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Por favor, no usar esta imagen sin mi permiso explícito.

Please, don´t use this image without my explicit permission.

Mejor sobre negro. Haz click en la foto.

Better on black.Just click on the pic.

 

Corteza Cerebral de Erizo de tierra.

 

En esta estación del año, de árboles con ramas desnudas, yo os muestro estas otras ramas, las neuronales del " Bosque Cerebral ".

Proceden de mis trabajos de investigación en el cerebro de diferentes mamíferos. Son fotografías obtenidas con microscopio sobre material teñido de forma que las neuronas se visualizan en color pardo-negro sobre fondo amarillento.

Así visto, el Bosque Cerebral me parece un elemento muy sugestivo.

Mi pretensión en esta foto y en todas las del album, es únicamente ESTÉTICA, no científica, y así os pido que lo mireis.

Si deseais alguna aclaración técnica podeis leer la introducción del Album.

 

Hedgehog Cerebral Cortex.

 

In this season of naked branches in the trees, i show you these neuronal branchings on Cerebral Trees. They come from my research works about differents mammals´ brains. These are micro-photographies on stained matherial, so the neurons appear in dark brown-black on yellow background.

I think this Brain Forest is a very attractive and aesthetic element. So, my aim in this picture and in the album is JUST AESTHETIC, no scientific, and like that you should look at it.

For more technical details, please, read the album introduction.

 

At the UCSF Decoding the Brain Academy last night, Dr. Tomasz Nowakowski plotted the progress of neurons sequenced over time, including the project planned for 2025. It has been following a Moore’s Law-like exponential curve for a decade now. So, we should have a sequence of every one of the 86 billion neurons in a human brain by 2032, and maybe all the glial cells too.

 

Why sequence every cell? The genetic makeup varies, as does the methylation. On the left, you can see clustering analysis of 121 different cell types in the thalamus alone. Out neurons are very different, and they vary over time. I was reminded of an amazing discovery Ed Boyden of MIT shared with me — they found HIV-like encodings being expressed in human neurons. This might be a transposon-like viral vector for horizontal gene transfer in the brain. This is a controversial finding, partially because it is difficult to detect the signal from the noise of the delta from the human reference genome, which is still Craig Venter. I asked Ed how many neuron types does he suspect we’ll find? “An infinite number. They are all different.”

 

When we asked Nowakowski about the compute capacity of a neuron, he lit up with excitement. By analogy to our machine learning neural nets with back prop, we now know that our neurons don’t just adjust weights at the synapse. Firing-feedback adjusts the weights in all of the ~1000 inputs embedded in the dendrite of each neuron. This is called synaptic tagging, and it serves as an overlay to the relaxation back to a nominal rate of firing, a process that is not fully understood.

 

From the other speakers on stage, left to right:

• Shawn Hervey-Jumper: in 50 years of neurosurgery advances, we have added just 4 months to expected lifespan

 

• Mercedes Paredes: in the pre-natal and infant brain (up to 6 months), neurons migrate great distances (1-10cm) by sending out an axon and pulling the neuron cell body along, a sequence of push and pulls. For a sense of scale, if the neuron was a car, it is like driving across America. How do they navigate over these distances?

 

• Christine Liu: We can keep brain tissues alive for weeks now (for glass probe insertion into neural cell bodies). Learning how to juggle can increase cortical thickness by 3%.

Left: This is your brain. Right: This is your brain after intermittent binge drinking.

 

DURHAM, N.C. -- Studies have demonstrated how just a few sessions of binge drinking during adolescence can knock out neurons (shown in blue arch) in the hippocampus, the brain’s memory core.

 

But researchers at Duke Medicine have found that binge drinking can also send hippocampal cells called astrocytes (shown in green) awry later in adulthood, potentially impairing the brain’s ability to form new synapses and heal itself from injury.

 

The study, published November 5 in Alcoholism: Clinical & Experimental Research, used a rodent model as a surrogate for the adolescent human brain. The researchers exposed the animals to alcohol doses that would result in a blood-alcohol concentration of about .15 in humans.

 

Researchers didn’t see immediate effects on astrocytes, but once the animals reached adulthood, the cells appeared to go into overdrive.

 

Image credit: Mary-Louise Risher/Duke Medicine

 

This image is not owned by the NIH. It is shared with the public under license. If you have a question about using or reproducing this image, please contact the creator listed in the credits. All rights to the work remain with the original creator.

 

NIH funding from:

The National Institute on Alcohol Abuse and Alcoholism (NIAAA),

The National Institute on Drug Abuse (NIDA)

  

Cracking day today, sun was out and not a drop of rain, so went for walk with the girlfriend up the local woods, took the camera along too,Haven't done one of these shots before so thought i would try one as we were out.

 

shot of the trees then when i got home,went crazy with the hue slider,liking the green ;)

Amanita muscaria, commonly known as the fly agaric or fly amanita, is a basidiomycete of the genus Amanita. It is a large white-gilled, white-spotted, and usually red mushroom.

 

Despite its easily distinguishable features, A. muscaria is a fungus with several known variations, or subspecies. These subspecies are slightly different, some having yellow or white caps, but are all usually called fly agarics, most often recognizable by their notable white spots. Recent DNA fungi research, however, has shown that some mushrooms called 'fly agaric' are in fact unique species, such as A. persicina (the peach-colored fly agaric).

 

Native throughout the temperate and boreal regions of the Northern Hemisphere, A. muscaria has been unintentionally introduced to many countries in the Southern Hemisphere, generally as a symbiont with pine and birch plantations, and is now a true cosmopolitan species. It associates with various deciduous and coniferous trees.

 

Although poisonous, death due to poisoning from A. muscaria ingestion is quite rare. Parboiling twice with water draining weakens its toxicity and breaks down the mushroom's psychoactive substances; it is eaten in parts of Europe, Asia, and North America. All A. muscaria varieties, but in particular A. muscaria var. muscaria, are noted for their hallucinogenic properties, with the main psychoactive constituents being muscimol and its neurotoxic precursor ibotenic acid. A local variety of the mushroom was used as an intoxicant and entheogen by the indigenous peoples of Siberia.

 

Arguably the most iconic toadstool species, the fly agaric is one of the most recognizable and widely encountered in popular culture, including in video games—for example, the frequent use of a recognizable A. muscaria in the Mario franchise (e.g. its Super Mushroom power-up)—and television—for example, the houses in The Smurfs franchise. There have been cases of children admitted to hospitals after consuming this poisonous mushroom; the children may have been attracted to it because of its pop-culture associations.

 

Taxonomy

The name of the mushroom in many European languages is thought to derive from its use as an insecticide when sprinkled in milk. This practice has been recorded from Germanic- and Slavic-speaking parts of Europe, as well as the Vosges region and pockets elsewhere in France, and Romania. Albertus Magnus was the first to record it in his work De vegetabilibus some time before 1256, commenting vocatur fungus muscarum, eo quod in lacte pulverizatus interficit muscas, "it is called the fly mushroom because it is powdered in milk to kill flies."

 

The 16th-century Flemish botanist Carolus Clusius traced the practice of sprinkling it into milk to Frankfurt in Germany, while Carl Linnaeus, the "father of taxonomy", reported it from Småland in southern Sweden, where he had lived as a child. He described it in volume two of his Species Plantarum in 1753, giving it the name Agaricus muscarius, the specific epithet deriving from Latin musca meaning "fly". It gained its current name in 1783, when placed in the genus Amanita by Jean-Baptiste Lamarck, a name sanctioned in 1821 by the "father of mycology", Swedish naturalist Elias Magnus Fries. The starting date for all the mycota had been set by general agreement as January 1, 1821, the date of Fries's work, and so the full name was then Amanita muscaria (L.:Fr.) Hook. The 1987 edition of the International Code of Botanical Nomenclature changed the rules on the starting date and primary work for names of fungi, and names can now be considered valid as far back as May 1, 1753, the date of publication of Linnaeus's work. Hence, Linnaeus and Lamarck are now taken as the namers of Amanita muscaria (L.) Lam..

