2020 argued that advice on droplet distance might be based on 1930s research which ignored the effects of warm moist exhaled air surrounding the droplets and that an uncovered cough or sneeze can travel up to 8.2 metres (27 feet).[17]

  

Respiratory droplets may also be produced while breathing out, including when talking. Though the virus is not generally airborne,[6][67] the National Academy of Sciences has suggested that bioaerosol transmission may be possible.[68] In one study cited, air collectors positioned in the hallway outside of people's rooms yielded samples positive for viral RNA but finding infectious virus has proven elusive.[68] The droplets can land in the mouths or noses of people who are nearby or possibly be inhaled into the lungs.[16] Some medical procedures such as intubation and cardiopulmonary resuscitation (CPR) may cause respiratory secretions to be aerosolised and thus result in an airborne spread.[67] Initial studies suggested a doubling time of the number of infected persons of 6–7 days and a basic reproduction number (R0 ) of 2.2–2.7, but a study published on April 7, 2020, calculated a much higher median R0 value of 5.7 in Wuhan.[69]

 

It may also spread when one touches a contaminated surface, known as fomite transmission, and then touches one's eyes, nose or mouth.[6] While there are concerns it may spread via faeces, this risk is believed to be low.[6][16]

 

The virus is most contagious when people are symptomatic; though spread is may be possible before symptoms emerge and from those who never develop symptoms.[6][70] A portion of individuals with coronavirus lack symptoms.[71] The European Centre for Disease Prevention and Control (ECDC) says while it is not entirely clear how easily the disease spreads, one person generally infects two or three others.[18]

 

The virus survives for hours to days on surfaces.[6][18] Specifically, the virus was found to be detectable for one day on cardboard, for up to three days on plastic (polypropylene) and stainless steel (AISI 304), and for up to four hours on 99% copper.[21][23] This, however, varies depending on the humidity and temperature.[72][73] Surfaces may be decontaminated with many solutions (with one minute of exposure to the product achieving a 4 or more log reduction (99.99% reduction)), including 78–95% ethanol (alcohol used in spirits), 70–100% 2-propanol (isopropyl alcohol), the combination of 45% 2-propanol with 30% 1-propanol, 0.21% sodium hypochlorite (bleach), 0.5% hydrogen peroxide, or 0.23–7.5% povidone-iodine. Soap and detergent are also effective if correctly used; soap products degrade the virus' fatty protective layer, deactivating it, as well as freeing them from the skin and other surfaces.[74] Other solutions, such as benzalkonium chloride and chlorhexidine gluconate (a surgical disinfectant), are less effective.[75]

 

In a Hong Kong study, saliva samples were taken a median of two days after the start of hospitalization. In five of six patients, the first sample showed the highest viral load, and the sixth patient showed the highest viral load on the second day tested.[64]

 

Virology

Main article: Severe acute respiratory syndrome coronavirus 2

 

Illustration of SARSr-CoV virion

Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is a novel severe acute respiratory syndrome coronavirus, first isolated from three people with pneumonia connected to the cluster of acute respiratory illness cases in Wuhan.[76] All features of the novel SARS-CoV-2 virus occur in related coronaviruses in nature.[77] Outside the human body, the virus is killed by household soap, which bursts its protective bubble.[26]

 

SARS-CoV-2 is closely related to the original SARS-CoV.[78] It is thought to have a zoonotic origin. Genetic analysis has revealed that the coronavirus genetically clusters with the genus Betacoronavirus, in subgenus Sarbecovirus (lineage B) together with two bat-derived strains. It is 96% identical at the whole genome level to other bat coronavirus samples (BatCov RaTG13).[47] In February 2020, Chinese researchers found that there is only one amino acid difference in the binding domain of the S protein between the coronaviruses from pangolins and those from humans; however, whole-genome comparison to date found that at most 92% of genetic material was shared between pangolin coronavirus and SARS-CoV-2, which is insufficient to prove pangolins to be the intermediate host.[79]

 

Pathophysiology

The lungs are the organs most affected by COVID‑19 because the virus accesses host cells via the enzyme angiotensin-converting enzyme 2 (ACE2), which is most abundant in type II alveolar cells of the lungs. The virus uses a special surface glycoprotein called a "spike" (peplomer) to connect to ACE2 and enter the host cell.[80] The density of ACE2 in each tissue correlates with the severity of the disease in that tissue and some have suggested that decreasing ACE2 activity might be protective,[81][82] though another view is that increasing ACE2 using angiotensin II receptor blocker medications could be protective and these hypotheses need to be tested.[83] As the alveolar disease progresses, respiratory failure might develop and death may follow.[82]

 

The virus also affects gastrointestinal organs as ACE2 is abundantly expressed in the glandular cells of gastric, duodenal and rectal epithelium[84] as well as endothelial cells and enterocytes of the small intestine.[85]

 

ACE2 is present in the brain, and there is growing evidence of neurological manifestations in people with COVID‑19. It is not certain if the virus can directly infect the brain by crossing the barriers that separate the circulation of the brain and the general circulation. Other coronaviruses are able to infect the brain via a synaptic route to the respiratory centre in the medulla, through mechanoreceptors like pulmonary stretch receptors and chemoreceptors (primarily central chemoreceptors) within the lungs.[medical citation needed] It is possible that dysfunction within the respiratory centre further worsens the ARDS seen in COVID‑19 patients. Common neurological presentations include a loss of smell, headaches, nausea, and vomiting. Encephalopathy has been noted to occur in some patients (and confirmed with imaging), with some reports of detection of the virus after cerebrospinal fluid assays although the presence of oligoclonal bands seems to be a common denominator in these patients.[86]

 

The virus can cause acute myocardial injury and chronic damage to the cardiovascular system.[87] An acute cardiac injury was found in 12% of infected people admitted to the hospital in Wuhan, China,[88] and is more frequent in severe disease.[89] Rates of cardiovascular symptoms are high, owing to the systemic inflammatory response and immune system disorders during disease progression, but acute myocardial injuries may also be related to ACE2 receptors in the heart.[87] ACE2 receptors are highly expressed in the heart and are involved in heart function.[87][90] A high incidence of thrombosis (31%) and venous thromboembolism (25%) have been found in ICU patients with COVID‑19 infections and may be related to poor prognosis.[91][92] Blood vessel dysfunction and clot formation (as suggested by high D-dimer levels) are thought to play a significant role in mortality, incidences of clots leading to pulmonary embolisms, and ischaemic events within the brain have been noted as complications leading to death in patients infected with SARS-CoV-2. Infection appears to set off a chain of vasoconstrictive responses within the body, constriction of blood vessels within the pulmonary circulation has also been posited as a mechanism in which oxygenation decreases alongside with the presentation of viral pneumonia.[93]

 

Another common cause of death is complications related to the kidneys[93]—SARS-CoV-2 directly infects kidney cells, as confirmed in post-mortem studies. Acute kidney injury is a common complication and cause of death; this is more significant in patients with already compromised kidney function, especially in people with pre-existing chronic conditions such as hypertension and diabetes which specifically cause nephropathy in the long run.[94]

 

Autopsies of people who died of COVID‑19 have found diffuse alveolar damage (DAD), and lymphocyte-containing inflammatory infiltrates within the lung.[95]

 

Immunopathology

Although SARS-COV-2 has a tropism for ACE2-expressing epithelial cells of the respiratory tract, patients with severe COVID‑19 have symptoms of systemic hyperinflammation. Clinical laboratory findings of elevated IL-2, IL-7, IL-6, granulocyte-macrophage colony-stimulating factor (GM-CSF), interferon-γ inducible protein 10 (IP-10), monocyte chemoattractant protein 1 (MCP-1), macrophage inflammatory protein 1-α (MIP-1α), and tumour necrosis factor-α (TNF-α) indicative of cytokine release syndrome (CRS) suggest an underlying immunopathology.[96]

 

Additionally, people with COVID‑19 and acute respiratory distress syndrome (ARDS) have classical serum biomarkers of CRS, including elevated C-reactive protein (CRP), lactate dehydrogenase (LDH), D-dimer, and ferritin.[97]

 

Systemic inflammation results in vasodilation, allowing inflammatory lymphocytic and monocytic infiltration of the lung and the heart. In particular, pathogenic GM-CSF-secreting T-cells were shown to correlate with the recruitment of inflammatory IL-6-secreting monocytes and severe lung pathology in COVID‑19 patients.[98] Lymphocytic infiltrates have also been reported at autopsy.[95]

 

Diagnosis

Main article: COVID-19 testing

 

Demonstration of a nasopharyngeal swab for COVID-19 testing

 

CDC rRT-PCR test kit for COVID-19[99]

The WHO has published several testing protocols for the disease.[100] The standard method of testing is real-time reverse transcription polymerase chain reaction (rRT-PCR).[101] The test is typically done on respiratory samples obtained by a nasopharyngeal swab; however, a nasal swab or sputum sample may also be used.[25][102] Results are generally available within a few hours to two days.[103][104] Blood tests can be used, but these require two blood samples taken two weeks apart, and the results have little immediate value.[105] Chinese scientists were able to isolate a strain of the coronavirus and publish the genetic sequence so laboratories across the world could independently develop polymerase chain reaction (PCR) tests to detect infection by the virus.[9][106][107] As of 4 April 2020, antibody tests (which may detect active infections and whether a person had been infected in the past) were in development, but not yet widely used.[108][109][110] The Chinese experience with testing has shown the accuracy is only 60 to 70%.[111] The FDA in the United States approved the first point-of-care test on 21 March 2020 for use at the end of that month.[112]

 

Diagnostic guidelines released by Zhongnan Hospital of Wuhan University suggested methods for detecting infections based upon clinical features and epidemiological risk. These involved identifying people who had at least two of the following symptoms in addition to a history of travel to Wuhan or contact with other infected people: fever, imaging features of pneumonia, normal or reduced white blood cell count, or reduced lymphocyte count.[113]

 

A study asked hospitalised COVID‑19 patients to cough into a sterile container, thus producing a saliva sample, and detected the virus in eleven of twelve patients using RT-PCR. This technique has the potential of being quicker than a swab and involving less risk to health care workers (collection at home or in the car).[64]

 

Along with laboratory testing, chest CT scans may be helpful to diagnose COVID-19 in individuals with a high clinical suspicion of infection but are not recommended for routine screening.[26][27] Bilateral multilobar ground-glass opacities with a peripheral, asymmetric, and posterior distribution are common in early infection.[26] Subpleural dominance, crazy paving (lobular septal thickening with variable alveolar filling), and consolidation may appear as the disease progresses.[26][114]

 

In late 2019, WHO assigned the emergency ICD-10 disease codes U07.1 for deaths from lab-confirmed SARS-CoV-2 infection and U07.2 for deaths from clinically or epidemiologically diagnosed COVID‑19 without lab-confirmed SARS-CoV-2 infection.[115]

  

Typical CT imaging findings

 

CT imaging of rapid progression stage

Pathology

Few data are available about microscopic lesions and the pathophysiology of COVID‑19.[116][117] The main pathological findings at autopsy are:

 

Macroscopy: pleurisy, pericarditis, lung consolidation and pulmonary oedema

Four types of severity of viral pneumonia can be observed:

minor pneumonia: minor serous exudation, minor fibrin exudation

mild pneumonia: pulmonary oedema, pneumocyte hyperplasia, large atypical pneumocytes, interstitial inflammation with lymphocytic infiltration and multinucleated giant cell formation

severe pneumonia: diffuse alveolar damage (DAD) with diffuse alveolar exudates. DAD is the cause of acute respiratory distress syndrome (ARDS) and severe hypoxemia.

healing pneumonia: organisation of exudates in alveolar cavities and pulmonary interstitial fibrosis

plasmocytosis in BAL[118]

Blood: disseminated intravascular coagulation (DIC);[119] leukoerythroblastic reaction[120]

Liver: microvesicular steatosis

Prevention

See also: 2019–20 coronavirus pandemic § Prevention, flatten the curve, and workplace hazard controls for COVID-19

 

Progressively stronger mitigation efforts to reduce the number of active cases at any given time—known as "flattening the curve"—allows healthcare services to better manage the same volume of patients.[121][122][123] Likewise, progressively greater increases in healthcare capacity—called raising the line—such as by increasing bed count, personnel, and equipment, helps to meet increased demand.[124]

 

Mitigation attempts that are inadequate in strictness or duration—such as premature relaxation of distancing rules or stay-at-home orders—can allow a resurgence after the initial surge and mitigation.[122][125]

Preventive measures to reduce the chances of infection include staying at home, avoiding crowded places, keeping distance from others, washing hands with soap and water often and for at least 20 seconds, practising good respiratory hygiene, and avoiding touching the eyes, nose, or mouth with unwashed hands.[126][127][128] The CDC recommends covering the mouth and nose with a tissue when coughing or sneezing and recommends using the inside of the elbow if no tissue is available.[126] Proper hand hygiene after any cough or sneeze is encouraged.[126] The CDC has recommended the use of cloth face coverings in public settings where other social distancing measures are difficult to maintain, in part to limit transmission by asymptomatic individuals.[129] The U.S. National Institutes of Health guidelines do not recommend any medication for prevention of COVID‑19, before or after exposure to the SARS-CoV-2 virus, outside of the setting of a clinical trial.[130]

 

Social distancing strategies aim to reduce contact of infected persons with large groups by closing schools and workplaces, restricting travel, and cancelling large public gatherings.[131] Distancing guidelines also include that people stay at least 6 feet (1.8 m) apart.[132] There is no medication known to be effective at preventing COVID‑19.[133] After the implementation of social distancing and stay-at-home orders, many regions have been able to sustain an effective transmission rate ("Rt") of less than one, meaning the disease is in remission in those areas.[134]

 

As a vaccine is not expected until 2021 at the earliest,[135] a key part of managing COVID‑19 is trying to decrease the epidemic peak, known as "flattening the curve".[122] This is done by slowing the infection rate to decrease the risk of health services being overwhelmed, allowing for better treatment of current cases, and delaying additional cases until effective treatments or a vaccine become available.[122][125]

 

According to the WHO, the use of masks is recommended only if a person is coughing or sneezing or when one is taking care of someone with a suspected infection.[136] For the European Centre for Disease Prevention and Control (ECDC) face masks "... could be considered especially when visiting busy closed spaces ..." but "... only as a complementary measure ..."[137] Several countries have recommended that healthy individuals wear face masks or cloth face coverings (like scarves or bandanas) at least in certain public settings, including China,[138] Hong Kong,[139] Spain,[140] Italy (Lombardy region),[141] and the United States.[129]

 

Those diagnosed with COVID‑19 or who believe they may be infected are advised by the CDC to stay home except to get medical care, call ahead before visiting a healthcare provider, wear a face mask before entering the healthcare provider's office and when in any room or vehicle with another person, cover coughs and sneezes with a tissue, regularly wash hands with soap and water and avoid sharing personal household items.[30][142] The CDC also recommends that individuals wash hands often with soap and water for at least 20 seconds, especially after going to the toilet or when hands are visibly dirty, before eating and after blowing one's nose, coughing or sneezing. It further recommends using an alcohol-based hand sanitiser with at least 60% alcohol, but only when soap and water are not readily available.[126]

 

For areas where commercial hand sanitisers are not readily available, the WHO provides two formulations for local production. In these formulations, the antimicrobial activity arises from ethanol or isopropanol. Hydrogen peroxide is used to help eliminate bacterial spores in the alcohol; it is "not an active substance for hand antisepsis". Glycerol is added as a humectant.[143]

  

Prevention efforts are multiplicative, with effects far beyond that of a single spread. Each avoided case leads to more avoided cases down the line, which in turn can stop the outbreak in its tracks.

