View allAll Photos Tagged neuron
1998 frame built with Columbus Neuron, fully chromed, lovely lugwork. Unfortunately paint is in very poor condition, lots of rust spots on the chainstays and inside the fork crown and legs. Awaiting full restoration including new chrome and wet paint. Will be dressed in mid 90's Campagnolo Chorus.
Original auction photos.
a close up (width of photo = roughly .0001m) of young neurons born recently in the adult mouse brain. the blue is NeuN, a protein found only in neurons (and is therefore good for identifying them) and the red/pink is doublecortin, a protein found only in young neurons. doublecortin is needed for neuronal processes to be able to grow and contact other neurons. when the neurons mature and have formed all their connections they no longer need doublecortin. you can see that all the neurons but those at the left-most edge are therefore mature.
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Frederick “Rusty” Gage has spent his life asking a question many neuroscientists once considered heretical: can the adult brain grow new neurons?
When I photographed him at the Salk Institute in February 2026, that question felt less like rebellion and more like legacy. We made portraits in his study overlooking the Pacific, a quiet room washed in coastal light. The ocean moved below the cliffs in long, steady breaths. It is the same office once occupied by Jonas Salk, who recruited Rusty decades ago. The desk, the view, the gravity of the place. You feel it immediately. History is not abstract there. It presses in from the walls.
In the late twentieth century, neuroscience was built on a stark premise: you are born with a fixed number of neurons. Damage them and they are gone. Memory fades. Injury lingers. Aging narrows possibility. Rusty challenged that dogma with careful, methodical experiments that showed new neurons could, in fact, form in the adult hippocampus. The implications were enormous. Learning, mood, resilience, even the biology of hope took on new dimensions.
In person, what strikes you first is his attentiveness. He leans in slightly when you speak, hands folded, eyes steady behind round glasses. There is warmth in him that feels unforced. Soft spoken, yes, but never distant. You sense a mind that is constantly mapping connections, not only between neurons but between people. Students drift in and out of his orbit with ease. Colleagues seek him out. He listens more than he declares.
The study itself holds layers of meaning. Jonas Salk built the institute as a place where scientists could think expansively, where architecture and intellect met the horizon. Standing in that room with Rusty, you understand that recruitment was more than a hire. It was a passing of trust. Salk had imagined a future for biology that included imagination and risk. Rusty carried that forward into the living brain.
His work has since expanded beyond neurogenesis into how the genome shapes the nervous system over time. His lab explores mosaicism in the brain, the idea that our neurons are not genetically identical but subtly varied. The brain becomes not a static organ but a dynamic landscape, shaped by experience and by the restless choreography of DNA. It is a vision of the self that is fluid and intricate.
Photographing him in that office felt less like documenting a single scientist and more like tracing a lineage. Salk sought a vaccine that would protect children from paralysis. Rusty sought evidence that the adult brain was not condemned to decline. Both projects required a certain stubborn optimism. A belief that the body holds more possibility than we assume.
The weight of history was there, yes. But so was something lighter. A current of curiosity that refuses to settle. In Rusty Gage’s presence, you feel that science is not a monument. It is a conversation, still unfolding, with the ocean as witness.
This group exhibition, including work by Catherine Richards, Michael Snow, Scott Rogers, Thomson & Craighead and Simon Pope, draws on ideas of scientific experimentation, media processing, and time delay. Each work acts to slow down our senses of perception, causing within us an awareness of both time passing and our experience of it. The title refers to that fact that we often watch other people interact with responsive art, and mirror their behaviour, consciously or not.
Catherine Richards’ I was scared to death / I could have died of joy features glass replicas of the brain, which react to your presence with pulses of electromagnetic light. Scott Rogers’ Between Nonesuch Place juxtaposes an actual non-functioning glass object, a ‘self-flowing flask’ with its virtual working counterpart. Thomson & Craighead’s Flipped Clock is a modified digital clock display, where each individual digit is rotated by 180-degrees. Simon Pope’s Recall From Memory the Space of Another Gallery is an invitation for the visitor to recall experiences of being in other gallery spaces from memory. The seminal filmmaker Michael Snow’s WVLNT: Wavelength for those who don't have the time. Originally 45 minutes, Now 15! remixes his own seminal work Wavelength.