 

The English mycologist John Ramsbottom reported that Amanita muscaria was used for getting rid of bugs in England and Sweden, and bug agaric was an old alternative name for the species. French mycologist Pierre Bulliard reported having tried without success to replicate its fly-killing properties in his work Histoire des plantes vénéneuses et suspectes de la France (1784), and proposed a new binomial name Agaricus pseudo-aurantiacus because of this. One compound isolated from the fungus is 1,3-diolein (1,3-di(cis-9-octadecenoyl)glycerol), which attracts insects. It has been hypothesised that the flies intentionally seek out the fly agaric for its intoxicating properties. An alternative derivation proposes that the term fly- refers not to insects as such but rather the delirium resulting from consumption of the fungus. This is based on the medieval belief that flies could enter a person's head and cause mental illness. Several regional names appear to be linked with this connotation, meaning the "mad" or "fool's" version of the highly regarded edible mushroom Amanita caesarea. Hence there is oriol foll "mad oriol" in Catalan, mujolo folo from Toulouse, concourlo fouolo from the Aveyron department in Southern France, ovolo matto from Trentino in Italy. A local dialect name in Fribourg in Switzerland is tsapi de diablhou, which translates as "Devil's hat".

 

Classification

Amanita muscaria is the type species of the genus. By extension, it is also the type species of Amanita subgenus Amanita, as well as section Amanita within this subgenus. Amanita subgenus Amanita includes all Amanita with inamyloid spores. Amanita section Amanita includes the species with patchy universal veil remnants, including a volva that is reduced to a series of concentric rings, and the veil remnants on the cap to a series of patches or warts. Most species in this group also have a bulbous base. Amanita section Amanita consists of A. muscaria and its close relatives, including A. pantherina (the panther cap), A. gemmata, A. farinosa, and A. xanthocephala. Modern fungal taxonomists have classified Amanita muscaria and its allies this way based on gross morphology and spore inamyloidy. Two recent molecular phylogenetic studies have confirmed this classification as natural.

 

Description

A large, conspicuous mushroom, Amanita muscaria is generally common and numerous where it grows, and is often found in groups with basidiocarps in all stages of development. Fly agaric fruiting bodies emerge from the soil looking like white eggs. After emerging from the ground, the cap is covered with numerous small white to yellow pyramid-shaped warts. These are remnants of the universal veil, a membrane that encloses the entire mushroom when it is still very young. Dissecting the mushroom at this stage reveals a characteristic yellowish layer of skin under the veil, which helps identification. As the fungus grows, the red colour appears through the broken veil and the warts become less prominent; they do not change in size, but are reduced relative to the expanding skin area. The cap changes from globose to hemispherical, and finally to plate-like and flat in mature specimens. Fully grown, the bright red cap is usually around 8–20 centimetres (3–8 inches) in diameter, although larger specimens have been found. The red colour may fade after rain and in older mushrooms.

 

The free gills are white, as is the spore print. The oval spores measure 9–13 by 6.5–9 μm; they do not turn blue with the application of iodine. The stipe is white, 5–20 cm (2–8 in) high by 1–2 cm (1⁄2–1 in) wide, and has the slightly brittle, fibrous texture typical of many large mushrooms. At the base is a bulb that bears universal veil remnants in the form of two to four distinct rings or ruffs. Between the basal universal veil remnants and gills are remnants of the partial veil (which covers the gills during development) in the form of a white ring. It can be quite wide and flaccid with age. There is generally no associated smell other than a mild earthiness.

 

Although very distinctive in appearance, the fly agaric has been mistaken for other yellow to red mushroom species in the Americas, such as Armillaria cf. mellea and the edible A. basii—a Mexican species similar to A. caesarea of Europe. Poison control centres in the U.S. and Canada have become aware that amarill (Spanish for 'yellow') is a common name for the A. caesarea-like species in Mexico. A. caesarea is distinguished by its entirely orange to red cap, which lacks the numerous white warty spots of the fly agaric (though these sometimes wash away during heavy rain). Furthermore, the stem, gills and ring of A. caesarea are bright yellow, not white. The volva is a distinct white bag, not broken into scales. In Australia, the introduced fly agaric may be confused with the native vermilion grisette (Amanita xanthocephala), which grows in association with eucalypts. The latter species generally lacks the white warts of A. muscaria and bears no ring. Additionally, immature button forms resemble puffballs.

 

Controversy

Amanita muscaria var. formosa is now a synonym for Amanita muscaria var. guessowii.

Amanita muscaria varies considerably in its morphology, and many authorities recognize several subspecies or varieties within the species. In The Agaricales in Modern Taxonomy, German mycologist Rolf Singer listed three subspecies, though without description: A. muscaria ssp. muscaria, A. muscaria ssp. americana, and A. muscaria ssp. flavivolvata.

 

However, a 2006 molecular phylogenetic study of different regional populations of A. muscaria by mycologist József Geml and colleagues found three distinct clades within this species representing, roughly, Eurasian, Eurasian "subalpine", and North American populations. Specimens belonging to all three clades have been found in Alaska; this has led to the hypothesis that this was the centre of diversification for this species. The study also looked at four named varieties of the species: var. alba, var. flavivolvata, var. formosa (including var. guessowii), and var. regalis from both areas. All four varieties were found within both the Eurasian and North American clades, evidence that these morphological forms are polymorphisms rather than distinct subspecies or varieties. Further molecular study by Geml and colleagues published in 2008 show that these three genetic groups, plus a fourth associated with oak–hickory–pine forest in the southeastern United States and two more on Santa Cruz Island in California, are delineated from each other enough genetically to be considered separate species. Thus A. muscaria as it stands currently is, evidently, a species complex. The complex also includes at least three other closely related taxa that are currently regarded as species: A. breckonii is a buff-capped mushroom associated with conifers from the Pacific Northwest, and the brown-capped A. gioiosa and A. heterochroma from the Mediterranean Basin and from Sardinia respectively. Both of these last two are found with Eucalyptus and Cistus trees, and it is unclear whether they are native or introduced from Australia.

 

Distribution and habitat

A. muscaria is a cosmopolitan mushroom, native to conifer and deciduous woodlands throughout the temperate and boreal regions of the Northern Hemisphere, including higher elevations of warmer latitudes in regions such as Hindu Kush, the Mediterranean and also Central America. A recent molecular study proposes that it had an ancestral origin in the Siberian–Beringian region in the Tertiary period, before radiating outwards across Asia, Europe and North America. The season for fruiting varies in different climates: fruiting occurs in summer and autumn across most of North America, but later in autumn and early winter on the Pacific coast. This species is often found in similar locations to Boletus edulis, and may appear in fairy rings. Conveyed with pine seedlings, it has been widely transported into the southern hemisphere, including Australia, New Zealand, South Africa and South America, where it can be found in the Brazilian states of Paraná, São Paulo, Minas Gerais, Rio Grande do Sul.

 

Ectomycorrhizal, A. muscaria forms symbiotic relationships with many trees, including pine, oak, spruce, fir, birch, and cedar. Commonly seen under introduced trees, A. muscaria is the fungal equivalent of a weed in New Zealand, Tasmania and Victoria, forming new associations with southern beech (Nothofagus). The species is also invading a rainforest in Australia, where it may be displacing the native species. It appears to be spreading northwards, with recent reports placing it near Port Macquarie on the New South Wales north coast. It was recorded under silver birch (Betula pendula) in Manjimup, Western Australia in 2010. Although it has apparently not spread to eucalypts in Australia, it has been recorded associating with them in Portugal. Commonly found throughout the great Southern region of western Australia, it is regularly found growing on Pinus radiata.

 

Toxicity

a tall red mushroom with a few white spots on the cap

Mature. The white spots may wash off with heavy rainfall.