 

File:COVID19 W ENG.ogv

Handwashing instructions

Management

People are managed with supportive care, which may include fluid therapy, oxygen support, and supporting other affected vital organs.[144][145][146] The CDC recommends that those who suspect they carry the virus wear a simple face mask.[30] Extracorporeal membrane oxygenation (ECMO) has been used to address the issue of respiratory failure, but its benefits are still under consideration.[41][147] Personal hygiene and a healthy lifestyle and diet have been recommended to improve immunity.[148] Supportive treatments may be useful in those with mild symptoms at the early stage of infection.[149]

 

The WHO, the Chinese National Health Commission, and the United States' National Institutes of Health have published recommendations for taking care of people who are hospitalised with COVID‑19.[130][150][151] Intensivists and pulmonologists in the U.S. have compiled treatment recommendations from various agencies into a free resource, the IBCC.[152][153]

 

Medications

See also: Coronavirus disease 2019 § Research

As of April 2020, there is no specific treatment for COVID‑19.[6][133] Research is, however, ongoing. For symptoms, some medical professionals recommend paracetamol (acetaminophen) over ibuprofen for first-line use.[154][155][156] The WHO and NIH do not oppose the use of non-steroidal anti-inflammatory drugs (NSAIDs) such as ibuprofen for symptoms,[130][157] and the FDA says currently there is no evidence that NSAIDs worsen COVID‑19 symptoms.[158]

 

While theoretical concerns have been raised about ACE inhibitors and angiotensin receptor blockers, as of 19 March 2020, these are not sufficient to justify stopping these medications.[130][159][160][161] Steroids, such as methylprednisolone, are not recommended unless the disease is complicated by acute respiratory distress syndrome.[162][163]

 

Medications to prevent blood clotting have been suggested for treatment,[91] and anticoagulant therapy with low molecular weight heparin appears to be associated with better outcomes in severe COVID‐19 showing signs of coagulopathy (elevated D-dimer).[164]

 

Protective equipment

See also: COVID-19 related shortages

 

The CDC recommends four steps to putting on personal protective equipment (PPE).[165]

Precautions must be taken to minimise the risk of virus transmission, especially in healthcare settings when performing procedures that can generate aerosols, such as intubation or hand ventilation.[166] For healthcare professionals caring for people with COVID‑19, the CDC recommends placing the person in an Airborne Infection Isolation Room (AIIR) in addition to using standard precautions, contact precautions, and airborne precautions.[167]

 

The CDC outlines the guidelines for the use of personal protective equipment (PPE) during the pandemic. The recommended gear is a PPE gown, respirator or facemask, eye protection, and medical gloves.[168][169]

 

When available, respirators (instead of facemasks) are preferred.[170] N95 respirators are approved for industrial settings but the FDA has authorised the masks for use under an Emergency Use Authorisation (EUA). They are designed to protect from airborne particles like dust but effectiveness against a specific biological agent is not guaranteed for off-label uses.[171] When masks are not available, the CDC recommends using face shields or, as a last resort, homemade masks.[172]

 

Mechanical ventilation

Most cases of COVID‑19 are not severe enough to require mechanical ventilation or alternatives, but a percentage of cases are.[173][174] The type of respiratory support for individuals with COVID‑19 related respiratory failure is being actively studied for people in the hospital, with some evidence that intubation can be avoided with a high flow nasal cannula or bi-level positive airway pressure.[175] Whether either of these two leads to the same benefit for people who are critically ill is not known.[176] Some doctors prefer staying with invasive mechanical ventilation when available because this technique limits the spread of aerosol particles compared to a high flow nasal cannula.[173]

 

Severe cases are most common in older adults (those older than 60 years,[173] and especially those older than 80 years).[177] Many developed countries do not have enough hospital beds per capita, which limits a health system's capacity to handle a sudden spike in the number of COVID‑19 cases severe enough to require hospitalisation.[178] This limited capacity is a significant driver behind calls to flatten the curve.[178] One study in China found 5% were admitted to intensive care units, 2.3% needed mechanical support of ventilation, and 1.4% died.[41] In China, approximately 30% of people in hospital with COVID‑19 are eventually admitted to ICU.[4]

 

Acute respiratory distress syndrome

Main article: Acute respiratory distress syndrome

Mechanical ventilation becomes more complex as acute respiratory distress syndrome (ARDS) develops in COVID‑19 and oxygenation becomes increasingly difficult.[179] Ventilators capable of pressure control modes and high PEEP[180] are needed to maximise oxygen delivery while minimising the risk of ventilator-associated lung injury and pneumothorax.[181] High PEEP may not be available on older ventilators.

 

Options for ARDS[179]

TherapyRecommendations

High-flow nasal oxygenFor SpO2 <93%. May prevent the need for intubation and ventilation

Tidal volume6mL per kg and can be reduced to 4mL/kg

Plateau airway pressureKeep below 30 cmH2O if possible (high respiratory rate (35 per minute) may be required)

Positive end-expiratory pressureModerate to high levels

Prone positioningFor worsening oxygenation

Fluid managementGoal is a negative balance of 0.5–1.0L per day

AntibioticsFor secondary bacterial infections

GlucocorticoidsNot recommended

Experimental treatment

See also: § Research

Research into potential treatments started in January 2020,[182] and several antiviral drugs are in clinical trials.[183][184] Remdesivir appears to be the most promising.[133] Although new medications may take until 2021 to develop,[185] several of the medications being tested are already approved for other uses or are already in advanced testing.[186] Antiviral medication may be tried in people with severe disease.[144] The WHO recommended volunteers take part in trials of the effectiveness and safety of potential treatments.[187]

 

The FDA has granted temporary authorisation to convalescent plasma as an experimental treatment in cases where the person's life is seriously or immediately threatened. It has not undergone the clinical studies needed to show it is safe and effective for the disease.[188][189][190]

 

Information technology

See also: Contact tracing and Government by algorithm

In February 2020, China launched a mobile app to deal with the disease outbreak.[191] Users are asked to enter their name and ID number. The app can detect 'close contact' using surveillance data and therefore a potential risk of infection. Every user can also check the status of three other users. If a potential risk is detected, the app not only recommends self-quarantine, it also alerts local health officials.[192]

 

Big data analytics on cellphone data, facial recognition technology, mobile phone tracking, and artificial intelligence are used to track infected people and people whom they contacted in South Korea, Taiwan, and Singapore.[193][194] In March 2020, the Israeli government enabled security agencies to track mobile phone data of people supposed to have coronavirus. The measure was taken to enforce quarantine and protect those who may come into contact with infected citizens.[195] Also in March 2020, Deutsche Telekom shared aggregated phone location data with the German federal government agency, Robert Koch Institute, to research and prevent the spread of the virus.[196] Russia deployed facial recognition technology to detect quarantine breakers.[197] Italian regional health commissioner Giulio Gallera said he has been informed by mobile phone operators that "40% of people are continuing to move around anyway".[198] German government conducted a 48 hours weekend hackathon with more than 42.000 participants.[199][200] Two million people in the UK used an app developed in March 2020 by King's College London and Zoe to track people with COVID‑19 symptoms.[201] Also, the president of Estonia, Kersti Kaljulaid, made a global call for creative solutions against the spread of coronavirus.[202]

 

Psychological support

See also: Mental health during the 2019–20 coronavirus pandemic

Individuals may experience distress from quarantine, travel restrictions, side effects of treatment, or fear of the infection itself. To address these concerns, the National Health Commission of China published a national guideline for psychological crisis intervention on 27 January 2020.[203][204]

 

The Lancet published a 14-page call for action focusing on the UK and stated conditions were such that a range of mental health issues was likely to become more common. BBC quoted Rory O'Connor in saying, "Increased social isolation, loneliness, health anxiety, stress and an economic downturn are a perfect storm to harm people's mental health and wellbeing."[205][206]

 

Prognosis

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The severity of diagnosed cases in China

The severity of diagnosed COVID-19 cases in China[207]

Case fatality rates for COVID-19 by age by country.

Case fatality rates by age group:

China, as of 11 February 2020[208]

South Korea, as of 15 April 2020[209]

Spain, as of 24 April 2020[210]

Italy, as of 23 April 2020[211]

Case fatality rate depending on other health problems

Case fatality rate in China depending on other health problems. Data through 11 February 2020.[208]

Case fatality rate by country and number of cases

The number of deaths vs total cases by country and approximate case fatality rate[212]

The severity of COVID‑19 varies. The disease may take a mild course with few or no symptoms, resembling other common upper respiratory diseases such as the common cold. Mild cases typically recover within two weeks, while those with severe or critical diseases may take three to six weeks to recover. Among those who have died, the time from symptom onset to death has ranged from two to eight weeks.[47]

 

Children make up a small proportion of reported cases, with about 1% of cases being under 10 years, and 4% aged 10-19 years.[22] They are likely to have milder symptoms and a lower chance of severe disease than adults; in those younger than 50 years, the risk of death is less than 0.5%, while in those older than 70 it is more than 8%.[213][214][215] Pregnant women may be at higher risk for severe infection with COVID-19 based on data from other similar viruses, like SARS and MERS, but data for COVID-19 is lacking.[216][217] In China, children acquired infections mainly through close contact with their parents or other family members who lived in Wuhan or had traveled there.[213]

 

In some people, COVID‑19 may affect the lungs causing pneumonia. In those most severely affected, COVID-19 may rapidly progress to acute respiratory distress syndrome (ARDS) causing respiratory failure, septic shock, or multi-organ failure.[218][219] Complications associated with COVID‑19 include sepsis, abnormal clotting, and damage to the heart, kidneys, and liver. Clotting abnormalities, specifically an increase in prothrombin time, have been described in 6% of those admitted to hospital with COVID-19, while abnormal kidney function is seen in 4% of this group.[220] Approximately 20-30% of people who present with COVID‑19 demonstrate elevated liver enzymes (transaminases).[133] Liver injury as shown by blood markers of liver damage is frequently seen in severe cases.[221]

 

Some studies have found that the neutrophil to lymphocyte ratio (NLR) may be helpful in early screening for severe illness.[222]

 

Most of those who die of COVID‑19 have pre-existing (underlying) conditions, including hypertension, diabetes mellitus, and cardiovascular disease.[223] The Istituto Superiore di Sanità reported that out of 8.8% of deaths where medical charts were available for review, 97.2% of sampled patients had at least one comorbidity with the average patient having 2.7 diseases.[224] According to the same report, the median time between the onset of symptoms and death was ten days, with five being spent hospitalised. However, patients transferred to an ICU had a median time of seven days between hospitalisation and death.[224] In a study of early cases, the median time from exhibiting initial symptoms to death was 14 days, with a full range of six to 41 days.[225] In a study by the National Health Commission (NHC) of China, men had a death rate of 2.8% while women had a death rate of 1.7%.[226] Histopathological examinations of post-mortem lung samples show diffuse alveolar damage with cellular fibromyxoid exudates in both lungs. Viral cytopathic changes were observed in the pneumocytes. The lung picture resembled acute respiratory distress syndrome (ARDS).[47] In 11.8% of the deaths reported by the National Health Commission of China, heart damage was noted by elevated levels of troponin or cardiac arrest.[49] According to March data from the United States, 89% of those hospitalised had preexisting conditions.[227]

 

The availability of medical resources and the socioeconomics of a region may also affect mortality.[228] Estimates of the mortality from the condition vary because of those regional differences,[229] but also because of methodological difficulties. The under-counting of mild cases can cause the mortality rate to be overestimated.[230] However, the fact that deaths are the result of cases contracted in the past can mean the current mortality rate is underestimated.[231][232] Smokers were 1.4 times more likely to have severe symptoms of COVID‑19 and approximately 2.4 times more likely to require intensive care or die compared to non-smokers.[233]

 

Concerns have been raised about long-term sequelae of the disease. The Hong Kong Hospital Authority found a drop of 20% to 30% in lung capacity in some people who recovered from the disease, and lung scans suggested organ damage.[234] This may also lead to post-intensive care syndrome following recovery.[235]

 

Case fatality rates (%) by age and country

Age0–910–1920–2930–3940–4950–5960–6970–7980-8990+

China as of 11 February[208]0.00.20.20.20.41.33.68.014.8

Denmark as of 25 April[236]0.24.515.524.940.7

Italy as of 23 April[211]0.20.00.10.40.92.610.024.930.826.1

Netherlands as of 17 April[237]0.00.30.10.20.51.57.623.230.029.3

Portugal as of 24 April[238]0.00.00.00.00.30.62.88.516.5

S. Korea as of 15 April[209]0.00.00.00.10.20.72.59.722.2

Spain as of 24 April[210]0.30.40.30.30.61.34.413.220.320.1

Switzerland as of 25 April[239]0.90.00.00.10.00.52.710.124.0

Case fatality rates (%) by age in the United States

Age0–1920–4445–5455–6465–7475–8485+

United States as of 16 March[240]0.00.1–0.20.5–0.81.4–2.62.7–4.94.3–10.510.4–27.3

Note: The lower bound includes all cases. The upper bound excludes cases that were missing data.

