View allAll Photos Tagged neuron
Javier Velasco, Director General de Neuron Bio, habló sobre el “Presente y futuro de los biocombustibles de 2ª generación
startshape thing
rule thing 10 {
wig {}
shatter { hue 200 sat 60 }
shatter { s 1 .7 r 180 hue 200 sat 20 }
sig { x -3 hue 120 sat 80 b .5}
sig { x +3 hue 30 sat 80 b .5}
}
rule thing {}
rule shatter 2 {
shatter {}
limbo {}
}
rule shatter .05 {}
rule limbo {
limbo { r 1 b .0001 }
}
rule limbo {
limbo { r -1 b .005 }
}
rule limbo .1 { fiber {} }
rule limbo .001 {}
rule fiber {
CIRCLE {}
limbo { y .7 s .92}
}
rule fiber .05 {
CIRCLE {}
limbo { y .7 s 1.02}
}
rule fiber .05 {
CIRCLE {}
limbo { y .7 }
limbo { y .7 }
}
rule sig {
CIRCLE { s .5 y +.5 }
sig { s .93 r 20 }
}
rule sig {
CIRCLE { s .5 y -.5 }
sig { s .93 r -20 }
}
rule sig {
CIRCLE { s .5 x +.5 }
sig { s .93 r 30 b .1}
}
rule sig {
CIRCLE { s .5 x -.5 }
sig { s .93 r -30 b .1}
}
rule sig .1 {}
Neurons in the hindbrain of a zebrafish embryo are illuminated due to the use of flourscent chemicals. The branchiomotor neurons are labeled with green and the commissural axons are labeled with red. | Photo by Suman Gurung
miles de problemas
neuronas vivas
lenguaje, quimica, arte
canto metricas de atake defensivas
bombas de saliva
formas sanas de llevar esta vida destructiva.
va cada uno con su cuento
suma esta resta
vida desonesta
es la ke nadie kiere tomar
pero si pagan bien por ella da igual
ya ke al final, cada cual
define el bien del mal.
maneras de sobrevivir
llorar y reir
dificil asunto esto de apegarse a existir
ya ke abrir tu boca puede concluir en morir
los policias no te kieren como amigo
y pieles de zorro se lucen como abrigos.
carne humana, huesos , desecho
ahora ke te recuerdo ke estas vivo sacale provecho
yo pegado mirando el techo
tu rutina es mas repugnante ke la ley y mis supuestos derechos.
choca mi paso con el tuyo
muerdes mi mente, se caen tus dientes
concluyo
si yo, bien nacido
sabor picante y por zeus maldecido
herido pero nunca vencido
liberado en la libertad ke me libera
una paloma de ciudad que vuela mucho mas alla de la frontera
siguen tirando basura
no ven ke el mundo se envenena
Neurone du striatum exprimant la protéine huntingtine mutante (en rouge) - à l’origine de la maladie de Huntington - qui s’accumule dans le noyau (bleu) pour former un agrégat composé de huntingtine et d’autres protéines dont l’ubiquitine (en jaune).
© Frédéric Saudou/Inserm.licence CC-BY-NC 4.0 international
Image accompagnant le communiqué publié le 31 mars 2021 : "Maladie de Huntington : un espoir de traitement pour protéger le cerveau" presse.inserm.fr/maladie-de-huntington-un-espoir-de-trait...
En savoir plus :
La maladie de Huntington est une pathologie héréditaire qui entraîne la dégénérescence de neurones impliqués dans des fonctions motrices, cognitives et psychiatriques. Les traitements actuels sont symptomatiques et soulagent certains troubles mais ne permettent pas de modifier le cours de la maladie. Des chercheurs de l’Inserm, de l’Université Grenoble Alpes et du CHU Grenoble Alpes au Grenoble Institut des Neurosciences espèrent y remédier. En 2021, ils proposent une nouvelle piste thérapeutique dans l’espoir de proposer aux patients un premier traitement protégeant les neurones, dans les années qui suivront. Ils ont testé une molécule thérapeutique qui présente des résultats prometteurs chez la souris et dont ils ont ensuite lancé l'évaluation préclinique. Ce travail est publié dans la revue Science Advances.
Sources : doi.org/10.1126/sciadv.abb0799
Nuclei of squirrel brain cortical neurons. These are oval shaped nuclei, possibly because the cells were a bit crushed during the preparation for imaging :P The Hoechst stain is coloring the substructures in the nucleus a pretty blue.
New Graphic for a story on brain plasticity on Labgrab. www.labgrab.com/users/labgrab/blog/new-period-brain-%e2%8...
Campo oscuro. Filtro DiI. Tinción con cristales.