A. muscaria poisoning has occurred in young children and in people who ingested the mushrooms for a hallucinogenic experience, or who confused it with an edible species.

 

A. muscaria contains several biologically active agents, at least one of which, muscimol, is known to be psychoactive. Ibotenic acid, a neurotoxin, serves as a prodrug to muscimol, with a small amount likely converting to muscimol after ingestion. An active dose in adults is approximately 6 mg muscimol or 30 to 60 mg ibotenic acid; this is typically about the amount found in one cap of Amanita muscaria. The amount and ratio of chemical compounds per mushroom varies widely from region to region and season to season, which can further confuse the issue. Spring and summer mushrooms have been reported to contain up to 10 times more ibotenic acid and muscimol than autumn fruitings.

 

Deaths from A. muscaria have been reported in historical journal articles and newspaper reports, but with modern medical treatment, fatal poisoning from ingesting this mushroom is extremely rare. Many books list A. muscaria as deadly, but according to David Arora, this is an error that implies the mushroom is far more toxic than it is. Furthermore, The North American Mycological Association has stated that there were "no reliably documented cases of death from toxins in these mushrooms in the past 100 years".

 

The active constituents of this species are water-soluble, and boiling and then discarding the cooking water at least partly detoxifies A. muscaria. Drying may increase potency, as the process facilitates the conversion of ibotenic acid to the more potent muscimol. According to some sources, once detoxified, the mushroom becomes edible. Patrick Harding describes the Sami custom of processing the fly agaric through reindeer.

 

Pharmacology

Ibotenic acid, a prodrug to muscimol found in A. muscaria

Muscarine, discovered in 1869, was long thought to be the active hallucinogenic agent in A. muscaria. Muscarine binds with muscarinic acetylcholine receptors leading to the excitation of neurons bearing these receptors. The levels of muscarine in Amanita muscaria are minute when compared with other poisonous fungi such as Inosperma erubescens, the small white Clitocybe species C. dealbata and C. rivulosa. The level of muscarine in A. muscaria is too low to play a role in the symptoms of poisoning.

 

The major toxins involved in A. muscaria poisoning are muscimol (3-hydroxy-5-aminomethyl-1-isoxazole, an unsaturated cyclic hydroxamic acid) and the related amino acid ibotenic acid. Muscimol is the product of the decarboxylation (usually by drying) of ibotenic acid. Muscimol and ibotenic acid were discovered in the mid-20th century. Researchers in England, Japan, and Switzerland showed that the effects produced were due mainly to ibotenic acid and muscimol, not muscarine. These toxins are not distributed uniformly in the mushroom. Most are detected in the cap of the fruit, a moderate amount in the base, with the smallest amount in the stalk. Quite rapidly, between 20 and 90 minutes after ingestion, a substantial fraction of ibotenic acid is excreted unmetabolised in the urine of the consumer. Almost no muscimol is excreted when pure ibotenic acid is eaten, but muscimol is detectable in the urine after eating A. muscaria, which contains both ibotenic acid and muscimol.

 

Ibotenic acid and muscimol are structurally related to each other and to two major neurotransmitters of the central nervous system: glutamic acid and GABA respectively. Ibotenic acid and muscimol act like these neurotransmitters, muscimol being a potent GABAA agonist, while ibotenic acid is an agonist of NMDA glutamate receptors and certain metabotropic glutamate receptors which are involved in the control of neuronal activity. It is these interactions which are thought to cause the psychoactive effects found in intoxication.

 

Muscazone is another compound that has more recently been isolated from European specimens of the fly agaric. It is a product of the breakdown of ibotenic acid by ultra-violet radiation. Muscazone is of minor pharmacological activity compared with the other agents. Amanita muscaria and related species are known as effective bioaccumulators of vanadium; some species concentrate vanadium to levels of up to 400 times those typically found in plants. Vanadium is present in fruit-bodies as an organometallic compound called amavadine. The biological importance of the accumulation process is unknown.

 

Symptoms

Fly agarics are best known for the unpredictability of their effects. Depending on habitat and the amount ingested per body weight, effects can range from mild nausea and twitching to drowsiness, cholinergic crisis-like effects (low blood pressure, sweating and salivation), auditory and visual distortions, mood changes, euphoria, relaxation, ataxia, and loss of equilibrium (like with tetanus.)

 

In cases of serious poisoning the mushroom causes delirium, somewhat similar in effect to anticholinergic poisoning (such as that caused by Datura stramonium), characterised by bouts of marked agitation with confusion, hallucinations, and irritability followed by periods of central nervous system depression. Seizures and coma may also occur in severe poisonings. Symptoms typically appear after around 30 to 90 minutes and peak within three hours, but certain effects can last for several days. In the majority of cases recovery is complete within 12 to 24 hours. The effect is highly variable between individuals, with similar doses potentially causing quite different reactions. Some people suffering intoxication have exhibited headaches up to ten hours afterwards.[56] Retrograde amnesia and somnolence can result following recovery.

 

Treatment

Medical attention should be sought in cases of suspected poisoning. If the delay between ingestion and treatment is less than four hours, activated charcoal is given. Gastric lavage can be considered if the patient presents within one hour of ingestion. Inducing vomiting with syrup of ipecac is no longer recommended in any poisoning situation.

 

There is no antidote, and supportive care is the mainstay of further treatment for intoxication. Though sometimes referred to as a deliriant and while muscarine was first isolated from A. muscaria and as such is its namesake, muscimol does not have action, either as an agonist or antagonist, at the muscarinic acetylcholine receptor site, and therefore atropine or physostigmine as an antidote is not recommended. If a patient is delirious or agitated, this can usually be treated by reassurance and, if necessary, physical restraints. A benzodiazepine such as diazepam or lorazepam can be used to control combativeness, agitation, muscular overactivity, and seizures. Only small doses should be used, as they may worsen the respiratory depressant effects of muscimol. Recurrent vomiting is rare, but if present may lead to fluid and electrolyte imbalances; intravenous rehydration or electrolyte replacement may be required. Serious cases may develop loss of consciousness or coma, and may need intubation and artificial ventilation. Hemodialysis can remove the toxins, although this intervention is generally considered unnecessary. With modern medical treatment the prognosis is typically good following supportive treatment.

 

Uses

The wide range of psychoactive effects have been variously described as depressant, sedative-hypnotic, psychedelic, dissociative, or deliriant; paradoxical effects such as stimulation may occur however. Perceptual phenomena such as synesthesia, macropsia, and micropsia may occur; the latter two effects may occur either simultaneously or alternatingly, as part of Alice in Wonderland syndrome, collectively known as dysmetropsia, along with related distortions pelopsia and teleopsia. Some users report lucid dreaming under the influence of its hypnotic effects. Unlike Psilocybe cubensis, A. muscaria cannot be commercially cultivated, due to its mycorrhizal relationship with the roots of pine trees. However, following the outlawing of psilocybin mushrooms in the United Kingdom in 2006, the sale of the still legal A. muscaria began increasing.

 

Marija Gimbutas reported to R. Gordon Wasson that in remote areas of Lithuania, A. muscaria has been consumed at wedding feasts, in which mushrooms were mixed with vodka. She also reported that the Lithuanians used to export A. muscaria to the Sami in the Far North for use in shamanic rituals. The Lithuanian festivities are the only report that Wasson received of ingestion of fly agaric for religious use in Eastern Europe.

 

Siberia

A. muscaria was widely used as an entheogen by many of the indigenous peoples of Siberia. Its use was known among almost all of the Uralic-speaking peoples of western Siberia and the Paleosiberian-speaking peoples of the Russian Far East. There are only isolated reports of A. muscaria use among the Tungusic and Turkic peoples of central Siberia and it is believed that on the whole entheogenic use of A. muscaria was not practised by these peoples. In western Siberia, the use of A. muscaria was restricted to shamans, who used it as an alternative method of achieving a trance state. (Normally, Siberian shamans achieve trance by prolonged drumming and dancing.) In eastern Siberia, A. muscaria was used by both shamans and laypeople alike, and was used recreationally as well as religiously. In eastern Siberia, the shaman would take the mushrooms, and others would drink his urine. This urine, still containing psychoactive elements, may be more potent than the A. muscaria mushrooms with fewer negative effects such as sweating and twitching, suggesting that the initial user may act as a screening filter for other components in the mushroom.