Estimate of infection fatality rates and probability of severe disease course (%) by age based on cases from China[241]

0–910–1920–2930–3940–4950–5960–6970–7980+

Severe disease0.0

(0.0–0.0)0.04

(0.02–0.08)1.0

(0.62–2.1)3.4

(2.0–7.0)4.3

(2.5–8.7)8.2

(4.9–17)11

(7.0–24)17

(9.9–34)18

(11–38)

Death0.0016

(0.00016–0.025)0.0070

(0.0015–0.050)0.031

(0.014–0.092)0.084

(0.041–0.19)0.16

(0.076–0.32)0.60

(0.34–1.3)1.9

(1.1–3.9)4.3

(2.5–8.4)7.8

(3.8–13)

Total infection fatality rate is estimated to be 0.66% (0.39–1.3). Infection fatality rate is fatality per all infected individuals, regardless of whether they were diagnosed or had any symptoms. Numbers in parentheses are 95% credible intervals for the estimates.

Reinfection

As of March 2020, it was unknown if past infection provides effective and long-term immunity in people who recover from the disease.[242] Immunity is seen as likely, based on the behaviour of other coronaviruses,[243] but cases in which recovery from COVID‑19 have been followed by positive tests for coronavirus at a later date have been reported.[244][245][246][247] These cases are believed to be worsening of a lingering infection rather than re-infection.[247]

 

History

Main article: Timeline of the 2019–20 coronavirus pandemic

The virus is thought to be natural and has an animal origin,[77] through spillover infection.[248] The actual origin is unknown, but by December 2019 the spread of infection was almost entirely driven by human-to-human transmission.[208][249] A study of the first 41 cases of confirmed COVID‑19, published in January 2020 in The Lancet, revealed the earliest date of onset of symptoms as 1 December 2019.[250][251][252] Official publications from the WHO reported the earliest onset of symptoms as 8 December 2019.[253] Human-to-human transmission was confirmed by the WHO and Chinese authorities by 20 January 2020.[254][255]

 

Epidemiology

Main article: 2019–20 coronavirus pandemic

Several measures are commonly used to quantify mortality.[256] These numbers vary by region and over time and are influenced by the volume of testing, healthcare system quality, treatment options, time since the initial outbreak, and population characteristics such as age, sex, and overall health.[257]

 

The death-to-case ratio reflects the number of deaths divided by the number of diagnosed cases within a given time interval. Based on Johns Hopkins University statistics, the global death-to-case ratio is 7.0% (203,044/2,899,830) as of 26 April 2020.[7] The number varies by region.[258]

 

Other measures include the case fatality rate (CFR), which reflects the percent of diagnosed individuals who die from a disease, and the infection fatality rate (IFR), which reflects the percent of infected individuals (diagnosed and undiagnosed) who die from a disease. These statistics are not time-bound and follow a specific population from infection through case resolution. Many academics have attempted to calculate these numbers for specific populations.[259]

  

Total confirmed cases over time

 

Total deaths over time

 

Total confirmed cases of COVID‑19 per million people, 10 April 2020[260]

 

Total confirmed deaths due to COVID‑19 per million people, 10 April 2020[261]

Infection fatality rate

Our World in Data states that as of March 25, 2020, the infection fatality rate (IFR) cannot be accurately calculated.[262] In February, the World Health Organization estimated the IFR at 0.94%, with a confidence interval between 0.37 percent to 2.9 percent.[263] The University of Oxford Centre for Evidence-Based Medicine (CEBM) estimated a global CFR of 0.72 percent and IFR of 0.1 percent to 0.36 percent.[264] According to CEBM, random antibody testing in Germany suggested an IFR of 0.37 percent there.[264] Firm lower limits to local infection fatality rates were established, such as in Bergamo province, where 0.57% of the population has died, leading to a minimum IFR of 0.57% in the province. This population fatality rate (PFR) minimum increases as more people get infected and run through their disease.[265][266] Similarly, as of April 22 in the New York City area, there were 15,411 deaths confirmed from COVID-19, and 19,200 excess deaths.[267] Very recently, the first results of antibody testing have come in, but there are no valid scientific reports based on them available yet. A Bloomberg Opinion piece provides a survey.[268][269]

 

Sex differences

Main article: Gendered impact of the 2019–20 coronavirus pandemic

The impact of the pandemic and its mortality rate are different for men and women.[270] Mortality is higher in men in studies conducted in China and Italy.[271][272][273] The highest risk for men is in their 50s, with the gap between men and women closing only at 90.[273] In China, the death rate was 2.8 percent for men and 1.7 percent for women.[273] The exact reasons for this sex-difference are not known, but genetic and behavioural factors could be a reason.[270] Sex-based immunological differences, a lower prevalence of smoking in women, and men developing co-morbid conditions such as hypertension at a younger age than women could have contributed to the higher mortality in men.[273] In Europe, of those infected with COVID‑19, 57% were men; of those infected with COVID‑19 who also died, 72% were men.[274] As of April 2020, the U.S. government is not tracking sex-related data of COVID‑19 infections.[275] Research has shown that viral illnesses like Ebola, HIV, influenza, and SARS affect men and women differently.[275] A higher percentage of health workers, particularly nurses, are women, and they have a higher chance of being exposed to the virus.[276] School closures, lockdowns, and reduced access to healthcare following the 2019–20 coronavirus pandemic may differentially affect the genders and possibly exaggerate existing gender disparity.[270][277]

 

Society and culture

Name

During the initial outbreak in Wuhan, China, the virus and disease were commonly referred to as "coronavirus" and "Wuhan coronavirus",[278][279][280] with the disease sometimes called "Wuhan pneumonia".[281][282] In the past, many diseases have been named after geographical locations, such as the Spanish flu,[283] Middle East Respiratory Syndrome, and Zika virus.[284]

 

In January 2020, the World Health Organisation recommended 2019-nCov[285] and 2019-nCoV acute respiratory disease[286] as interim names for the virus and disease per 2015 guidance and international guidelines against using geographical locations (e.g. Wuhan, China), animal species or groups of people in disease and virus names to prevent social stigma.[287][288][289]

 

The official names COVID‑19 and SARS-CoV-2 were issued by the WHO on 11 February 2020.[290] WHO chief Tedros Adhanom Ghebreyesus explained: CO for corona, VI for virus, D for disease and 19 for when the outbreak was first identified (31 December 2019).[291] The WHO additionally uses "the COVID‑19 virus" and "the virus responsible for COVID‑19" in public communications.[290] Both the disease and virus are commonly referred to as "coronavirus" in the media and public discourse.

 

Misinformation

Main article: Misinformation related to the 2019–20 coronavirus pandemic

After the initial outbreak of COVID‑19, conspiracy theories, misinformation, and disinformation emerged regarding the origin, scale, prevention, treatment, and other aspects of the disease and rapidly spread online.[292][293][294][295]

 

Protests

Beginning April 17, 2020, news media began reporting on a wave of demonstrations protesting against state-mandated quarantine restrictions in in Michigan, Ohio, and Kentucky.[296][297]

 

Other animals

Humans appear to be capable of spreading the virus to some other animals. A domestic cat in Liège, Belgium, tested positive after it started showing symptoms (diarrhoea, vomiting, shortness of breath) a week later than its owner, who was also positive.[298] Tigers at the Bronx Zoo in New York, United States, tested positive for the virus and showed symptoms of COVID‑19, including a dry cough and loss of appetite.[299]

 

A study on domesticated animals inoculated with the virus found that cats and ferrets appear to be "highly susceptible" to the disease, while dogs appear to be less susceptible, with lower levels of viral replication. The study failed to find evidence of viral replication in pigs, ducks, and chickens.[300]

 

Research

Main article: COVID-19 drug development

No medication or vaccine is approved to treat the disease.[186] International research on vaccines and medicines in COVID‑19 is underway by government organisations, academic groups, and industry researchers.[301][302] In March, the World Health Organisation initiated the "SOLIDARITY Trial" to assess the treatment effects of four existing antiviral compounds with the most promise of efficacy.[303]

 

Vaccine

Main article: COVID-19 vaccine

There is no available vaccine, but various agencies are actively developing vaccine candidates. Previous work on SARS-CoV is being used because both SARS-CoV and SARS-CoV-2 use the ACE2 receptor to enter human cells.[304] Three vaccination strategies are being investigated. First, researchers aim to build a whole virus vaccine. The use of such a virus, be it inactive or dead, aims to elicit a prompt immune response of the human body to a new infection with COVID‑19. A second strategy, subunit vaccines, aims to create a vaccine that sensitises the immune system to certain subunits of the virus. In the case of SARS-CoV-2, such research focuses on the S-spike protein that helps the virus intrude the ACE2 enzyme receptor. A third strategy is that of the nucleic acid vaccines (DNA or RNA vaccines, a novel technique for creating a vaccination). Experimental vaccines from any of these strategies would have to be tested for safety and efficacy.[305]

 

On 16 March 2020, the first clinical trial of a vaccine started with four volunteers in Seattle, United States. The vaccine contains a harmless genetic code copied from the virus that causes the disease.[306]

 

Antibody-dependent enhancement has been suggested as a potential challenge for vaccine development for SARS-COV-2, but this is controversial.[307]

 

Medications

Main article: COVID-19 drug repurposing research

At least 29 phase II–IV efficacy trials in COVID‑19 were concluded in March 2020 or scheduled to provide results in April from hospitals in China.[308][309] There are more than 300 active clinical trials underway as of April 2020.[133] Seven trials were evaluating already approved treatments, including four studies on hydroxychloroquine or chloroquine.[309] Repurposed antiviral drugs make up most of the Chinese research, with nine phase III trials on remdesivir across several countries due to report by the end of April.[308][309] Other candidates in trials include vasodilators, corticosteroids, immune therapies, lipoic acid, bevacizumab, and recombinant angiotensin-converting enzyme 2.[309]

 

The COVID‑19 Clinical Research Coalition has goals to 1) facilitate rapid reviews of clinical trial proposals by ethics committees and national regulatory agencies, 2) fast-track approvals for the candidate therapeutic compounds, 3) ensure standardised and rapid analysis of emerging efficacy and safety data and 4) facilitate sharing of clinical trial outcomes before publication.[310][311]

 

Several existing medications are being evaluated for the treatment of COVID‑19,[186] including remdesivir, chloroquine, hydroxychloroquine, lopinavir/ritonavir, and lopinavir/ritonavir combined with interferon beta.[303][312] There is tentative evidence for efficacy by remdesivir, as of March 2020.[313][314] Clinical improvement was observed in patients treated with compassionate-use remdesivir.[315] Remdesivir inhibits SARS-CoV-2 in vitro.[316] Phase III clinical trials are underway in the U.S., China, and Italy.[186][308][317]

 

In 2020, a trial found that lopinavir/ritonavir was ineffective in the treatment of severe illness.[318] Nitazoxanide has been recommended for further in vivo study after demonstrating low concentration inhibition of SARS-CoV-2.[316]

 

There are mixed results as of 3 April 2020 as to the effectiveness of hydroxychloroquine as a treatment for COVID‑19, with some studies showing little or no improvement.[319][320] The studies of chloroquine and hydroxychloroquine with or without azithromycin have major limitations that have prevented the medical community from embracing these therapies without further study.[133]

 

Oseltamivir does not inhibit SARS-CoV-2 in vitro and has no known role in COVID‑19 treatment.[133]

 

Anti-cytokine storm

Cytokine release syndrome (CRS) can be a complication in the later stages of severe COVID‑19. There is preliminary evidence that hydroxychloroquine may have anti-cytokine storm properties.[321]

 

Tocilizumab has been included in treatment guidelines by China's National Health Commission after a small study was completed.[322][323] It is undergoing a phase 2 non-randomised trial at the national level in Italy after showing positive results in people with severe disease.[324][325] Combined with a serum ferritin blood test to identify cytokine storms, it is meant to counter such developments, which are thought to be the cause of death in some affected people.[326][327][328] The interleukin-6 receptor antagonist was approved by the FDA to undergo a phase III clinical trial assessing the medication's impact on COVID‑19 based on retrospective case studies for the treatment of steroid-refractory cytokine release syndrome induced by a different cause, CAR T cell therapy, in 2017.[329] To date, there is no randomised, controlled evidence that tocilizumab is an efficacious treatment for CRS. Prophylactic tocilizumab has been shown to increase serum IL-6 levels by saturating the IL-6R, driving IL-6 across the blood-brain barrier, and exacerbating neurotoxicity while having no impact on the incidence of CRS.[330]

 

Lenzilumab, an anti-GM-CSF monoclonal antibody, is protective in murine models for CAR T cell-induced CRS and neurotoxicity and is a viable therapeutic option due to the observed increase of pathogenic GM-CSF secreting T-cells in hospitalised patients with COVID‑19.[331]

 

The Feinstein Institute of Northwell Health announced in March a study on "a human antibody that may prevent the activity" of IL-6.[332]

 

Passive antibodies

Transferring purified and concentrated antibodies produced by the immune systems of those who have recovered from COVID‑19 to people who need them is being investigated as a non-vaccine method of passive immunisation.[333] This strategy was tried for SARS with inconclusive results.[333] Viral neutralisation is the anticipated mechanism of action by which passive antibody therapy can mediate defence against SARS-CoV-2. Other mechanisms, however, such as antibody-dependent cellular cytotoxicity and/or phagocytosis, may be possible.[333] Other forms of passive antibody therapy, for example, using manufactured monoclonal antibodies, are in development.[333] Production of convalescent serum, which consists of the liquid portion of the blood from recovered patients and contains antibodies specific to this virus, could be increased for quicker deployment.[334]

  

en.wikipedia.org/wiki/Coronavirus_disease_2019

Spotted at the Art Institute of Chicago.