Prácticas NeurobiologÃa. Diciembre 2011. Universidad de Málaga
A member of the University of Oregon explains some of the properties of neurons and how our brains work during University Science Day exhibit at the Science Factory in Eugene, Oregon
Schwann cells lacking Rac1 are impaired in their ability to send cytoplasmic processes around axons and thus delay axon sorting and myelination. (JCB 177(6) TOC2)
This image is available to the public to copy, distribute, or display under a Creative Commons Attribution-Noncommercial-Share Alike 3.0 Unported license.
Reference: Nodari et al. (2007) J. Cell Biol. 187:1063-1075.
Published on: July 30, 2007.
Doi: 10.1083/jcb.200610014.
Read the full article at:
Carla Shatz has spent her life asking a question that sounds simple but turns out to be profound: how does the brain build itself?
When you meet her, you sense the discipline behind that inquiry. She is composed and quietly exacting. Her focus is the kind that defines a scientific life. In conversation, she moves carefully, assembling ideas the way neurons assemble circuits, piece by piece, connection by connection.
Shatz transformed our understanding of brain development by showing that neural circuits are not merely laid down by a genetic blueprint. They are sculpted by activity. Even before experience floods in through the senses, the brain generates its own patterns of electrical signaling. Those patterns guide which connections are strengthened and which are pruned away. The brain rehearses its future. It refines itself through internal dialogue.
This idea shifted the field. Development was no longer a passive unfolding. It was dynamic, competitive, and alive. Neurons communicate, negotiate, and withdraw. They respond to timing and rhythm. In her work, the young brain is not a blank slate waiting to be written on. It is an active participant in its own formation.
Later, she uncovered another layer of complexity. Molecules once believed to belong solely to the immune system turned out to play critical roles in shaping synapses. The boundaries between systems blurred. Immunology and neuroscience began to overlap in ways few had anticipated. The implications stretch into our understanding of neurodevelopmental disorders and neurodegeneration. Connections that fail to refine properly can echo across a lifetime.
What stands out most is her intellectual courage. To propose that spontaneous neural activity guides wiring before sensory experience was to challenge prevailing assumptions. She followed the data where it led, even when it unsettled established frameworks. She has built institutions, led the Bio-X program at Stanford, and mentored generations of neuroscientists. Her influence radiates through both published papers and the people she has trained.
The brain, in her telling, is not fixed at birth. It is shaped by activity, by competition, and by molecular conversations between cells. It is sculpted over time. Carla Shatz has spent her career illuminating that delicate process. The story of how a brain forms is, after all, the story of how each of us comes into being.
There are micrographs from my undergrad days. My first published photographs. These are looking at the expression of BMP-7 in neurons cultured in the presence vs. absence of glia. (Though i guess technically this one is looking at RNA not protein.)
Lein PJ, Beck HN, Chandrasekaran V, Gallagher PJ, Chen HL, Lin Y, Guo X, Kaplan PL, Tiedge H, Higgins D.
Glia induce dendritic growth in cultured sympathetic neurons by modulating the balance between bone morphogenetic proteins (BMPs) and BMP antagonists.
J Neurosci. 2002 Dec 1;22(23):10377-87.
Excitatory pyramidal neuron of cortex highlighting structure of dendrites, branches that receive synaptic input from axons of other cells.
Render by Amy Sterling from reconstructions by Seung Lab, Princeton Neuroscience Institute using images acquired by The Allen Institute. Funded by IARPA MICrONS. Rendered in Cinema 4D using Otoy Octane GPU renderer.
Fluorescence microscope image taken of mouse hippocampal cells dyed with Oregon Green 488 BAPTA-1. Neurons appear as a wiry, bright object (with the cell body illuminated) while the larger, spread out shapes are neuroglia.
similar to the other picture but zoomed out. young neurons in pink line the inner edge of a tightly packed, 6-7 neuron deep structure called the dentate gyrus. their dendrites reach out to receive incoming information. the green cells are neurons that have just been "activated" when this mouse recalled a 1 week old memory. that the neurons involved in representing the memory (green) are not at the inner edge would suggest that the young neurons are not so involved in long-term memory retrieval/storage. unfortunately, broad and qualitative descriptions don't cut it and so thousands of cells must be quantified manually. gives one a good amount of time to catch up on all that music.
Nuclei of squirrel brain cortical neurons. The big faint circle is the nucleus, the bright blue ball in the middle is the nucleolus. There is both blue and green fluorescent staining in this picture but for these cells only the blue nuclear Hoechst stain is really visible.
*Wireless Display (WiDi) Requiere una PC habilitada con Intel® Wireless Display, Adaptador receptopr para TV, televisor compatible.
Además consigue ya tu Lanix Neuron R 1104-LFA07 en bit.ly/t8piFv o tu Lanix Neuron R 1104-LFA06 en bit.ly/urij09
Motor neuron progenitors (green) derived from human embryonic stem cells. Such neurons could be used to treat spinal cord injuries and diseases of the motor neurons.
This photo was taken by Sharyn Rossi in the lab of Hans Keirstead at the University of California, Irvine.
Learn more about CIRM-funded stem cell research: www.cirm.ca.gov