 

The Koryak of eastern Siberia have a story about the fly agaric (wapaq) which enabled Big Raven to carry a whale to its home. In the story, the deity Vahiyinin ("Existence") spat onto earth, and his spittle became the wapaq, and his saliva becomes the warts. After experiencing the power of the wapaq, Raven was so exhilarated that he told it to grow forever on earth so his children, the people, could learn from it. Among the Koryaks, one report said that the poor would consume the urine of the wealthy, who could afford to buy the mushrooms. It was reported that the local reindeer would often follow an individual intoxicated by the muscimol mushroom, and if said individual were to urinate in snow the reindeer would become similarly intoxicated and the Koryak people's would use the drunken state of the reindeer to more easily rope and hunt them.

 

Other reports and theories

The Finnish historian T. I. Itkonen mentions that A. muscaria was once used among the Sámi peoples. Sorcerers in Inari would consume fly agarics with seven spots. In 1979, Said Gholam Mochtar and Hartmut Geerken published an article in which they claimed to have discovered a tradition of medicinal and recreational use of this mushroom among a Parachi-speaking group in Afghanistan. There are also unconfirmed reports of religious use of A. muscaria among two Subarctic Native American tribes. Ojibwa ethnobotanist Keewaydinoquay Peschel reported its use among her people, where it was known as miskwedo (an abbreviation of the name oshtimisk wajashkwedo (= "red-top mushroom"). This information was enthusiastically received by Wasson, although evidence from other sources was lacking. There is also one account of a Euro-American who claims to have been initiated into traditional Tlicho use of Amanita muscaria. The flying reindeer of Santa Claus, who is called Joulupukki in Finland, could symbolize the use of A. muscaria by Sámi shamans. However, Sámi scholars and the Sámi peoples themselves refute any connection between Santa Claus and Sámi history or culture.

 

"The story of Santa emerging from a Sámi shamanic tradition has a critical number of flaws," asserts Tim Frandy, assistant professor of Nordic Studies at the University of British Columbia and a member of the Sámi descendent community in North America. "The theory has been widely criticized by Sámi people as a stereotypical and problematic romanticized misreading of actual Sámi culture."

 

Vikings

The notion that Vikings used A. muscaria to produce their berserker rages was first suggested by the Swedish professor Samuel Ödmann in 1784. Ödmann based his theories on reports about the use of fly agaric among Siberian shamans. The notion has become widespread since the 19th century, but no contemporary sources mention this use or anything similar in their description of berserkers. Muscimol is generally a mild relaxant, but it can create a range of different reactions within a group of people. It is possible that it could make a person angry, or cause them to be "very jolly or sad, jump about, dance, sing or give way to great fright". Comparative analysis of symptoms have, however, since shown Hyoscyamus niger to be a better fit to the state that characterises the berserker rage.

 

Soma

See also: Botanical identity of Soma-Haoma

In 1968, R. Gordon Wasson proposed that A. muscaria was the soma talked about in the Rigveda of India, a claim which received widespread publicity and popular support at the time. He noted that descriptions of Soma omitted any description of roots, stems or seeds, which suggested a mushroom, and used the adjective hári "dazzling" or "flaming" which the author interprets as meaning red. One line described men urinating Soma; this recalled the practice of recycling urine in Siberia. Soma is mentioned as coming "from the mountains", which Wasson interpreted as the mushroom having been brought in with the Aryan migrants from the north. Indian scholars Santosh Kumar Dash and Sachinanda Padhy pointed out that both eating of mushrooms and drinking of urine were proscribed, using as a source the Manusmṛti. In 1971, Vedic scholar John Brough from Cambridge University rejected Wasson's theory and noted that the language was too vague to determine a description of Soma. In his 1976 survey, Hallucinogens and Culture, anthropologist Peter T. Furst evaluated the evidence for and against the identification of the fly agaric mushroom as the Vedic Soma, concluding cautiously in its favour. Kevin Feeney and Trent Austin compared the references in the Vedas with the filtering mechanisms in the preparation of Amanita muscaria and published findings supporting the proposal that fly-agaric mushrooms could be a likely candidate for the sacrament. Other proposed candidates include Psilocybe cubensis, Peganum harmala, and Ephedra.

 

Christianity

Philologist, archaeologist, and Dead Sea Scrolls scholar John Marco Allegro postulated that early Christian theology was derived from a fertility cult revolving around the entheogenic consumption of A. muscaria in his 1970 book The Sacred Mushroom and the Cross. This theory has found little support by scholars outside the field of ethnomycology. The book was widely criticized by academics and theologians, including Sir Godfrey Driver, emeritus Professor of Semitic Philology at Oxford University and Henry Chadwick, the Dean of Christ Church, Oxford. Christian author John C. King wrote a detailed rebuttal of Allegro's theory in the 1970 book A Christian View of the Mushroom Myth; he notes that neither fly agarics nor their host trees are found in the Middle East, even though cedars and pines are found there, and highlights the tenuous nature of the links between biblical and Sumerian names coined by Allegro. He concludes that if the theory were true, the use of the mushroom must have been "the best kept secret in the world" as it was so well concealed for two thousand years.

 

Fly trap

Amanita muscaria is traditionally used for catching flies possibly due to its content of ibotenic acid and muscimol, which lead to its common name "fly agaric". Recently, an analysis of nine different methods for preparing A. muscaria for catching flies in Slovenia have shown that the release of ibotenic acid and muscimol did not depend on the solvent (milk or water) and that thermal and mechanical processing led to faster extraction of ibotenic acid and muscimol.

 

Culinary

The toxins in A. muscaria are water-soluble: parboiling A. muscaria fruit bodies can detoxify them and render them edible, although consumption of the mushroom as a food has never been widespread. The consumption of detoxified A. muscaria has been practiced in some parts of Europe (notably by Russian settlers in Siberia) since at least the 19th century, and likely earlier. The German physician and naturalist Georg Heinrich von Langsdorff wrote the earliest published account on how to detoxify this mushroom in 1823. In the late 19th century, the French physician Félix Archimède Pouchet was a populariser and advocate of A. muscaria consumption, comparing it to manioc, an important food source in tropical South America that must also be detoxified before consumption.

 

Use of this mushroom as a food source also seems to have existed in North America. A classic description of this use of A. muscaria by an African-American mushroom seller in Washington, D.C., in the late 19th century is described by American botanist Frederick Vernon Coville. In this case, the mushroom, after parboiling, and soaking in vinegar, is made into a mushroom sauce for steak. It is also consumed as a food in parts of Japan. The most well-known current use as an edible mushroom is in Nagano Prefecture, Japan. There, it is primarily salted and pickled.

 

A 2008 paper by food historian William Rubel and mycologist David Arora gives a history of consumption of A. muscaria as a food and describes detoxification methods. They advocate that Amanita muscaria be described in field guides as an edible mushroom, though accompanied by a description on how to detoxify it. The authors state that the widespread descriptions in field guides of this mushroom as poisonous is a reflection of cultural bias, as several other popular edible species, notably morels, are also toxic unless properly cooked.

 

In culture

The red-and-white spotted toadstool is a common image in many aspects of popular culture. Garden ornaments and children's picture books depicting gnomes and fairies, such as the Smurfs, often show fly agarics used as seats, or homes. Fly agarics have been featured in paintings since the Renaissance, albeit in a subtle manner. For instance, in Hieronymus Bosch's painting, The Garden of Earthly Delights, the mushroom can be seen on the left-hand panel of the work. In the Victorian era they became more visible, becoming the main topic of some fairy paintings. Two of the most famous uses of the mushroom are in the Mario franchise (specifically two of the Super Mushroom power-up items and the platforms in several stages which are based on a fly agaric), and the dancing mushroom sequence in the 1940 Disney film Fantasia.