Our Lady of Al-Natour monastery

 

Is standing on the Cape of Al-Natour, was built by the Crusaders on Byzantine ruins. The monastery is surrounded by large fields of the myrtle (Myrtus Ugni), a nearly extinct plant on the Lebanese coast.

In the last few years, the monastery church and rooms have been renovated, and icons of saints are painted on the church walls.

I found on the internet that this is monastery is 900 years old.

photos taken 26/12/2014

Iandra Castle. George and Elizabeth Greene were settlers with a difference when they purchased their 32,000 acre property in 1878. They built their first homestead named Mount Oriel House in 1880. Greene was a NSW politician and a man with ideas. By the time he died in 1911 his Mount Oriel estate had over 20,000 acres sown in crop. His obituary said he came to a landscape of bush and transformed it into a granary. His estate used 700 horses and employed over 600 people plus various chaffcutters, thrashers etc and the woolshed sheared 30,000 sheep. He came to NSW in 1847 with his parents and spent most of his life on pastoral properties before he purchased Iandra. He dreamed of a medieval feudal system to grow vast areas of crop with little labour. He was regarded as the most important wheat farmer in Australia along with William Farrer who developed his rust resistant wheat type. Greene claims to have introduced the concept of share farming to Australia. He provided the land and took half of the value of the crop but the share farmer bought the seed, fertiliser and provided the labour to sow and reap the crop. But did he? Share cropping was common in the American south after the Civil War and Elizabeth Onlsow (nee MacArthur of Camden Park NSW) introduced share farming on their dairy property in 1887. Greene established his village with houses for the fifty sharefarmers contracted to work his lands. He started with one share farmer in 1891 and gradually expanded the system. His estate manager, named Leonard l’Anson came from Waterloo in South Australia and members of the Freebairn family from Alma in SA also moved to Iandra to be share farmers. George Greene wanted a medieval castle like a medieval lord. Iandra castle was built in 1908 with 57 rooms, castellations and towers but the construction was decidedly modern with reinforced concrete walls. The style was slightly Gothic but the interior was very Edwardian with wood panelling and Art Nouveau stained glass panels etc. The external concrete was rendered to appear like stone. It cost around £63,000 to build. The property included stables, a manager’s residence, outbuildings, blacksmith shop, sheds etc. Near the house was a chapel built in 1886 and a cemetery. When George Greene died in 1911 he was buried there. The estate was partially broken up in 1914 and most share farmers were able to buy their 640 acre blocks. I’Anson continued as manager for Elizabeth Greene until her death in 1927. He was then able to buy 2,500 acres and the castle. Later Iandra castle was used as the Methodist Boys Home from 1954 to 1974. The Methodist Boys Farm School was for 15 to 18 year old first time offenders. They were taught farming skills. The Methodist Church sold the centre in 1974.

  

Bodybuilders at the FIBO 2012 expo, Essen, Germany.

examples on how to tell apart the ranks of geisha. Featuring miyagawacho maiko Toshiteru and Geiko Kikusturu.

....a day makes. Today I can feel a hint of spring in the air!

Hit 'L' to view on large.

 

On euro tour with Sean and Neil for 3 days of mayhem and a grand total of 8 hours sleep. Taking in some old and new sites and showing them some of what is out there as it was their first time. Also made time to meet up with the SPEXS crew.

 

Unsure on the history of when this rectory/convent was abandoned but seems to be a while now.

 

My blog:

 

timster1973.wordpress.com

 

Also on Facebook

 

www.Facebook.com/TimKniftonPhotography

 

online store: www.artfinder.com/tim-knifton

"Two roads diverged in a wood, and I-

I took the one less traveled by,

And that has made all the difference."

(Robert Frost)

Although the panel shape and palette of this painting and another mummy portrait in the Met’s collection (09.181.2) are very similar, differences in the hair and beards suggest that the portrait of the man with the mole (09.181.2) is about a generation earlier than the portrait here. This man's body is turned more directly toward the viewer. He has bushy black hair, large round eyes, tawny skin, and a thick beard that emphasizes his emaciated cheeks. The use of light and shadow on various parts of the face and neck is very skilled.

 

The term encaustic designates painting in which beeswax is mixed with pigments. The technique was first used by Greek painters of the fifth and fourth centuries B.C. Ancient descriptions are difficult to interpret, and Egyptian mummy portraits--the only works in the medium surviving from antiquity--have not yet been analyzed extensively. Therefore, many questions concerning the details of the techniques used by mummy-portrait painters remain open, although the work of a few scholars and modern artists who paint in encaustic has provided some insight.

 

The first step in producing a mummy portrait was to sketch the outlines of face and garment on a wood panel that had been prepared with distemper, transparent glue, or dark wax. Then, a mixture of beeswax with powdered pigments was prepared. The wax could be used hot or cold. When used in molten state, the wax was either pure or supplemented with resin, glue, egg, gum, or oil. The molten beeswax/ pigment mixture was laid on quickly with long even brushstrokes, employing a thinner mixture for the background and garments and thick creamy paste-like

paints for the facial features. When the wax cooled, a hard tool was used to blend the various flesh tones of face and neck.

 

When used cold, the wax had to be made suitable for painting by a process called emulsification, in which alkalis were added that allowed the wax to be suspended in egg or oil. The ancient writers appear to refer to this as "Punic wax." Emulsified wax paints are said to dry more slowly than molten wax paints, giving the painter greater control, and time to correct and adjust during the painting process. Mummy portraits that show only brushstrokes, no hard tool marks, are thought to have been created with emulsified cold wax. Many modern artists who paint in encaustic actually employ a heated tool to fuse paints, hence the term encaustic, which means "burnt in." Ancient painters probably did not use such a technique.

 

Romano-Egyptian, from Egypt, 160-180 CE

Encaustic (pigmented wax) on limewood

 

Met Museum, New York (09.181.1)

These two very different East Lancs bodied buses are seen here on display at a rally in Keighley, in 19/06/2005. On the right of the photo and obviously the elder of the two is preserved Plymouth City Transport, 168, TTT168X, a Leyland Atlantean AN68C/1R. This was delivered in the final batch of ten, 162-71, TTT162X etc., in 12/1981. The following three numerically numbered buses delivered to the south-west municipal were 172-4, TTT172-4X, being East Lancs bodied Leyland Olympian ONLXB/1R's. They were delivered in 03/1982. Alongside the Atlantean, and a very much different beast is Yorkshire Traction, 802, YN54VKB, a Scania N94 UD. This coach seated East Lancs OmniDekka bodied Scania was delivered in a batch of six, 801-6, YN54VKA-F. It later became Stagecoach Yorkshire Traction 15416 and then later passed to Harrogate Coach Travel - Connexions Buses, Tockwith, where it was re-registered UUG384 and still remains in service today.

 

The camera being a Pentax MZ-M with the film being a Fujichrome Colourslide.

 

I would request, as with all my photos, that they are not copied or downloaded in any way, shape or form. © Peter Steel 2005.

what a difference a day makes - well actually it was less than 4 hours. Same flower in the process from the opening of a bud to bloom.

if there is interest for other works: www.facebook.com/ronnyengelmannphotography

Since flickr updated/was down for a bit ... and because I upload at least one per day ... here are several photos to look at ... :)

 

Thanks to Kim for putting this diptych together.

 

The bird with pink on the right is a Turkey Vulture (Cathartes aura). It is a common, almost abundant species along the Texas Gulf Coast plains. The bird on the left is a Black Vulture (Coragyps atratus) - not the same species as Black Vulture in the Old World. As you can see the head and neck are featherless with the skin being grey and wrinkled. Like Turkey Vulture, the Black Vulture is also reasonably frequent along the Gulf Coast.

 

The two species were actually photographed with a few yards of each other. Both are scavengers, cleaning up nature's carcasses.

Woman, but there's jewellery in there. Dog, yes, but I can smell something down the alley.

Op de foto van vandaag -5 februari. 2015 - 3 Kleine Zwanen en 5 Wilde Zwanen .

Kun je zien welke de 3 kleine zwanen zijn? Best moeilijk, ik moet ook altijd goed kijken.

 

Oplossing in tweede comment

 

De boer is hier niet blij mee. De 51 zwanen vreten in hoog tempo het pas ingezaaide gras op. Hij krijgt hiervoor geen compensatie.

 

Kleine Zwaan:

De kleine zwaan (Cygnus bewickii) is de kleinste van de drie soorten zwanen die regelmatig in Nederland en België voorkomen. De soort is nauw verwant aan de fluitzwaan, uit Noord-Amerika, en wordt ook wel als een ondersoort van deze soort beschouwd: Cygnus columbianus bewickii. Kleine zwanen broeden 's zomers op de Russische toendra. In de winter komen ze met duizenden naar onder andere Nederland om daar te overwinteren.

 

Kleine zwanen verschillen van de knobbelzwaan onder andere door hun grootte. De kleine zwaan wordt zo'n 127 centimeter, en de knobbelzwaan wel 150 centimeter (snavelpunt tot staart). Kleine zwanen hebben bovendien geen knobbel bovenop de snavel, en de vlek op de snavel is geel, in tegenstelling tot de oranje kleur die een groot deel van de snavel van de knobbelzwaan heeft. Van de wilde zwaan, die even groot is als de knobbelzwaan, verschilt de kleine zwaan behalve door het formaat ook doordat de gele vlek op de snavel van de wilde zwaan groter is, en naar voren in een punt uitloopt; de gele vlek van de kleine zwaan is kleiner, aan de voorkant afgerond, en loopt niet zo ver naar voren door. Kleine zwanen foerageren soms met honderden tegelijk op graslanden. 's Avonds is hun roep te horen als ze van de voedselplek naar een veilige slaapplek vliegen. Zo'n plaats is vaak een meer of afgesloten natuurgebied dat 's nachts voldoende veiligheid biedt. Kleine zwanen zijn vanaf oktober tot maart te zien in Nederland.

 

Kenmerken[bewerken]

Kleur: ....................wit, snavel is zwart-geel

Geluid:................. melodieus hoge tonen "klioe-kloeie"

Voedsel:............ gras, toendramossen en waterplanten.

Lengte: ............. ongeveer 120 cm.

Spanwijdte:.......ongeveer 180 cm.

  

bron wikipedia

  

D7000, Nikon,

 

14X_8140PVCCN+.jpg

這邊好像不小心就會拍到類似的構圖吼~~

I recently uploaded an image showing this location with trees planted to hide the scene seen here. As you can possibly see, the difference in the view between both pictures is immense.

All my photo here NON HDR/NON DRi or blended images, they are taken from single shoot

 

Facebook I 500px I Google+ I ArtFlakes | Microstockers Malaysia

 

Technical info:

 

Nikkor 50mm (1.4D)

f8

ISO 100

50 mm

10s exposure

 

Post Processing:

PS CC(30%)+Lightroom 5.2 (70%)

 

-----------------------------------------------------------------------------------------------------------------------------------

©1435/2013 AZIRULL AMIN ARIPIN

I'm now licensing my photos through the Flickr Collection on Getty Images. If you'd like to use this image for commercial purposes please request to license (just there on the right) or drop me a message through flickr.

This trio of Leyland Atlantean buses is seen here at the "Leyland Atlantean 40" event at The Historic Commercial Vehicle Museum, Leyland, on 27/09/1998. This event was being held to commemorate the 40th. anniversary of the production of the first Leyland Atlantean and it coincided with Preston Bus first ever "enthusiasts day", being held at their Deepdale Road depot and a free bus service was provided between the two events.

 

Left to right, facing the camera, are; Preserved Ribble, 1686, NRN586, a Metro-Cammell bodied PDR1/1 type, new to Ribble, in 10/1960; Centre is, First Manchester, 7077, WBN955L, an open-top converted Park Royal bodied AN68/1R type. New to SELNEC(7077), in 07/1972, this early 'Manchester Standard' was converted by SELNEC's successor, Greater Manchester PTE, in 09/1985. Moving onto GM Buses North, it was based at Bolton when seen here. On the right is former Glasgow Corporation Transport, LA352, KUS607E, an Alexander bodied PDR1/1 type. In the livery of Edwards Travel, Newtown, Llantwit Fardre, Wales, it was new to Glasgow, in 05/1967 and I believe, as of 2020, it still survives as a preservation/restoration candidate having recently arrived back in it's former home city, at the GVVT Museum, Bridgeton, Glasgow, Scotland, having been a onetime resident at the North West Museum of Road Transport, St. Helens. As a footnote, this bus carried registration MUS281F, from LA396, when with Beestons, Hadleigh, and then some other operators, for some strange reason.