Credit
Curated by Sarah Cook. Supported by CRUMB and The University of Sunderland.
This group exhibition, including work by Catherine Richards, Michael Snow, Scott Rogers, Thomson & Craighead and Simon Pope, draws on ideas of scientific experimentation, media processing, and time delay. Each work acts to slow down our senses of perception, causing within us an awareness of both time passing and our experience of it. The title refers to that fact that we often watch other people interact with responsive art, and mirror their behaviour, consciously or not.
Catherine Richards’ I was scared to death / I could have died of joy features glass replicas of the brain, which react to your presence with pulses of electromagnetic light. Scott Rogers’ Between Nonesuch Place juxtaposes an actual non-functioning glass object, a ‘self-flowing flask’ with its virtual working counterpart. Thomson & Craighead’s Flipped Clock is a modified digital clock display, where each individual digit is rotated by 180-degrees. Simon Pope’s Recall From Memory the Space of Another Gallery is an invitation for the visitor to recall experiences of being in other gallery spaces from memory. The seminal filmmaker Michael Snow’s WVLNT: Wavelength for those who don't have the time. Originally 45 minutes, Now 15! remixes his own seminal work Wavelength.
Credit
Curated by Sarah Cook. Supported by CRUMB and The University of Sunderland.
…neuroni alla ricerca di una strada mai percorsa, / labirinti cerebrali specchiati, / tutto è come amplificato, bizzarro, / sono un proiettile impazzito nella fantasia cosmica….
...neurons in search for a never covered route, / cerebral labyrinths mirrored, / everything is somehow amplified, bizarre, / I am a bullet gone wild in the cosmic fantasy...
Encéphale. Les péricaryons des neurones moteurs de l’encéphale contenant les corps de Nissl
(flèches bleues) donnent à ces cellules un aspect moucheté. Les corps de Nissl sont des amas de réticulum endoplasmique granuleux. Les cercles entourent diverses cellules gliales (astrocytes, oligodendrocytes, microgliocytes)
non reconnaissables avec la méthode de coloration utilisée. Le noyau (X) des neurones est imposant, ovoide et plus ou moins en position centrale.
- Pour plus de détails ou précisions, voir « Atlas of Fish Histology » CRC Press, ou « Histologie illustrée du poisson » (QUAE) ou s'adresser à Franck Genten (fgenten@gmail.com)
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Encephalon. Motor neurons containing NISSL bodies
(blue arrows) give the neuronal cytoplasm a granular
appearance. NISSL bodies are large granules which
consist of rough endoplasmic reticulum (i.e. with ribosomes).
The circles show various glial cells (astrocytes,
oligodendrocytes or microglial cells) not distinguishable
with the trichrome stain. The nucleus (X) of the neuron is large, ovoid and centrally located in the cytoplasm.
- For more information or details, see « Atlas of Fish Histology » CRC Press, or « Histologie illustrée du poisson » (QUAE) or contact Franck Genten (fgenten@gmail.com)
H. 30 ; L.40 cm
Avec encadrement H.40 ; L.50 cm
Acrylique brillante
Paris, 2012
> Original en vente: flic.kr/p/sNaXaX
> Estampe numérotée avec passe-partout: flic.kr/p/2kUjZ6P
> Impression grand format: flic.kr/p/2kU7mRc
=> impression encadrée: flic.kr/p/XkYpM7
> Dessous de verre: flic.kr/p/2kTJFZh
> Dessous de plat: flic.kr/p/2kTL7xh
> Set de table: flic.kr/p/2kTUzZ9
> Petits cadres noirs: flic.kr/p/2kTWgNL
> Cadres acrylique profondeur: flic.kr/p/2kTUN4u
> Sac imperméable: flic.kr/p/2kDKZo5
> Sac de courses: flic.kr/p/2j1i1wM
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The neuron is the large cell surrounded by very small glial cells. Cellular processes, dendrites and an axon, extend from the cell body of the neuron (where most of the cytoplasm and nucleus is located).
What's the role of an astrocyte in the human brain? New research at the University of California in San Diego suggests that the mysterious cells -- which link neurons in the human brain -- may be more important in understanding diseases such as Alzheimer's than once believed. In one study using a rat cortex, researchers observed how astrocytes were triggered and saw how calcium signals would rise and fall. The research could help scientists develop new drug treatments.