 

An account of the journeys of Philip von Strahlenberg to Siberia and his descriptions of the use of the mukhomor there was published in English in 1736. The drinking of urine of those who had consumed the mushroom was commented on by Anglo-Irish writer Oliver Goldsmith in his widely read 1762 novel, Citizen of the World. The mushroom had been identified as the fly agaric by this time. Other authors recorded the distortions of the size of perceived objects while intoxicated by the fungus, including naturalist Mordecai Cubitt Cooke in his books The Seven Sisters of Sleep and A Plain and Easy Account of British Fungi. This observation is thought to have formed the basis of the effects of eating the mushroom in the 1865 popular story Alice's Adventures in Wonderland. A hallucinogenic "scarlet toadstool" from Lappland is featured as a plot element in Charles Kingsley's 1866 novel Hereward the Wake based on the medieval figure of the same name. Thomas Pynchon's 1973 novel Gravity's Rainbow describes the fungus as a "relative of the poisonous Destroying angel" and presents a detailed description of a character preparing a cookie bake mixture from harvested Amanita muscaria. Fly agaric shamanism is also explored in the 2003 novel Thursbitch by Alan Garner.

Me he armado de valor y he hecho la pulsera aquella

www.firemountaingems.com/galleryofdesigns/jewelry_design_...

que fue la idea base para estos pendientes:

www.flickr.com/photos/alia_h/1854766165/in/set-7215760023...

Lo malo no es que te deje la paciencia maltrecha (que te la deja) o que se te queden las neuronas en carne viva, como diría mi amiga Mª Jesús (que se te quedan...) El mayor escollo de esta pieza es que necesitas más hilo que Spiderman. Cogí 5 m. para empezar y me faltó a medio camino. Tuve que hacer un trabajo de cirujano, no me quiero ni acordar...

 

Neuron is a European collaboration project to develop a UCAV demonstrator, Unmanned Combat Aerial Vehicle. Six aviation industries are participating. The work packages being managed by Saab relate to low signature (stealth) technology, flight testing, aerodynamics, avionics, fuel system, the critical part of the ground station, as well as the design and production of the main fuselage. The crafts’ length is 10 metres, width 12 metres and weight 7 tonnes. The first flight is planned for 2012.

Un buen amigo alguna vez sabiamente dijo: hormona mata neurona.

 

---

 

A good friend once wisely said: hormone beats neuron.

Por favor, no usar esta imagen sin mi permiso explícito.

Please, don´t use this picture without my explicit permission.

 

Proceso:

Arriba.- Izda. Microfotografía de Cerebro

( Sistema Límbico ) de

rata, mostrando una N.

Bipolar ( Dcha.) y un

elemento glial. ( Izda.).

Dcha. Escaneado en grises.

Abajo.- " Neurona Bipolar ama...".

Dibujo a mano.

 

Process:

Above.- Left.-Microphotography of rat

Brain ( Limbic System ),

showing a Bipolar Neur.

( Right ) and a Glial

element. ( Left )

Right.- Scanned in grey.

Below.- " Bipolar Neuron loves...".

Hand-Drawn.

Nearly everyone has had headache pain, and most of us have had it many times. A minor headache is little more than a nuisance that's relieved by an over-the-counter pain reliever, some food or coffee, or a short rest. But if your headache is severe or unusual, you might worry about stroke, a tumor, or a blood clot. Fortunately, such problems are rare. Still, you should know when a headache needs urgent care and how to control the vast majority of headaches that are not threatening to your health. Headache is defined as a pain arising from the head or upper neck of the body. The pain originates from the tissues and structures that surround the skull or the brain because the brain itself has no nerves that give rise to the sensation of pain (pain fibers). The thin layer of tissue (periosteum) that surrounds bones, muscles that encase the skull, sinuses, eyes, and ears, as well as thin tissues that cover the surface of the brain and spinal cord (meninges), arteries, veins, and nerves, all can become inflamed or irritated and cause headache. The pain may be a dull ache, sharp, throbbing, constant, intermittent, mild, or intense. Headaches can be more complicated than most people realize. Different kinds can have their own set of symptoms, happen for unique reasons, and need different kinds of treatment. Once you know the type of headache you have, you and your doctor can find the treatment that’s most likely to help and even try to prevent them.

Headache is the symptom of pain anywhere in the region of the head or neck. It occurs in migraines (sharp, or throbbing pains), tension-type headaches, and cluster headaches.Frequent headaches can affect relationships and employment. There is also an increased risk of depression in those with severe headaches. Headaches can occur as a result of many conditions whether serious or not. There are a number of different classification systems for headaches. The most well-recognized is that of the International Headache Society. Causes of headaches may include fatigue, sleep deprivation, stress, the effects of medications, the effects of recreational drugs, viral infections, loud noises, common colds, head injury, rapid ingestion of a very cold food or beverage, and dental or sinus issues.The pain you feel during a headache comes from a mix of signals between your brain, blood vessels, and nearby nerves. Specific nerves of the blood vessels and head muscles switch on and send pain signals to your brain. But it's not clear why these signals turn on in the first place. People often get headaches because of:

Illness: such as an infection, cold, or fever. They’re also common with conditions like sinusitis (inflammation of the sinuses), a throat infection, or an ear infection. In some cases, the headaches may be the result of a blow to the head or rarely, a sign of a more serious medical problem. Stress: Emotional stress and depression as well as alcohol use, skipping meals, changes in sleep patterns, and taking too much medication. Other causes include eyestrain and neck or back strain due to poor posture. Your environment, including secondhand tobacco smoke, strong smells from household chemicals or perfumes, allergens, and certain foods. Stress, pollution, noise, lighting, and weather changes are other possible triggers. Headaches, especially migraine headaches, tend to run in families. Most children and teens (90%) who have migraines have other family members who get them. When both parents have a history of migraines, there is a 70% chance that their child will also have them. If only one parent has a history of these headaches, the risk drops to 25%-50%.

Doctors don’t know exactly what causes migraines. A popular theory is that triggers cause unusual brain activity, which causes changes in the blood vessels there. Some forms of migraines are linked to genetic problems in certain parts of the brain. Too much physical activity can also trigger a migraine in adults. Treatment of a headache depends on the underlying cause, but commonly involves pain medication. Some form of headache is one of the most commonly experienced of all physical discomforts. About half of adults have a headache in a given year. Tension headaches are the most common, affecting about 1.6 billion people (21.8% of the population) followed by migraine headaches which affect about 848 million (11.7%)

 

There are more than two hundred types of headaches. Some are harmless and some are life-threatening. The description of the headache and findings on neurological examination, determine whether additional tests are needed and what treatment is best. Once you get your headaches diagnosed correctly, you can start the right treatment plan for your symptoms. The first step is to talk to your doctor about your headaches. She’ll give you a physical exam and ask you about the symptoms you have and how often they happen. It’s important to be as complete as possible with these descriptions. Give your doctor a list of things that cause your headaches, make them worse, and what helps you feel better. You can track details in a headache diary to help your doctor diagnose your problem. Most people don’t need special diagnostic tests. But sometimes, doctors suggest a CT scan or MRI to look for problems inside your brain that might cause your headaches. Skull X-rays are not helpful. An EEG (electroencephalogram) is also unnecessary unless you have passed out when you had a headache. If your headache symptoms get worse or happen more often despite treatment, ask your doctor to refer you to a headache specialist. If you need more information, contact one of the organizations in the resource list for a list of member doctors in your state.