 

The camera being a Praktica MTL3 with the film being a Boots Colourslide II.

 

I would request, as with all my photos, that they are not copied or downloaded in any way, shape or form. © Peter Steel 1998.

A missing filter... Picture or maybe in the air of the time that brews a little anguish, it grinds ideas by dint of filtering the words... the cunning life with a twist. Angel or mill?

  

The COVID-19 pandemic has resulted in conspiracy theories and misinformation about the scale of the pandemic and the origin, prevention, diagnosis, and treatment of the disease.[1][2][3] False information, including intentional disinformation, has been spread through social media,[2][4] text messages,[5] and mass media,[6] including the tabloid media,[7] conservative media,[8][9] state media of countries such as China,[10][11] Russia,[12][13] Iran,[14] and Turkmenistan.[2][15] It has also been spread by state-backed covert operations to generate panic and sow distrust in other countries.[16][17]

 

Misinformation has been propagated by celebrities, politicians[18][19] (including heads of state in countries such as the United States,[20][21] Iran,[22] and Brazil[23]), and other prominent public figures.[24] Commercial scams have claimed to offer at-home tests, supposed preventives, and "miracle" cures.[25][26] Politicians and leaders of some countries have promoted purported cures, while some religious groups said that the faith of their followers and God will protect them from the virus.[27][28][29] Others have claimed the virus is a lab-developed bio-weapon that was accidentally leaked,[30][31] or deliberately designed to target a country,[32] or one with a patented vaccine, a population control scheme, the result of a spy operation,[3][4] or linked to 5G networks.[33]

 

The World Health Organization has declared an "infodemic" of incorrect information about the virus, which poses risks to global health.[2]

 

Types and origin and effect

On January 30, the BBC reported about the increasing spread of conspiracy theories and false health advice in relation to COVID-19. Notable examples at the time included false health advice shared on social media and private chats, as well as conspiracy theories such as the origin in bat soup and the outbreak being planned with the participation of the Pirbright Institute.[1][34] On January 31, The Guardian listed seven instances of misinformation, adding the conspiracy theories about bioweapons and the link to 5G technology, and including varied false health advice.[35]

 

In an attempt to speed up research sharing, many researches have turned to preprint servers such as arXiv, bioRxiv, medRxiv or SSRN. Papers can be uploaded to these servers without peer review or any other editorial process that ensures research quality. Some of these papers have contributed to the spread of conspiracy theories. The most notable case was a preprint paper uploaded to bioRxiv which claimed that the virus contained HIV "insertions". Following the controversy, the paper was withdrawn.[36][37][38]

 

According to a study published by the Reuters Institute for the Study of Journalism, most misinformation related to COVID-19 involves "various forms of reconfiguration, where existing and often true information is spun, twisted, recontextualised, or reworked". While less misinformation "was completely fabricated". The study found no deep fakes in the studied sample. The study also found that "top-down misinformation from politicians, celebrities, and other prominent public figures", while accounting for a minority of the samples, captured a majority of the social media engagement. According to their classification, the largest category of misinformation (39%) includes "misleading or false claims about the actions or policies of public authorities, including government and international bodies like the WHO or the UN".[39]

 

A natural experiment correlated coronavirus misinformation with increased infection and death; of two similar television news shows on the same network, one took coronavirus seriously about a month earlier than the other. People and groups exposed to the slow-response news show had higher infection and death rates.[40]

 

The misinformations have been used by politicians, interest groups, and state actors in many countries to scapegoat other countries for the mishandling of the domestic responses, as well as furthering political, financial agenda.[41][42][43]

 

Combative efforts

Further information: Impact of the 2019–20 coronavirus pandemic on journalism

File:ITU - AI for Good Webinar Series - COVID-19 Misinformation and Disinformation during COVID-19.webm

International Telecommunication Union

On February 2, the World Health Organization (WHO) described a "massive infodemic", citing an over-abundance of reported information, accurate and false, about the virus that "makes it hard for people to find trustworthy sources and reliable guidance when they need it". The WHO stated that the high demand for timely and trustworthy information has incentivised the creation of a direct WHO 24/7 myth-busting hotline where its communication and social media teams have been monitoring and responding to misinformation through its website and social media pages.[44][45][46] The WHO specifically debunked several claims as false, including the claim that a person can tell if they have the virus or not simply by holding their breath; the claim that drinking large amounts of water will protect against the virus; and the claim that gargling salt water prevents infection.[47]

 

In early February, Facebook, Twitter and Google said they were working with WHO to address "misinformation".[48] In a blogpost, Facebook stated they would remove content flagged by global health organizations and local authorities that violate its content policy on misinformation leading to "physical harm".[49] Facebook is also giving free advertising to WHO.[50] Nonetheless, a week after Trump's speculation that sunlight could kill the virus, the New York Times found "780 Facebook groups, 290 Facebook pages, nine Instagram accounts and thousands of tweets pushing UV light therapies," content which those companies declined to remove from their platforms.[51]

 

At the end of February, Amazon removed more than a million products claimed to cure or protect against coronavirus, and removed tens of thousands of listings for health products whose prices were "significantly higher than recent prices offered on or off Amazon", although numerous items were "still being sold at unusually high prices" as of February 28.[52]

 

Millions of instances of COVID-19 misinformation have occurred across a number of online platforms.[53] Other fake news researchers noted certain rumors started in China; many of them later spread to Korea and the United States, prompting several universities in Korea to start the multilingual Facts Before Rumors campaign to separate common claims seen online.[54][55][56][57]

 

The media has praised Wikipedia's coverage of COVID-19 and its combating the inclusion of misinformation through efforts led by the Wiki Project Med Foundation and the English-language Wikipedia's WikiProject Medicine, among other groups.[58][59][60]

 

Many local newspapers have been severely affected by losses in advertising revenues from coronavirus; journalists have been laid off, and some have closed altogether.[61]

 

Many newspapers with paywalls lowered them for some or all their coronavirus coverage.[62][63] Many scientific publishers made scientific papers related to the outbreak open access.[64]

 

The Turkish Interior Ministry has been arresting social media users whose posts were "targeting officials and spreading panic and fear by suggesting the virus had spread widely in Turkey and that officials had taken insufficient measures".[65] Iran's military said 3600 people have been arrested for "spreading rumors" about coronavirus in the country.[66] In Cambodia, some individuals who expressed concerns about the spread of COVID-19 have been arrested on fake news charges.[67][68] Algerian lawmakers passed a law criminalising "fake news" deemed harmful to "public order and state security".[69] In the Philippines,[70] China,[71] India,[72][73] Egypt,[74] Bangladesh,[75] Morocco,[76] Pakistan,[77] Saudi Arabia,[78] Oman,[79] Iran,[80] Vietnam, Laos,[81] Indonesia,[73] Mongolia,[73] Sri Lanka,[73] Kenya, South Africa,[82] Somalia,[83] Thailand,[84] Kazakhstan,[85] Azerbaijan,[86] Malaysia[87] and Hong Kong, people have been arrested for allegedly spreading false information about the coronavirus pandemic.[88][73] The United Arab Emirates have introduced criminal penalties for the spread of misinformation and rumours related to the outbreak.[89]

 

Conspiracy theories

Conspiracy theories have appeared both in social media and in mainstream news outlets, and are heavily influenced by geopolitics.[90]

 

Accidental leakage

 

Virologist and immunologist Vincent R. Racaniello said that "accident theories – and the lab-made theories before them – reflect a lack of understanding of the genetic make-up of Sars-CoV-2."[91]

A number of allegations have emerged supposing a link between the virus and Wuhan Institute of Virology (WIV); among these is that the virus was an accidental leakage from WIV.[92] In 2017, U.S. molecular biologist Richard H. Ebright expressed caution when the WIV was expanded to become mainland China's first biosafety level 4 (BSL-4) laboratory, noting previous escapes of the SARS virus at other Chinese laboratories.[93] While Ebright refuted several conspiracy theories regarding the WIV (e.g., bioweapons research, or that the virus was engineered), he told BBC China this did not represent the possibility that the virus can be "completely ruled out" from entering the population due to a laboratory accident.[92] Various researchers contacted by NPR concluded there was "virtually no chance" (in NPR's words) that the pandemic virus had accidentally escaped from a laboratory.[94] Disinformation researcher Nina Jankowicz from Wilson Center indicates the lab leakage claim entered mainstream media in United States during April, propagated by pro-Trump news outlet.[43]

 

On February 14, 2020, Chinese scientists explored the possibility of accidental leakage and published speculations on scientific social networking website ResearchGate. The paper was neither peer-reviewed nor presented any evidence for its claims.[95] On March 5, the author of paper told Wall Street Journal in an interview why he decided to withdrew the paper by the end of February, stating: "the speculation about the possible origins in the post was based on published papers and media, and was not supported by direct proofs."[96][97] Several newspapers have referenced the paper.[95] Scientific American reported that Shi Zhengli, the lead researcher at WIV, started investigation on mishandling of experimental materials in the lab records, especially during disposal. She also tried to cross-check the novel coronavirus genome with the genetic information of other bat coronaviruses her team had collected. The result showed none of the sequences matched those of the viruses her team had sampled from bat caves.[98]

 

In February, it was alleged that the first person infected may have been a researcher at the institute named Huang Yanling.[99] Rumours circulated on Chinese social media that the researcher had become infected and died, prompting a denial from WIV, saying she was a graduate student enrolled in the Institute until 2015 and is not the patient zero.[100][99] In April, the conspiracy theory started to circulate around on Youtube and got picked up by conservative media, National Review.[101][6]

 

The South China Morning Post (SCMP) reported that one of the WIV's lead researchers, Shi Zhengli, was the particular focus of personal attacks in Chinese social media alleging that her work on bat-based viruses was the source of the virus; this led Shi to post: "I swear with my life, [the virus] has nothing to do with the lab". When asked by the SCMP to comment on the attacks, Shi responded: "My time must be spent on more important matters".[102] Caixin reported Shi made further public statements against "perceived tinfoil-hat theories about the new virus's source", quoting her as saying: "The novel 2019 coronavirus is nature punishing the human race for keeping uncivilized living habits. I, Shi Zhengli, swear on my life that it has nothing to do with our laboratory".[103] Immunologist Vincent Racaniello stated that virus leaking theory "reflect a lack of understanding of the genetic make-up of Sars-CoV-2 and its relationship to the bat virus". He says the bat virus researched in the institution "would not have been able to infect humans—the human Sars-CoV-2 has additional changes that allows it to infect humans."[91]

 

On April 14, the U.S. Chairman of the Joint Chiefs of Staff, General Mark Milley, in response to questions about the virus being manufactured in a lab, said "... it's inconclusive, although the weight of evidence seems to indicate natural. But we don't know for certain."[104] On that same day, Washington Post columnist Josh Rogin detailed a leaked cable of a 2018 trip made to the WIV by scientists from the U.S. Embassy. The article was referenced and cited by conservative media to push the lab leakage theory.[43] Rogin's article went on to say that "What the U.S. officials learned during their visits concerned them so much that they dispatched two diplomatic cables categorized as Sensitive But Unclassified back to Washington. The cables warned about safety and management weaknesses at the WIV lab and proposed more attention and help. The first cable, which I obtained, also warns that the lab's work on bat coronaviruses and their potential human transmission represented a risk of a new SARS-like pandemic."[105] Rogin's article pointed out there was no evidence that the coronavirus was engineered, "But that is not the same as saying it didn't come from the lab, which spent years testing bat coronaviruses in animals."[105] The article went on to quote Xiao Qiang, a research scientist at the School of Information at the University of California, Berkeley, "I don't think it's a conspiracy theory. I think it's a legitimate question that needs to be investigated and answered. To understand exactly how this originated is critical knowledge for preventing this from happening in the future."[105] Washington Post's article and subsequent broadcasts drew criticism from virologist Angela Rasmussen of Columbia University, which she states "It's irresponsible for political reporters like Rogin [to] uncritically regurgitate a secret 'cable' without asking a single virologist or ecologist or making any attempt to understand the scientific context."[43] Rasmussen later compared biosafety procedure concerns to "having the health inspector come to your restaurant. It could just be, ‘Oh, you need to keep your chemical showers better stocked.’ It doesn’t suggest, however, that there are tremendous problems.”[106]

 

Days later, multiple media outlets confirmed that U.S. intelligence officials were investigating the possibility that the virus started in the WIV.[107][108][109][110] On April 23, Vox presented disputed arguments on lab leakage claims from several scientists.[111] Scientists suggested that virus samples cultured in the lab have significant amount of difference compare to SARS-CoV-2. The virus institution sampled RaTG13 in Yunnan, the closest known relative of the novel coronavirus with 96% shared genome. Edward Holmes, SARS-CoV-2 researcher at the University of Sydney, explained 4% of difference "is equivalent to an average of 50 years (and at least 20 years) of evolutionary change."[111][112] Virologist Peter Daszak, president of the EcoHealth Alliance, which studies emerging infectious diseases, noted the estimation that 1–7 million people in Southeast Asia who live or work in proximity to bats are infected each year with bat coronaviruses. In the interview with Vox, he comments, "There are probably half a dozen people that do work in those labs. So let's compare 1 million to 7 million people a year to half a dozen people; it's just not logical."[94][111]

 