Source: UCSD
Via. FoxNews.com Eyepoppers
Sur cette image de neurone mutant d’hippocampe de rat, obtenue en microscopie confocale après 7 jours en culture (in vitro), on observe les détails des compartiments du neurone permettant la transmission d'une information au sein du cerveau. En magenta, on peut distinguer les dendrites (ramifications) du neurone qui recueillent de l’information et en cyan la partie proximale des axones, une extension normalement unique du neurone, qui propage l'information depuis le corps jusqu'aux terminaisons nerveuses (ou synapses). Ici, le rat auquel appartient ce neurone est muté pour le gène Prickle 2 qui ne s'exprime pas. Cette mutation conduit à la genèse anoemale de 4 axones au lieu d'un seul.
Cette image est extraite d'un travail mené par une équipe de chercheurs de l'Inserm qui cherche à mieux comprendre les troubles du développement neurologique (TND). Ces troubles entrainent des pathologies telles que les troubles du spectre autistique (TSA) ou l'épilepsie. Les résultats de leurs recherches rapportent que lorsque la protéine Prickle 2 est défectueuse, différents compartiments du neurone sont affectés ce qui entraîne une mauvaise transmission d'information. Ces défauts peuvent avoir des conséquences sur l’efficacité des approches thérapeutiques actuellement proposées pour les TND. Les avoir repérés et avoir identifié cette protéine défectueuse est un pas de plus vers la mise au point de thérapies plus efficaces.
© Ana Dorrego-Rivas et Mireille Montcouquiol /Inserm.licence CC-BY-NC 4.0 international
Source : The core PCP protein Prickle2 regulates axon number and AIS maturation by binding to AnkG and modulating microtubule bundling, ScienceAdvances, 9 septembre 2022
Clustering of endogenous ankyrinG or Na+ channel (red) at the axonal initial segment is not altered by a mutant ankyrinG (green) that disrupts βIV spectrin localization. (JCB 176(4) TOC2)
This image is available to the public to copy, distribute, or display under a Creative Commons Attribution-Noncommercial-Share Alike 3.0 Unported license.
Reference: Yang et al. (2007) J. Cell Biol. 176:509-519.
Published on: February 5, 2007.
Doi: 10.1083/jcb.200610128.
Read the full article at:
Agrandissement de l'image P11a_024 montrant les corps cellulaires des neurones géants de Mauthner. Le canal de l'épendyme, au centre, contient le liquide cérébrospinal (en turquoise). La substance grise est bien présente en haut du cliché. L’appareil de Mauthner est une spécialisation neuromusculaire agissant comme centre de coordination motrice qui reçoit des impressions sensorielles multiples et variées. Les cellules transmettent, via leur long axone, les informations motrices aux muscles blancs (rapides) du tronc et de la queue.
- Pour plus de détails ou précisions, voir « Atlas of Fish Histology » CRC Press, ou « Histologie illustrée du poisson » (QUAE) ou s'adresser à Franck Genten (fgenten@gmail.com)
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Higher magnification of the P11a_024 micrograph. The paired Mauthner neuronal bodies are obvious because of their huge triangular size. The central canal containing cerebrospinal fluid (turquoise) is lined by ependymal cells. Grey matter is well present at the top.
The Mauthnerian system is a neurolocomotory system
well developed in teleost fish. Two nerves possess giant axons which extend the length of the body, synapsing with motor neurons. Stimulations of the acoustic nerve VIII initiate rapid locomotion forward and away from the direction of stimuli.
- For more information or details, see « Atlas of Fish Histology » CRC Press, or « Histologie illustrée du poisson » (QUAE) or contact Franck Genten (fgenten@gmail.com)
That's no ordinary neuron! It's the ever-vivid Shelley Buschur.
Houston Art Car Parade 2007
The image displays a number of spiral ganglion neurons surrounded by Schwann cells from the inner ear of a mouse. An image stack was acquired using the FIB/SEM technique and the image was post processed in Amira ResolveRT creating this 3D reconstruction.
Courtesy of Michael Larsen
Image Details
Instrument used: Quanta DualBeam Family
Magnification: 6500x
Horizontal Field Width: 52.5
Vacuum: High
Voltage: 5.00kV
Spot: 1.0
Working Distance: 10
Detector: BCD