 

Your doctor may recommend different types of treatment to try. She also might recommend more testing or refer you to a headache specialist. The treatment you need will depend on a lot of things, including the type of headache you get, how often, and its cause. Some people don’t need medical help at all. But those who do might get medications, counseling, stress management, and biofeedback. Your doctor will make a treatment plan to meet your specific needs. Once you start a treatment program, keep track of how well it’s working. A headache diary can help you note any patterns or changes in how you feel. Know that it may take some time for you and your doctor to find the best treatment plan, so try to be patient. Be honest with her about what is and isn’t working for you. Even though you’re getting treatment, you should still steer clear of the things you know can trigger your problem, like foods or smells. And it’s important to stick to healthy habits that will keep you feeling good, like regular exercise, enough sleep, and a healthy diet. Also, make your scheduled follow-up appointments so your doctor can see how you’re doing and make changes in the treatment program if you need them.

 

Headaches are broadly classified as "primary" or "secondary". Primary headaches are benign, recurrent headaches not caused by underlying disease or structural problems. For example, migraine is a type of primary headache. While primary headaches may cause significant daily pain and disability, they are not dangerous. Secondary headaches are caused by an underlying disease, like an infection, head injury, vascular disorders, brain bleed or tumors. Secondary headaches can be harmless or dangerous. Certain "red flags" or warning signs indicate a secondary headache may be dangerous.Occurring in about three of every four adults, tension headaches are the most common of all headaches. In most cases, they are mild to moderate in severity and occur infrequently. But a few people get severe tension headaches, and some are troubled by them for three or four times a week. The typical tension headache produces a dull, squeezing pain on both sides of the head. People with strong tension headaches may feel like their head is in a vise. The shoulders and neck can also ache. Some tension headaches are triggered by fatigue, emotional stress, or problems involving the muscles or joints of the neck or jaw. Most last for 20 minutes to two hours.If you get occasional tension-type headaches, you can take care of them yourself. Over-the-counter pain relievers such as acetaminophen (Tylenol, other brands) and nonsteroidal anti-inflammatories (NSAIDs) such as aspirin, naproxen (Aleve, other brands), or ibuprofen (Motrin, Advil, other brands) often do the trick, but follow the directions on the label, and never take more than you should. A heating pad or warm shower may help; some people feel better with a short nap or light snack. If you get frequent tension-type headaches, try to identify triggers so you can avoid them. Don't get overtired or skip meals. Learn relaxation techniques; yoga is particularly helpful because it can relax both your mind and your neck muscles. If you clench your jaw or grind your teeth at night, a bite plate may help. If you need more help, your doctor may prescribe a stronger pain medication or a muscle relaxant to control headache pain. Many people with recurrent tension-type headaches can prevent attacks by taking a tricyclic antidepressant such as amitriptyline (Elavil, generic). Fortunately, most people with tension-type headaches will do very well with simpler programs. 90% of all headaches are primary headaches. Primary headaches usually first start when people are between 20 and 40 years old. The most common types of primary headaches are migraines and tension-type headaches.[6] They have different characteristics. Migraines typically present with pulsing head pain, nausea, photophobia (sensitivity to light) and phonophobia (sensitivity to sound). Tension-type headaches usually present with non-pulsing "bandlike" pressure on both sides of the head, not accompanied by other symptoms. Other very rare types of primary headaches include: cluster headaches: This type is intense and feels like a burning or piercing pain behind or around one eye, either throbbing or constant. It’s the least common but the most severe type of headache. The pain can be so bad that most people with cluster headaches can’t sit still and will often pace during an attack. On the side of the pain, the eyelid droops, the eye reddens, pupil gets smaller or the eye tears. The nostril on that side runs or stuffs

They’re called “cluster headaches” because they tend to happen in groups. You might get them one to three times per day during a cluster period, which may last 2 weeks to 3 months. Each headache attack last 15 mins to 3 hours and often wakens the patient from sleep. The headaches may disappear completely (go into "remission") for months or years, only to come back again. Cluster headaches affect men 3-4 times more often than women.short episodes (15–180 minutes) of severe pain, usually around one eye, with autonomic symptoms (tearing, red eye, nasal congestion) which occur at the same time every day. Cluster headaches can be treated with triptans and prevented with prednisone, ergotamine or lithium. trigeminal neuralgia or occipital neuralgia: shooting face pain hemicrania continua: continuous unilateral pain with episodes of severe pain. Hemicrania continua can be relieved by the medication indomethacin.

primary stabbing headache: recurrent episodes of stabbing "ice pick pain" or "jabs and jolts" for 1 second to several minutes without autonomic symptoms (tearing, red eye, nasal congestion). These headaches can be treated with indomethacin. primary cough headache: starts suddenly and lasts for several minutes after coughing, sneezing or straining (anything that may increase pressure in the head). Serious causes (see secondary headaches red flag section) must be ruled out before a diagnosis of "benign" primary cough headache can be made. primary exertional headache: throbbing, pulsatile pain which starts during or after exercising, lasting for 5 minutes to 24 hours. The mechanism behind these headaches is unclear, possibly due to straining causing veins in the head to dilate, causing pain. These headaches can be prevented by not exercising too strenuously and can be treated with medications such as indomethacin. primary sex headache: dull, bilateral headache that starts during sexual activity and becomes much worse during orgasm. These headaches are thought to be due to lower pressure in the head during sex. It is important to realize that headaches that begin during orgasm may be due to a subarachnoid hemorrhage, so serious causes must be ruled out first. These headaches are treated by advising the person to stop sex if they develop a headache. Medications such as propranolol and diltiazem can also be helpful.

hypnic headache: moderate-severe headache that starts a few hours after falling asleep and lasts 15–30 minutes. The headache may recur several times during night. Hypnic headaches are usually in older women. They may be treated with lithium.

]

Headaches may be caused by problems elsewhere in the head or neck. Some of these are not harmful, such as cervicogenic headache (pain arising from the neck muscles). Medication overuse headache may occur in those using excessive painkillers for headaches, paradoxically causing worsening headaches.More serious causes of secondary headaches include: meningitis: inflammation of the meninges which presents with fever and meningismus, or stiff neck

bleeding inside the brain (intracranial hemorrhage)

subarachnoid hemorrhage (acute, severe headache, stiff neck WITHOUT fever) ruptured aneurysm, arteriovenous malformation, intraparenchymal hemorrhage (headache only)

brain tumor: dull headache, worse with exertion and change in position, accompanied by nausea and vomiting. Often, the person will have nausea and vomiting for weeks before the headache starts. temporal arteritis: inflammatory disease of arteries common in the elderly (average age 70) with fever, headache, weight loss, jaw claudication, tender vessels by the temples, polymyalgia rheumatica acute closed angle glaucoma (increased pressure in the eyeball): headache that starts with eye pain, blurry vision, associated with nausea and vomiting. On physical exam, the person will have a red eye and a fixed, mid dilated pupil. Post-ictal headaches: Headaches that happen after a convulsion or other type of seizure, as part of the period after the seizure (the post-ictal state) Gastrointestinal disorders may cause headaches, including Helicobacter pylori infection, celiac disease, non-celiac gluten sensitivity, irritable bowel syndrome, inflammatory bowel disease, gastroparesis, and hepatobiliary disorders. The treatment of the gastrointestinal disorders may lead to a remission or improvement of headaches.

 

The brain itself is not sensitive to pain, because it lacks pain receptors. However, several areas of the head and neck do have pain receptors and can thus sense pain. These include the extracranial arteries, middle meningeal artery, large veins, venous sinuses, cranial and spinal nerves, head and neck muscles, the meninges, falx cerebri, parts of the brainstem, eyes, ears, teeth and lining of the mouth.Pial arteries, rather than pial veins are responsible for pain production. Headaches often result from traction to or irritation of the meninges and blood vessels. The nociceptors may be stimulated by head trauma or tumors and cause headaches. Blood vessel spasms, dilated blood vessels, inflammation or infection of meninges and muscular tension can also stimulate nociceptors and cause pain. Once stimulated, a nociceptor sends a message up the length of the nerve fiber to the nerve cells in the brain, signaling that a part of the body hurts.