On April 30, The New York Times reported the Trump administration demanded intelligence agencies to find evidence linking WIV with the origin of SARS-Cov-2. Secretary of State and former Central Intelligence Agency (C.I.A) director Mike Pompeo was reportedly leading the push on finding information regarding the virus origin. Analysts were concerned that pressure from senior officials could distort assessments from the intelligence community. Anthony Ruggiero, the head of the National Security Council which responsible for tracking weapons of mass destruction, expressed frustration during a video conference that C.I.A. was unable to form conclusive answer on the origin of the virus. According to current and former government officials, as of April 30, C.I.A has yet to gather any information beyond circumstantial evidence to bolster the lab theory.[113][114] US intelligence officers suggested that Chinese officials tried to conceal the severity of the outbreak in early days, but no evidence had shown China attempted to cover up a lab accident.[115] One day later, Trump claimed he has evidence of the lab theory, but offers no further details on it.[116][117] Jamie Metzl, a senior fellow at the Atlantic Council, claimed the SARS-CoV-2 virus "likely" came from a Wuhan virology testing laboratory, based on "circumstantial evidence". He was quoted as saying, "I have no definitive way of proving this thesis."[118]

 

On April 30, 2020, the U.S. intelligence and scientific communities issued a public statement dismissing the idea that the virus was not natural, while the investigation of the lab accident theory was ongoing.[119][120] The White House suggested an alternative explanation, along with a seemingly contradictory message, that the virus was man-made. In an interview with ABC News, Secretary of State Pompeo said he has no reason to disbelieve the intelligence community that the virus was natural. However, this contradicted the comment he made earlier in the same interview, in which he said "the best experts so far seem to think it was man-made. I have no reason to disbelieve that at this point."[121][122][123] On May 4, Australian tabloid The Daily Telegraph claimed a reportedly leaked dossier from Five Eyes, which alleged the probable outbreak was from the Wuhan lab.[124] Fox News and national security commentators in the US quickly followed up The Telegraph story,[125][126] rising the tension within international intelligence community.[127] Australian government, which is part of the Five Eyes nations, determined the leaked dossier was not a Five Eyes document, but a compilation of open-source materials that contained no information generated by intelligence gathering.[128] German intelligence community denied the claim of the leaked dossier, instead supported the probability of a natural cause.[129][130] Australian government sees the promotion of the lab theory from the United States counterproductive to Australia’s push for a more broad international-supported independent inquiry into the virus origins.[127] Senior officials in Australian government speculated the dossier was leaked by US embassy in Canberra to promote a narrative in Australia media that diverged from the mainstream belief of Australia.[127][128][125]

 

Beijing rejected the White House's claim, calling the claim "part of an election year strategy by President Donald Trump’s Republican Party".[131] Hua Chunying, Chinese Foreign Ministry spokeswoman, urged Mike Pompeo to present evidence for his claim. "Mr. Pompeo cannot present any evidence because he does not have any," Hua told a journalist during a regular briefing, "This matter should be handled by scientists and professionals instead of politicians out of their domestic political needs."[131][132] The Chinese ambassador, in an opinion published in the Washington Post, called on the White House to end the "blame game" over the coronavirus.[133][134] As of May 5, assessments and internal sources from the Five Eyes nations indicated that the coronavirus outbreak was the result of a laboratory accident was "highly unlikely", since the human infection was "highly likely" a result of natural human and animal interaction. However, to reach such a conclusion with total certainty would still require greater cooperation and transparency from the Chinese side.[135]

 

Anti-Israeli and antisemitic

Further information: Antisemitic canard

Iran's Press TV asserted that "Zionist elements developed a deadlier strain of coronavirus against Iran".[14] Similarly, various Arab media outlets accused Israel and the United States of creating and spreading COVID-19, avian flu, and SARS.[136] Users on social media offered a variety of theories, including the supposition that Jews had manufactured COVID-19 to precipitate a global stock market collapse and thereby profit via insider trading,[137] while a guest on Turkish television posited a more ambitious scenario in which Jews and Zionists had created COVID-19, avian flu, and Crimean–Congo hemorrhagic fever to "design the world, seize countries, [and] neuter the world's population".[138]

 

Israeli attempts to develop a COVID-19 vaccine prompted mixed reactions. Grand Ayatollah Naser Makarem Shirazi denied initial reports that he had ruled that a Zionist-made vaccine would be halal,[139] and one Press TV journalist tweeted that "I'd rather take my chances with the virus than consume an Israeli vaccine".[140] A columnist for the Turkish Yeni Akit asserted that such a vaccine could be a ruse to carry out mass sterilization.[141]

 

An alert by the U.S. Federal Bureau of Investigation regarding the possible threat of far-right extremists intentionally spreading the coronavirus mentioned blame being assigned to Jews and Jewish leaders for causing the pandemic and several statewide shutdowns.[142]

 

Anti-Muslim

Further information: 2020 Tablighi Jamaat coronavirus hotspot in Delhi

In India, Muslims have been blamed for spreading infection following the emergence of cases linked to a Tablighi Jamaat religious gathering.[143] There are reports of vilification of Muslims on social media and attacks on individuals in India.[144] Claims have been made Muslims are selling food contaminated with coronavirus and that a mosque in Patna was sheltering people from Italy and Iran.[145] These claims were shown to be false.[146] In the UK, there are reports of far-right groups blaming Muslims for the coronavirus outbreak and falsely claiming that mosques remained open after the national ban on large gatherings.[147]

 

Bioengineered virus

It has been repeatedly claimed that the virus was deliberately created by humans.

 

Nature Medicine published an article arguing against the conspiracy theory that the virus was created artificially. The high-affinity binding of its peplomers to human angiotensin-converting enzyme 2 (ACE2) was shown to be "most likely the result of natural selection on a human or human-like ACE2 that permits another optimal binding solution to arise".[148] In case of genetic manipulation, one of the several reverse-genetic systems for betacoronaviruses would probably have been used, while the genetic data irrefutably showed that the virus is not derived from a previously used virus template.[148] The overall molecular structure of the virus was found to be distinct from the known coronaviruses and most closely resembles that of viruses of bats and pangolins that were little studied and never known to harm humans.[149]

 

In February 2020, the Financial Times quoted virus expert and global co-lead coronavirus investigator Trevor Bedford: "There is no evidence whatsoever of genetic engineering that we can find", and "The evidence we have is that the mutations [in the virus] are completely consistent with natural evolution".[150] Bedford further explained, "The most likely scenario, based on genetic analysis, was that the virus was transmitted by a bat to another mammal between 20–70 years ago. This intermediary animal—not yet identified—passed it on to its first human host in the city of Wuhan in late November or early December 2019".[150]

 

On February 19, 2020, The Lancet published a letter of a group of scientists condemning "conspiracy theories suggesting that COVID-19 does not have a natural origin".[151]

 

Chinese biological weapon

India

Amidst a rise in Sinophobia, there have been conspiracy theories reported on India's social networks that the virus is "a bioweapon that went rogue" and also fake videos alleging that Chinese authorities are killing citizens to prevent its spread.[152]

 

Ukraine

According to the Kyiv Post, two common conspiracy theories online in Ukraine are that American author Dean Koontz predicted the pandemic in his 1981 novel The Eyes of Darkness, and that the coronavirus is a bioweapon leaked from a secret lab in Wuhan.[153]

 

United Kingdom

 

Tobias Ellwood said, "It would be irresponsible to suggest the source of this outbreak was an error in a Chinese military biological weapons programme ... But without greater Chinese transparency we cannot entirely completely sure."[154]

In February, Conservative MP Tobias Ellwood, chair of the Defence Select Committee of the UK House of Commons, publicly questioned the role of the Chinese Army's Wuhan Institute for Biological Products and called for the "greater transparency over the origins of the coronavirus".[154][non-primary source needed] The Daily Mail reported in early April 2020 that a member of COBRA (an ad-hoc government committee tasked with advising on crises[citation needed]) has stated while government intelligence does not dispute that the virus has a zoonotic origin, it also does not discount the idea of a leak from a Wuhan laboratory, saying "Perhaps it is no coincidence that there is that laboratory in Wuhan"; the Asia Times reported the story as if it were factual,[155] perhaps unaware of the reputation of the Daily Mail.

 

United States

Further information: Cyberwarfare in the United States and Propaganda in the United States

In January 2020, BBC News published an article about coronavirus misinformation, citing two January 24 articles from The Washington Times that said the virus was part of a Chinese biological weapons program, based at the Wuhan Institute of Virology (WIV).[1] The Washington Post later published an article debunking the conspiracy theory, citing U.S. experts who explained why the WIV was unsuitable for bioweapon research, that most countries had abandoned bioweapons as fruitless, and that there was no evidence the virus was genetically engineered.[156]

 

On January 29, financial news website and blog ZeroHedge suggested without evidence that a scientist at the WIV created the COVID-19 strain responsible for the coronavirus outbreak. Zerohedge listed the full contact details of the scientist supposedly responsible, a practice known as doxing, by including the scientist's name, photo, and phone number, suggesting to readers that they "pay [the Chinese scientist] a visit" if they wanted to know "what really caused the coronavirus pandemic".[157] Twitter later permanently suspended the blog's account for violating its platform-manipulation policy.[158]

  

Logo of the fictional Umbrella Corporation, which some internet rumours linked to the pandemic. The corporation was invented for the Resident Evil game series.

In January 2020, Buzzfeed News reported on an internet meme of a link between the logo of the WIV and "Umbrella Corporation", the agency that created the virus responsible for a zombie apocalypse in the Resident Evil franchise. Posts online noted that "Racoon [sic]" (the main city in Resident Evil) was an anagram of "Corona".[159] Snopes noted that the logo was not from the WIV, but a company named Shanghai Ruilan Bao Hu San Biotech Ltd (located some 500 miles (800 km) away in Shanghai), and that the correct name of the city in Resident Evil was "Raccoon City".[159]

 

In February 2020, U.S. Senator Tom Cotton (R-AR) suggested the virus may have originated in a Chinese bioweapon laboratory.[160] Francis Boyle, a law professor, also expressed support for the bioweapon theory suggesting it was the result of unintended leaks.[161] Cotton elaborated on Twitter that his opinion was only one of "at least four hypotheses". Multiple medical experts have indicated there is no evidence for these claims.[162] Conservative political commentator Rush Limbaugh said on The Rush Limbaugh Show—the most popular radio show in the U.S.—that the virus was probably "a ChiCom laboratory experiment" and the Chinese government was using the virus and the media hysteria surrounding it to bring down Donald Trump.[163][164]

 

On February 6, the White House asked scientists and medical researchers to rapidly investigate the origins of the virus both to address the current spread and "to inform future outbreak preparation and better understand animal/human and environmental transmission aspects of coronaviruses".[165] American magazine Foreign Policy said Xi Jinping's "political agenda may turn out to be a root cause of the epidemic" and that his Belt and Road Initiative has "made it possible for a local disease to become a global menace".[90]

 

The Inverse reported that "Christopher Bouzy, the founder of Bot Sentinel, conducted a Twitter analysis for Inverse and found [online] bots and trollbots are making an array of false claims. These bots are claiming China intentionally created the virus, that it's a biological weapon, that Democrats are overstating the threat to hurt Donald Trump and more. While we can't confirm the origin of these bots, they are decidedly pro-Trump."[166]

 

Conservative commentator Josh Bernstein claimed that the Democratic Party and the "medical deep state" were collaborating with the Chinese government to create and release the coronavirus to bring down Donald Trump. Bernstein went on to suggest those responsible should be locked in a room with infected coronavirus patients as punishment.[167][168]

 

Jerry Falwell Jr., the president of Liberty University, promoted a conspiracy theory on Fox News that North Korea and China conspired together to create the coronavirus.[169] He also said people were overreacting to the coronavirus outbreak and that Democrats were trying to use the situation to harm President Trump.[170]

 

Hospital ship attack

The hospital ship USNS Mercy (T-AH-19) deployed to the Port of Los Angeles to provide backup medical services for the region. On March 31, 2020, a Pacific Harbor Line freight train was deliberately derailed by its onboard engineer in an attempt to crash into the ship, but the attack was unsuccessful and no one was injured.[171][172] According to U.S. federal prosecutors, the train's engineer "[...] was suspicious of the Mercy, believing it had an alternate purpose related to COVID-19 or a government takeover".[173]

 

Population control scheme

See also: List of conspiracy theories § RFID chips

According to the BBC, Jordan Sather, a conspiracy theory YouTuber supporting the far-right QAnon conspiracy theory and the anti-vax movement, has falsely claimed the outbreak was a population control scheme created by Pirbright Institute in England and by former Microsoft CEO Bill Gates. This belief is held mostly by right-wing libertarians, NWO conspiracy theorists, and Christian Fundamentalists.[1][174]

 

Spy operation

Some people have alleged that the coronavirus was stolen from a Canadian virus research lab by Chinese scientists. Health Canada and the Public Health Agency of Canada said that conspiracy theory had "no factual basis".[175] The stories seem to have been derived[176] from a July 2019 news article[177] stating that some Chinese researchers had their security access to a Canadian Level 4 virology facility revoked in a federal police investigation; Canadian officials described this as an administrative matter and "there is absolutely no risk to the Canadian public."[177]

 

This article was published by the Canadian Broadcasting Corporation (CBC);[176] responding to the conspiracy theories, the CBC later stated that "CBC reporting never claimed the two scientists were spies, or that they brought any version of the coronavirus to the lab in Wuhan". While pathogen samples were transferred from the lab in Winnipeg, Canada to Beijing, China, on March 31, 2019, neither of the samples was a coronavirus, the Public Health Agency of Canada says the shipment conformed to all federal policies, and there has not been any statement that the researchers under investigation were responsible for sending the shipment. The current location of the researchers under investigation by the Royal Canadian Mounted Police is not being released.[175][178][179]

 

In the midst of the coronavirus epidemic, a senior research associate and expert in biological warfare with the Begin-Sadat Center for Strategic Studies, referring to a NATO press conference, identified suspicions of espionage as the reason behind the expulsions from the lab, but made no suggestion that coronavirus was taken from the Canadian lab or that it is the result of bioweapons defense research in China.[180]

 

U.S. biological weapon

Arab world

According to Washington DC-based nonprofit Middle East Media Research Institute, numerous writers in the Arabic press have promoted the conspiracy theory that COVID-19, as well as SARS and the swine flu virus, were deliberately created and spread to sell vaccines against these diseases, and it is "part of an economic and psychological war waged by the U.S. against China with the aim of weakening it and presenting it as a backward country and a source of diseases".[181] Iraqi political analyst Sabah Al-Akili on Al-Etejah TV, Saudi daily Al-Watan writer Sa'ud Al-Shehry, Syrian daily Al-Thawra columnist Hussein Saqer, and Egyptian journalist Ahmad Rif'at on Egyptian news website Vetogate, were some examples given by MEMRI as propagators of the U.S. biowarfare conspiracy theory in the Arabic world.[181]

 

China

Further information: Cyberwarfare by China, Propaganda in China, and Chinese information operations and information warfare

 

The Xinhua News Agency is among the news outlets that have published false information about COVID-19's origins.

According to London-based The Economist, plenty of conspiracy theories exist on China's internet about COVID-19 being the CIA's creation to keep China down.[182] NBC News however has noted that there have also been debunking efforts of U.S.-related conspiracy theories posted online, with a WeChat search of "Coronavirus is from the U.S." reported to mostly yield articles explaining why such claims are unreasonable.[183] According to an investigation by ProPublica, such conspiracy theories and disinformation have been propagated under the direction of China News Service, the country's second largest government-owned media outlet controlled by the United Front Work Department.[184] Global Times and Xinhua News Agency have similarly been implicated in propagating disinformation related to COVID-19's origins.[185][186]

 

Multiple conspiracy articles in Chinese from the SARS era resurfaced during the outbreak with altered details, claiming SARS is biological warfare. Some said BGI Group from China sold genetic information of the Chinese people to the U.S., which then specifically targeted the genome of Chinese individuals.[187]

 

On January 26, Chinese military enthusiast website Xilu published an article, claimed how the U.S. artificially combined the virus to "precisely target Chinese people".[188][189] The article was removed in early February. The article was further distorted on social media in Taiwan, which claimed "Top Chinese military website admitted novel coronavirus was Chinese-made bio-weapons".[190] Taiwan Fact-check center debunked the original article and its divergence, suggesting the original Xilu article distorted the conclusion from a legitimate research on Chinese scientific magazine Science China Life Sciences, which never mentioned the virus was engineered.[190] The fact-check center explained Xilu is a military enthusiastic tabloid established by a private company, thus it doesn't represent the voice of Chinese military.[190]

 

Some articles on popular sites in China have also cast suspicion on U.S. military athletes participating in the Wuhan 2019 Military World Games, which lasted until the end of October 2019, and have suggested they deployed the virus. They claim the inattentive attitude and disproportionately below-average results of American athletes in the games indicate they might have been there for other purposes and they might actually be bio-warfare operatives. Such posts stated that their place of residence during their stay in Wuhan was also close to the Huanan Seafood Wholesale Market, where the first known cluster of cases occurred.[191]

 

In March 2020, this conspiracy theory was endorsed by Zhao Lijian, a spokesperson from the Ministry of Foreign Affairs of the People's Republic of China.[192][193][194][195] On March 13, the U.S. government summoned Chinese Ambassador Cui Tiankai to Washington over the coronavirus conspiracy theory.[196] Over the next month, conspiracy theorists narrowed their focus to one U.S. Army Reservist, a woman who participated in the games in Wuhan as a cyclist, claiming she is "patient zero". According to a CNN report, these theories have been spread by George Webb, who has nearly 100,000 followers on YouTube, and have been amplified by a report by CPC-owned newspaper Global Times.[197][198]

 

Iran

Further information: Propaganda in Iran

 

Reza Malekzadeh, deputy health minister, rejected bioterrorism theories.

According to Radio Farda, Iranian cleric Seyyed Mohammad Saeedi accused U.S. President Donald Trump of targeting Qom with coronavirus "to damage its culture and honor". Saeedi claimed that Trump is fulfilling his promise to hit Iranian cultural sites, if Iranians took revenge for the airstrike that killed of Quds Force Commander Qasem Soleimani.[199]

 

Iranian TV personality Ali Akbar Raefipour claimed the coronavirus was part of a "hybrid warfare" programme waged by the United States on Iran and China.[200] Brigadier General Gholam Reza Jalali, head of Iranian Civil Defense Organization, claimed the coronavirus is likely a biological attack on China and Iran with economic goals.[201][202]

 

Hossein Salami, the head of Islamic Revolutionary Guard Corps (IRGC), claimed the coronavirus outbreak in Iran may be due to a U.S. "biological attack".[203] Several Iranian politicians, including Hossein Amir-Abdollahian, Rasoul Falahati, Alireza Panahian, Abolfazl Hasanbeigi and Gholamali Jafarzadeh Imanabadi, also made similar remarks.[204] Iranian Supreme Leader, the Ayatollah Ali Khamenei, made similar suggestions.[205]

 

Former Iranian president Mahmoud Ahmadinejad sent a letter to the United Nations on March 9, claiming that "it is clear to the world that the mutated coronavirus was produced in lab" and that COVID-19 is "a new weapon for establishing and/or maintaining political and economic upper hand in the global arena".[206]

 

The late[207] Ayatollah Hashem Bathaie Golpayegani claimed that "America is the source of coronavirus, because America went head to head with China and realised it cannot keep up with it economically or militarily."[208]

 

Reza Malekzadeh, Iran's deputy health minister and former Minister of Health, rejected claims that the virus was a biological weapon, pointing out that the U.S. would be suffering heavily from it. He said Iran was hard-hit because its close ties to China and reluctance to cut air ties introduced the virus, and because early cases had been mistaken for influenza.[205]

 

Philippines

 

In the Philippine Senate, Tito Sotto has promoted his belief that COVID-19 is a bioweapon.

A Filipino Senator, Tito Sotto, played a bioweapon conspiracy video in a February 2020 Senate hearing, suggesting the coronavirus is biowarfare waged against China.[209][210]

 

Russia

Further information: Cyberwarfare by Russia and Propaganda in the Russian Federation

On February 22, U.S. officials alleged that Russia is behind an ongoing disinformation campaign, using thousands of social media accounts on Twitter, Facebook and Instagram to deliberately promote unfounded conspiracy theories, claiming the virus is a biological weapon manufactured by the CIA and the U.S. is waging economic war on China using the virus.[211][12][212] The acting assistant secretary of state for Europe and Eurasia, Philip Reeker, said "Russia's intent is to sow discord and undermine U.S. institutions and alliances from within" and "by spreading disinformation about coronavirus, Russian malign actors are once again choosing to threaten public safety by distracting from the global health response."[211] Russia denies the allegation, saying "this is a deliberately false story".[213]

 

According to U.S.-based The National Interest magazine, although official Russian channels had been muted on pushing the U.S. biowarfare conspiracy theory, other Russian media elements do not share the Kremlin's restraint.[214] Zvezda, a news outlet funded by the Russian Defense Ministry, published an article titled "Coronavirus: American biological warfare against Russia and China", claiming that the virus is intended to damage the Chinese economy, weakening its hand in the next round of trade negotiations.[214] Ultra-nationalist politician and leader of the Liberal Democratic Party of Russia, Vladimir Zhirinovsky, claimed on a Moscow radio station that the virus was an experiment by the Pentagon and pharmaceutical companies. Politician Igor Nikulin made rounds on Russian television and news media, arguing that Wuhan was chosen for the attack because the presence of a BSL-4 virus lab provided a cover story for the Pentagon and CIA about a Chinese bio-experiment leak.[214] An EU-document claims 80 attempts by Russian media to spread disinformation related to the epidemic.[215]

 

According to the East StratCom Task Force, the Sputnik news agency was active publishing stories speculating that the virus could've been invented in Latvia, that it was used by Communist Party of China to curb protests in Hong Kong, that it was introduced intentionally to reduce the number of elder people in Italy, that it was targeted against the Yellow Vests movement, and making many other speculations. Sputnik branches in countries including Armenia, Belarus, Spain, and in the Middle East came up with versions of these stories.[216]

 

Venezuela

Constituent Assembly member Elvis Méndez declared that the coronavirus was a "bacteriological sickness created in '89, in '90 and historically" and that it was a sickness "inoculated by the gringos". Méndez theorized that the virus was a weapon against Latin America and China and that its purpose was "to demoralize the person, to weaken to install their system".[217]

 

COVID-19 recovery

It has been wrongly claimed that anyone infected with COVID-19 will have the virus in their bodies for life. While there is no curative treatment, infected individuals can recover from the disease, eliminating the virus from their bodies; getting supportive medical care early can help.[279]

 

COVID-19 xenophobic blaming by ethnicity and religion

Main article: List of incidents of xenophobia and racism related to the 2019–20 coronavirus pandemic

File:IOM - Fighting Stigma and Discrimination against Migrants during COVID-19.webm

UN video warns that misinformation against groups may lower testing rates and increase transmission.

COVID-19-related xenophobic attacks have been made against people the attacker blamed for COVID-19 on the basis of their ethnicity. People who are considered to look Chinese have been subjected to COVID-19-related verbal and physical attacks in many other countries, often by people accusing them of transmitting the virus.[281][282][283] Within China, there has been discrimination (such as evictions and non-service in shops) against people from anywhere closer to Wuhan (where the pandemic started) and against anyone perceived as being non-Chinese (especially those considered African), as the Chinese government has blamed continuing cases on re-introductions of the virus from abroad (90% of reintroduced cases were by Chinese passport-holders). Neighbouring countries have also discriminated against people seen as Westerners.[284][285][286] People have also simply blamed other local groups along the lines of pre-existing social tensions and divisions, sometimes citing reporting of COVID-19 cases within that group. For instance, Muslims have been widely blamed, shunned, and discriminated against in India (including some violent attacks), amid unfounded claims that Muslims are deliberately spreading COVID-19, and a Muslim event at which the disease did spread has received far more public attention than many similar events run by other groups and the government.[287] White supremacist groups have blamed COVID-19 on non-whites and advocated deliberately infecting minorities they dislike, such as Jews.[288]

 

False causes

5G

 

5G towers have been burned by people wrongly blaming them for COVID-19.

 

Openreach engineers appealed on anti-5G Facebook groups, saying they aren't involved in mobile networks, and workplace abuse is making it difficult for them to maintain phonelines and broadband.

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In February 2020 BBC News reported that conspiracy theorists on social media groups alleged a link between coronavirus and 5G mobile networks, claiming that Wuhan and Diamond Princess outbreaks were directly caused by electromagnetic fields and by the introduction of 5G and wireless technologies. Some conspiracy theorists also alleged that the coronavirus outbreak was a cover-up for a 5G-related illness.[33] In March 2020, Thomas Cowan, a holistic medical practitioner who trained as a physician and operates on probation with Medical Board of California, alleged that coronavirus is caused by 5G, based on the claims that African countries were not affected significantly by the pandemic and Africa was not a 5G region.[289][290] Cowan also falsely alleged that the viruses were wastes from cells that are poisoned by electromagnetic fields and historical viral pandemics coincided with the major developments in radio technology.[290] The video of his claims went viral and was recirculated by celebrities including Woody Harrelson, John Cusack, and singer Keri Hilson.[291] The claims may also have been recirculated by an alleged "coordinated disinformation campaign", similar to campaigns used by the Internet Research Agency in Saint Petersburg, Russia.[292] The claims were criticized on social media and debunked by Reuters,[293] USA Today,[294] Full Fact[295] and American Public Health Association executive director Georges C. Benjamin.[289][296]

 

Professor Steve Powis, national medical director of NHS England, described theories linking 5G mobile phone networks to COVID-19 as the "worst kind of fake news".[297] Viruses cannot be transmitted by radio waves. COVID-19 has spread and continues to spread in many countries that do not have 5G networks.[279]

 

After telecommunications masts in several parts of the United Kingdom were the subject of arson attacks, British Cabinet Office Minister Michael Gove said the theory that COVID-19 virus may be spread by 5G wireless communication is "just nonsense, dangerous nonsense as well".[298] Vodafone announced that two Vodafone masts and two it shares with O2 had been targeted.[299][300]

 

By Monday April 6, 2020 at least 20 mobile phone masts in the UK had been vandalised since the previous Thursday.[301] Because of slow rollout of 5G in the UK, many of the damaged masts had only 3G and 4G equipment.[301] Mobile phone and home broadband operators estimated there were at least 30 incidents of confronting engineers maintaining equipment in the week up to April 6.[301] There have been eleven incidents of attempted arson at mobile phone masts in the Netherlands, including one case where "Fuck 5G" was written, as well as in Ireland and Cyprus.[302][303] Facebook has deleted multiple messages encouraging attacks on 5G equipment.[301]

 

Engineers working for Openreach posted pleas on anti-5G Facebook groups asking to be spared abuse as they are not involved with maintaining mobile networks.[304] Mobile UK said the incidents were affecting attempts to maintain networks that support home working and provide critical connections to vulnerable customers, emergency services and hospitals.[304] A widely circulated video shows people working for broadband company Community Fibre being abused by a woman who accuses them of installing 5G as part of a plan to kill the population.[304]

 

YouTube announced that it would reduce the amount of content claiming links between 5G and coronavirus.[299] Videos that are conspiratorial about 5G that do not mention coronavirus would not be removed, though they might be considered "borderline content", removed from search recommendations and losing advertising revenue.[299] The discredited claims had been circulated by British conspiracy theorist David Icke in videos (subsequently removed) on YouTube and Vimeo, and an interview by London Live TV network, prompting calls for action by Ofcom.[305][306]

 

On April 13, 2020, Gardaí were investigating fires at 5G masts in County Donegal, Ireland.[307] Gardaí and fire services had attended the fires the previous night in an attempt to put them out.[307] Although Gardaí were awaiting results of tests they were treating the fires as deliberate.[307]