Primary headaches are more difficult to understand than secondary headaches. The exact mechanisms which cause migraines, tension headaches and cluster headaches are not known. There have been different theories over time which attempt to explain what happens in the brain to cause these headaches.

 

Migraines are currently thought to be caused by dysfunction of the nerves in the brain. This condition is accompanied by intense headaches. These headaches are often described as pounding, throbbing pain. They can last from 4 hours to 3 days and usually happen one to four times per month. Along with the pain, people have other symptoms, such as sensitivity to light, noise, or smells; nausea or vomiting; loss of appetite; and upset stomach or belly pain. When a child has a migraine, she may look pale, feel dizzy, and have blurry vision, fever, and an upset stomach.Migraines occur less often than tension-type headaches, but they are usually much more severe. They are two to three times more common in women than men, but that's small consolation if you are among the 6% to 8% of all men who have migraines. And since a Harvard study of 20,084 men age 40 to 84 reported that having migraines boosts the risk of heart attacks by 42%, men with migraines should take their headaches to heart. Neurologists believe that migraines are caused by changes in the brain's blood flow and nerve cell activity. Genetics play a role since 70% of migraine victims have at least one close relative with the problem. Migraine triggers. Although a migraine can come on without warning, it is often set off by a trigger. The things that set off a migraine vary from person to person, but a migraine sufferer usually remains sensitive to the same triggers. A small percentage of children's migraines include digestive symptoms, like vomiting, that happen about once a month. Previously, migraines were thought to be caused by a primary problem with the blood vessels in the brain.This vascular theory, which was developed in the 20th century by Wolff, suggested that the aura in migraines is caused by constriction of intracranial vessels (vessels inside the brain), and the headache itself is caused by rebound dilation of extracranial vessels (vessels just outside the brain). Dilation of these extracranial blood vessels activates the pain receptors in the surrounding nerves, causing a headache. The vascular theory is no longer accepted. Studies have shown migraine head pain is not accompanied by extracranial vasodilation, but rather only has some mild intracranial vasodilation.

Currently, most specialists think migraines are due to a primary problem with the nerves in the brain. Auras are thought to be caused by a wave of increased activity of neurons in the cerebral cortex (a part of the brain) known as cortical spreading depression followed by a period of depressed activity. Some people think headaches are caused by the activation of sensory nerves which release peptides or serotonin, causing inflammation in arteries, dura and meninges and also cause some vasodilation. Triptans, medications which treat migraines, block serotonin receptors and constrict blood vessels. People who are more susceptible to experience migraines without headache are those who have a family history of migraines, women, and women who are experiencing hormonal changes or are taking birth control pills or are prescribed hormone replacement therapy. Tension headaches are thought to be caused by activation of peripheral nerves in the head and neck muscles Cluster headaches involve overactivation of the trigeminal nerve and hypothalamus in the brain, but the exact cause is unknown.

 

en.wikipedia.org/wiki/Headache

Photograph taken at an altitude of Fifty eight metres at 20:35pm on an beautiful summer evening as the light faded away on Monday 19th July 2021, off Hythe Avenue and Chessington Avenue in Bexleyheath, Kent.

  

Here we see an adult Female Carrion Crow (Corvus corone) teaching the last of her three juveniles to swoop, soar and fly. She stayed close to it at all times, on occasions touching it with her wingtips to guide it and reassure it that she was there, an absolutely amazing sight to witness. Beauty and elegance from these magnificent birds that it's been my privilege to get close to for the last few months.

  

I've played my part in the rearing of the three babies, putting out food and water, gaining mum and dad's confidence, and what joy they have given me watching their progression. A passerine bird of the family Corvidae and the genus Raven (Higher classification: Corvus), which is native to western Europe and eastern Asia. It can grow to twenty inches in length with a wingspan of up to thirty nine inches.

  

Unashamedly I will shout it from the rooftops.

  

I LOVE CARRION CROW.

   

There, I've said it. Words I use to describe these amazing birds would include stunning, beautiful, bold, magnificent, intelligent and fantastic, loving, tender, victimized...... oh yes, and sometimes they can be brutal (Let's face it, Mother nature often is).

  

Recently I had a resident pair of Carrion crows who decided that my garden was theirs, playing a game of cat and mouse with a pair of cheeky Magpies (Pica pica) for dominance and food rights. The male crow actually flew in and 'winged' the magpies to make them leave, an incredible sight to witness. It was an honour and a privilege to be able to win their trust and they have given me so much pleasure this year being able to get within a few feet of them, and their three youngsters, to photograph and feed them, and they have reinforced my already deep admiration for a bird that is brimming with beauty, intelligence, confidence and also surrounded by myths, legend and prejudice.

  

So let's begin with a look back over history.

  

LEGEND AND MYTHOLOGY

  

Crows appear in the Bible where Noah uses one to search for dry land and to check on the recession of the flood. Crows supposedly saved the prophet, Elijah, from famine and are an Inuit deity. Legend has it that England and its monarchy will end when there are no more crows in the Tower of London. And some believe that the crows went to the Tower attracted by the regular corpses following executions with written accounts of their presence at the executions of Anne Boleyn and Jane Gray.

  

In Welsh mythology, unfortunately Crows are seen as symbolic of evilness and black magic thanks to many references to witches transforming into crows or ravens and escaping. Indian legend tells of Kakabhusandi, a crow who sits on the branches of a wish-fulfilling tree called Kalpataru and a crow in Ramayana where Lord Rama blessed the crow with the power to foresee future events and communicate with the souls.

  

In Native American first nation legend the crow is sometimes considered to be something of a trickster, though they are also viewed positively by some tribes as messengers between this world and the next where they carry messages from the living to those deceased, and even carry healing medicines between both worlds. There is a belief that crows can foresee the future. The Klamath tribe in Oregon believe that when we die, we fly up to heaven as a crow. The Crow can also signify wisdom to some tribes who believe crows had the power to talk and were therefore considered to be one of the wisest of birds. Tribes with Crow Clans include the Chippewa (whose Crow Clan and its totem are called Aandeg), the Hopi (whose Crow Clan is called Angwusngyam or Ungwish-wungwa), the Menominee, the Caddo, the Tlingit, and the Pueblo tribes of New Mexico.

  

The crow features in the Nanissáanah (Ghost dance), popularized by Jerome Crow Dog, a Brulé Lakota sub-chief and warrior born at Horse Stealing Creek in Montana Territory in 1833, the crow symbolizing wisdom and the past, when the crow had became a guide and acted as a pathfinder during hunting. The Ghost dance movement was originally created in 1870 by Wodziwob, or Gray Hair, a prophet and medicine man of the Paiute tribe in an area that became known as Nevada. Ghost dancers wore crow and eagle feathers in their clothes and hair, and the fact that the Crow could talk placed it as one of the sages of the animal kingdom. The five day dances seeking trance,prophecy and exhortations would eventually play a major part in the pathway towards the white man's broken treaties, the infamous battle at Wounded knee and the surrender of Matȟó Wanáȟtaka (Kicking Bear), after officials began to fear the ghost dancers and rituals which seemed to occur prior to battle.

  

Historically the Vikings are the group who made so many references to the crow, and Ragnarr Loðbrók and his sons used this species in his banner as well as appearances in many flags and coats of arms. Also, it had some kind of association with Odin, one of their main deities. Norse legend tells us that Odin is accompanied by two crows. Hugin, who symbolizes thought, and Munin, who represents a memory. These two crows were sent out each dawn to fly the entire world, returning at breakfast where they informed the Lord of the Nordic gods of everything that went on in their kingdoms. Odin was also referred to as Rafnagud (raven-god). The raven appears in almost every skaldic poem describing warfare.Coins dating back to 940's minted by Olaf Cuaran depict the Viking war standard, the Raven and Viking war banners (Gonfalon) depicted the bird also.