 

There were 20 suspected arson attacks on phone masts in the UK over the Easter 2020 weekend.[297] These included an incident in Dagenham where three men were arrested on suspicion of arson, a fire in Huddersfield that affected a mast used by emergency services and a fire in a mast that provides mobile connectivity to the NHS Nightingale Hospital Birmingham.[297]

 

Ofcom issued guidance to ITV following comments by Eamonn Holmes after comments made by Holmes about 5G and coronavirus on This Morning.[308] Ofcom said the comments were "ambiguous" and "ill-judged" and they "risked undermining viewers' trust in advice from public authorities and scientific evidence".[308] Ofcom also local channel London Live in breach of standards for an interview it had with David Icke who it said had " expressed views which had the potential to cause significant harm to viewers in London during the pandemic".[308]

 

Some telecoms engineers have reported threats of violence, including threats to stab and murder them, by individuals who believe them to be working on 5G networks.[309] West Midlands Police said the crimes in question are being taken very seriously.[309]

 

On April 24, 2020 The Guardian revealed that an evangelical pastor from Luton had provided the male voice on a recording blaming 5G for deaths caused by coronavirus.[310] Jonathon James claimed to have formerly headed the largest business-unit at Vodafone, but insiders at the company said that he was hired for a sales position in 2014 when 5G was not a priority for the company and that 5G would not have been part of his job.[310] He left the company after less than a year.[310]

 

Mosquitoes

It has been claimed that mosquitoes transmit coronavirus. There is no evidence that this is true; coronavirus spreads through small droplets of saliva and mucus.[279]

 

Petrol pumps

A warning claiming to be from the Australia Department of Health said coronavirus spreads through petrol pumps and that everyone should wear gloves when filling up petrol in their cars.[311]

 

Shoe-wearing

There were claims that wearing shoes at one's home was the reason behind the spread of the coronavirus in Italy.[312]

 

Resistance/susceptibility based on ethnicity

There have been claims that specific ethnicities are more or less vulnerable to COVID-19. COVID-19 is a new zoonotic disease, so no population has yet had the time to develop population immunity.[medical citation needed]

 

Beginning on February 11, reports, quickly spread via Facebook, implied that a Cameroonian student in China had been completely cured of the virus due to his African genetics. While a student was successfully treated, other media sources have noted that no evidence implies Africans are more resistant to the virus and labeled such claims as false information.[313] Kenyan Secretary of Health Mutahi Kagwe explicitly refuted rumors that "those with black skin cannot get coronavirus", while announcing Kenya's first case on March 13.[314] This myth was cited as a contributing factor in the disproportionately high rates of infection and death observed among African Americans.[315][316]

 

There have been claims of "Indian immunity": that the people of India have more immunity to the COVID-19 virus due to living conditions in India. This idea was deemed "absolute drivel" by Anand Krishnan, professor at the Centre for Community Medicine of the All India Institute of Medical Sciences (AIIMS). He said there was no population immunity to the COVID-19 virus yet, as it is new, and it is not even clear whether people who have recovered from COVID-19 will have lasting immunity, as this happens with some viruses but not with others.[317]

 

Iran's Supreme Leader Ayatollah Ali Khamenei claimed the virus was genetically targeted at Iranians by the U.S., and this is why it is seriously affecting Iran. He did not offer any evidence.[318][22]

 

Religious protection

A number of religious groups have claimed protection due to their faith, some refusing to stop large religious gatherings. In Israel, some Ultra-Orthodox Jews initially refused to close synagogues and religious seminaries and disregarded government restrictions because "The Torah protects and saves",[319] which resulted in an 8 times faster rate of infection among some groups.[320] The Tablighi Jamaat movement organised mass gatherings in Malaysia, India, and Pakistan whose participants believed that God will protect them resulted the biggest rise in COVID-19 cases in a number of countries.[321][29][322] In Iran, the head of Fatima Masumeh Shrine encouraged pilgrims to visit the shrine despite calls to close the shrine, saying that they "consider this holy shrine to be a place of healing."[323] In South Korea the River of Grace Community Church in Gyeonggi Province spread the virus after spraying salt water into their members' mouths in the belief that it would kill the virus,[324] while the Shincheonji Church of Jesus in Daegu where a church leader claimed that no Shincheonji worshipers had caught the virus in February while hundreds died in Wuhan later caused in the biggest spread of the virus in the country.[325][326]

 

In Somalia, myths have spread claiming Muslims are immune to the virus.[327]

 

Unproven protective and aggravating factors

Vegetarian immunity

[icon]

This section needs expansion. You can help by adding to it. (April 2020)

Claims that vegetarians are immune to coronavirus spread online in India, causing "#NoMeat_NoCoronaVirus" to trend on Twitter.[328][better source needed] Eating meat does not have an effect on COVID-19 spread, except for people near where animals are slaughtered, said Anand Krishnan.[329] Fisheries, Dairying and Animal Husbandry Minister Giriraj Singh said the rumour had significantly affected industry, with the price of a chicken falling to a third of pre-pandemic levels. He also described efforts to improve the hygiene of the meat supply chain.[330]

 

Efficacy of hand sanitiser, "antibacterial" soaps

 

Washing in soap and water for at least 20 seconds is the best way to clean hands. Second-best is a hand sanitizer that is at least 60% alcohol.[331]

Claims that hand sanitiser is merely "antibacterial not antiviral", and therefore ineffective against COVID-19, have spread widely on Twitter and other social networks. While the effectiveness of sanitiser depends on the specific ingredients, most hand sanitiser sold commercially inactivates SARS-CoV-2, which causes COVID-19.[332][333] Hand sanitizer is recommended against COVID-19,[279] though unlike soap, it is not effective against all types of germs.[334] Washing in soap and water for at least 20 seconds is recommended by the U.S. Centers for Disease Control (CDC) as the best way to clean hands in most situations. However, if soap and water are not available, a hand sanitizer that is at least 60% alcohol can be used instead, unless hands are visibly dirty or greasy.[331][335] The CDC and the Food and Drug Administration both recommend plain soap; there is no evidence that "antibacterial soaps" are any better, and limited evidence that they might be worse long-term.[336][337]

 

Alcohol (ethanol and poisonous methanol)

Contrary to some reports, drinking alcohol does not protect against COVID-19, and can increase health risks[279] (short term and long term). Drinking alcohol is ethanol; other alcohols, such as methanol, which causes methanol poisoning, are acutely poisonous, and may be present in badly-prepared alcoholic beverages.[338]

 

Iran has reported incidents of methanol poisoning, caused by the false belief that drinking alcohol would cure or protect against coronavirus;[339] alcohol is banned in Iran, and bootleg alcohol may contain methanol.[340] According to Iranian media in March 2020, nearly 300 people have died and more than a thousand have become ill due to methanol poisoning, while Associated Press gave figures of around 480 deaths with 2,850 others affected.[341] The number of deaths due to methanol poisoning in Iran reached over 700 by April.[342] Iranian social media had circulated a story from British tabloids that a British man and others had been cured of coronavirus with whiskey and honey,[339][343] which combined with the use of alcohol-based hand sanitizers as disinfectants, led to the false belief that drinking high-proof alcohol can kill the virus.[339][340][341]

 

Similar incidents have occurred in Turkey, with 30 Turkmenistan citizens dying from methanol poisoning related to coronavirus cure claims.[344][345]

 

In Kenya, the Governor of Nairobi Mike Sonko has come under scrutiny for including small bottles of the cognac Hennessy in care packages, falsely claiming that alcohol serves as "throat sanitizer" and that, from research, it is believed that "alcohol plays a major role in killing the coronavirus."[346][347]

 

Cocaine

Cocaine does not protect against COVID-19. Several viral tweets purporting that snorting cocaine would sterilize one's nostrils of the coronavirus spread around Europe and Africa. In response, the French Ministry of Health released a public service announcement debunking this claim, saying "No, cocaine does NOT protect against COVID-19. It is an addictive drug that causes serious side effects and is harmful to people's health." The World Health Organisation also debunked the claim.[348]

 

Ibuprofen

A tweet from French health minister Olivier Véran, a bulletin from the French health ministry, and a small speculative study in The Lancet Respiratory Medicine raised concerns about ibuprofen worsening COVID-19, which spread extensively on social media. The European Medicines Agency[349] and the World Health Organization recommended COVID-19 patients keep taking ibuprofen as directed, citing lack of convincing evidence of any danger.[350]

 

Helicopter spraying

In some Asian countries, it has been claimed that one should stay at home on particular days when helicopters spray disinfectant over homes for killing off COVID-19; no such spraying is taking place.[351][352]

 

Cruise ships safety from infection

Main article: COVID-19 pandemic on cruise ships

 

Claims by cruise-ship operators notwithstanding, there are many cases of coronaviruses in hot climates; some countries in the Caribbean, the Mediterranean, and the Persian Gulf are severely affected.

In March 2020, the Miami New Times reported that managers at Norwegian Cruise Line had prepared a set of responses intended to convince wary customers to book cruises, including "blatantly false" claims that the coronavirus "can only survive in cold temperatures, so the Caribbean is a fantastic choice for your next cruise", that "[s]cientists and medical professionals have confirmed that the warm weather of the spring will be the end of the [c]oronavirus", and that the virus "cannot live in the amazingly warm and tropical temperatures that your cruise will be sailing to".[353]

 

Flu is seasonal (becoming less frequent in the summer) in some countries, but not in others. While it is possible that the COVID-19 coronavirus will also show some seasonality, it is not yet known.[354][355][356][medical citation needed] The COVID-19 coronavirus spread along international air travel routes, including to tropical locations.[357] Outbreaks on cruise ships, where an older population lives in close quarters, frequently touching surfaces which others have touched, were common.[358][359]

 

It seems that COVID-19 can be transmitted in all climates.[279] It has seriously affected many warm-climate countries. For instance, Dubai, with an year-round average daily high of 28.0 Celsius (82.3°F) and the airport said to have the world's most international traffic, has had thousands of cases.

 

Vaccine pre-existence

It was reported that multiple social media posts have promoted a conspiracy theory claiming the virus was known and that a vaccine was already available. PolitiFact and FactCheck.org noted that no vaccine currently exists for COVID-19. The patents cited by various social media posts reference existing patents for genetic sequences and vaccines for other strains of coronavirus such as the SARS coronavirus.[360][4] The WHO reported as of February 5, 2020, that amid news reports of "breakthrough" drugs being discovered to treat people infected with the virus, there were no known effective treatments;[361] this included antibiotics and herbal remedies not being useful.[362] Scientists are working to develop a vaccine, but as of March 18, 2020, no vaccine candidates have completed Phase II clinical trials.[citation needed]

 

Miscellaneous

Name of the disease

Social media posts and internet memes claimed that COVID-19 means "Chinese Originated Viral Infectious Disease 19", or similar, as supposedly the "19th virus to come out of China".[477] In fact, the WHO named the disease as follows: CO stands for corona, VI for virus, D for disease and 19 for when the outbreak was first identified (31 December 2019).[478]

 

Bat soup

Some media outlets, including Daily Mail and RT, as well as individuals, disseminated a video showing a Chinese woman eating a bat, falsely suggesting it was filmed in Wuhan and connecting it to the outbreak.[479][480] However, the widely circulated video contains unrelated footage of a Chinese travel vlogger, Wang Mengyun, eating bat soup in the island country of Palau in 2016.[479][480][481][482] Wang posted an apology on Weibo,[481][482] in which she said she had been abused and threatened,[481] and that she had only wanted to showcase Palauan cuisine.[481][482] The spread of misinformation about bat consumption has been characterized by xenophobic and racist sentiment toward Asians.[90][483][484] In contrast, scientists suggest the virus originated in bats and migrated into an intermediary host animal before infecting people.[90][485]

 

en.wikipedia.org/wiki/Misinformation_related_to_the_COVID...

Hammock Beach | Palm Coast | Florida

 

A 9 hour flight from London to Orlando and a -5 hour timezone difference meant me waking up ridiculously early on a quiet Sunday morning.

 

There's not a lot of photogenic interest in the UCF area (it's mainly all office blocks and a university campus), so I decided to head up the coast in my rental car. I ended up in a quiet little resort called Palm Coast (just over an hour north of Orlando, up the I95 interstate).

 

I parked up and walked onto the beach, noticing this log which caught my eye. I thought it made an unusual piece of foreground interest.

 

I setup with my tripod and mounted a 3-Stop ND Grad Filter in order to retain the amazing colours in the sky just before sunrise. About 15 minutes before the sun came above the horizon, the sky lit up like it was on fire, prompting me to name this image 'Inferno".

 

I have tried to capture how this place felt when I was stood there on the beach, the sound of crashing waves and the smell of the sea breeze. I chose a 1-second exposure to illustrate the movement in the waves and I did not add any extra saturation or contrast, the sky really was that bright!

 

Google Maps

What if there was no life? Nothing wrong, nothing right.

What if there was no time, and no reason, or rhyme?

What if you should decide that you don't want me there by your side?

 

I've started secondary school! It's senior year 1, and all professors are saying we'll have tons of HW, and this, and that...

Arg. Can't stand it anymore, and I haven't been there for a WEEK.

 

I have some ideas I'd like to execute, again, but i never get to. Just like the last time. Guess I have to start focusing on this a little more. I nagivate through Flickr more than I check my own boat.

Ok, don't know were that came from. Woah, I'm such a poet (?

Nevermind. I've seen Adele's concert at Royal Albert Hall. She is such an impressive woman. And funny, too! I haven't seen many like her who say the F word normally. Love her laugh :)

 

SOOOO, don't know what to write! Am, just knw there'll be better pics soon, and, well, happy March 4th :)

 

Do not publish my photos without my permission.