  

In Scandinavian legends, crows are a representative of the Goddess of Death, known as Valkyrie (from old Norse 'Valkyrja'), one of the group of maidens who served the Norse deity Odin, visiting battlefields and sending him the souls of the slain worthy of a place in Valhalla. Odin ( also called Wodan, Woden, or Wotan), preferred that heroes be killed in battle and that the most valiant of souls be taken to Valhöll, the hall of slain warriors. It is the crow that provides the Valkyries with important information on who should go. In Hindu ceremonies that are associated to ancestors, the crow has an important place in Vedic rituals. They are seen as messengers of death in Indian culture too.

  

In Germanic legend, Crows are seen as psychonomes, meaning the act of guiding spirits to their final destination, and that the feathers of a crow could cure a victim who had been cursed. And yet, a lone black crow could symbolize impending death, whilst a group symbolizes a lucky omen! Vikings also saw good omens in the crow and would leave offerings of meat as a token.

  

The crow also has sacred and prophetic meaning within the Celtic civilization, where it stood for flesh ripped off due to combat and Morrighan, the warrior goddess, often appears in Celtic mythology as a raven or crow, or else is found to be in the company of the birds. Crow is sacred to Lugdnum, the Celtic god of creation who gave his name to the city of Lug

  

In Greek mythology according to Appolodorus, Apollo is supposedly responsible for the black feathers of the crow, turning them forever black from their pristine white original plumage as a punishment after they brought news that Κορωνις (Coronis) a princess of the Thessalian kingdom of Phlegyantis, Apollo's pregnant lover had left him to marry a mortal, Ischys. In one legend, Apollo burned the crows feathers and then burned Coronis to death, in another Coronis herself was turned into a black crow, and another that she was slain by the arrows of Αρτεμις (Artemis - twin to Apollo). Koronis was later set amongst the stars as the constellation Corvus ("the Crow"). Her name means "Curved One" from the Greek word korônis or "Crow" from the word korônê.A similar Muslim legend allegedly tells of Muhammad, founder of Islam and the last prophet sent by God to Earth, who's secret location was given away by a white crow to his seekers, as he hid in caves. The crow shouted 'Ghar Ghar' (Cave, cave) and thus as punishment, Muhammad turned the crow black and cursed it for eternity to utter only one phrase, 'Ghar, ghar). Native Indian legend where the once rainbow coloured crows became forever black after shedding their colourful plumage over the other animals of the world.

  

In China the Crow is represented in art as a three legged bird on a solar disk, being a creature that helps the sun in its journey. In Japan there are myths of Crow Tengu who were priests who became vain, and turned into this spirit to serve as messengers until they learn the lesson of humility as well as a great Crow who takes part in Shinto creation stories.

  

In animal spirit guides there are general perceptions of what sightings of numbers of crows actually mean:

  

1 Crow Meaning: To carry a message from your near one who died recently.

 

2 Crows Meaning: Two crows sitting near your home signifies some good news is on your way.

 

3 Crows Meaning: An upcoming wedding in your family.

 

4 Crows Meaning: Symbolizes wealth and prosperity.

 

5 Crows Meaning: Diseases or pain.

 

6 Crows Meaning: A theft in your house!

 

7 Crows Meaning: Denotes travel or moving from your house.

 

8 Crows Meaning: Sorrowful events

  

Crows are generally seen as the symbolism when alive for doom bringing, misfortune and bad omens, and yet a dead crow symbolises potentially bringing good news and positive change to those who see it. This wonderful bird certainly gets a mixed bag of contradictory mythology and legend over the centuries and in modern days is often seen as a bit of a nuisance, attacking and killing the babies of other birds such as Starlings, Pigeons and House Sparrows as well as plucking the eyes out of lambs in the field, being loud and noisy and violently attacking poor victims in a 'crow court'....

  

There is even a classic horror film called 'THE CROW' released in 1994 by Miramax Films, directed by Alex Proyas and starring Brandon Lee in his final film appearance as Eric Draven, who is revived by a Crow tapping on his gravestone a year after he and his fiancée are murdered in Detroit by a street gang. The crow becomes his guide as he sets out to avenge the murders. The only son of martial arts expert Bruce Lee, Brandon lee suffered fatal injuries on the set of the film when the crew failed to remove the primer from a cartridge that hit Lee in the abdomen with the same force as a normal bullet. Lee died that day, March 31st 1993 aged 28.

  

The symbolism of the Crow resurrecting the dead star and accompanying him on his quest for revenge was powerful, and in some part based on the history of the carrion crow itself and the original film grossed more than $94 Million dollars with three subsequent sequels following.

  

TAKING A CLOSER LOOK

  

So let's move away from legend, mythology and stories passed down from our parents and grandparents and look at these amazing birds in isolation.

  

Carrion crow are passerines in the family Corvidae a group of Oscine passerine birds including Crows, Ravens, Rooks, Jackdaws, Jays, Magpies, Treepies, Choughs and Nutcrackers. Technically they are classed as Corvids, and the largest of passerine birds. Carrion crows are medium to large in size with rictal bristles and a single moult per year (most passerines moult twice). Carrion crow was one of the many species originally described by Swedish naturalist Carl Linnaeus (Carl Von Linne after his ennoblement) in his 1758 and 1759 editions of 'SYSTEMA NATURAE', and it still bears its original name of Corvus corone, derived from the Latin of Corvus, meaning Raven and the Greek κορώνη (korōnē), meaning crow.

  

Carrion crow are of the Animalia kingdom Phylum: Chordata Class: Aves Order: Passeriformes Family: Corvidae Genus: Corvus and Species: Corvus corone

  

Corvus corone can reach 45-47cm in length with a 93-104cm wingspan and weigh between 370-650g. They are protected under The Wildlife and Countryside Act 1981 in the United Kingdom with a Green UK conservation status which means they are of least concern with more than 1,000,000 territories. Breeding occurs in April with fledging of the chicks taking around twenty nine days following an incubation period of around twenty days with 3 to 4 eggs being the average norm. They are abundant in the UK apart from Northwest Scotland and Ireland where the Hooded crow (Corvus cornix) was considered the same species until 2002. They have a lifespan of around four years, whilst Crow species can live to the age of Twenty years old, and the oldest known American crow in the wild was almost Thirty years old. The oldest documented captive crow died at age Fifty nine. They are smaller and have a shorter lifespan than the Raven, which again is used as a symbol in history to live life to the full and not waste a moment!

  

They are often mistaken for the Rook (Corvus frugilegus), a similar bird, though in the UK, the Rook is actually technically smaller than the Carrion crow averaging 44-46cm in length, 81-99cm wingspan and weighing up to 340g. Rooks have white beaks compared to the black beaks of Carrion crow, a more steeply raked head to beak ratio and a longer, less curved beak than crows. There are documented cases in the UK of singular and grouped Rooks attacking and killing Carrion crows in their territory. Rooks nest in colonies unlike Carrion crows. Carrion crows have only a few natural enemies including powerful raptors such as the northern goshawk, the peregrine falcon, the Eurasian eagle-owl and the golden eagle which will all readily hunt them.

  

Regarded as one of the most intelligent birds, indeed creatures on the planet, studies suggest that Corvids cognitive abilities can rival that of primates such as chimpanzees and gorillas and even provide clues to understanding human intelligence. Crows have relatively large brains for their body size, compared to other animals. Their encephalization quotient (EQ) a ratio of brain to body size, adjusted for size because there isn’t a linear relationship is 4.1. That is remarkably close to chimps at 4.2 whilst humans are 8.1. Corvids also have a very high neuronal density, the number of neurons per gram of brain, factoring in the number of cortical neurons, neuron packing density, interneuronal distance and axonal conduction velocity shows that Corvids score high on this measure as well, with humans scoring the highest.

  

A corvid's pallium is packed with more neurons than a great ape's. Corvids have demonstrated the ability to use a combination of mental tools such as imagination, and anticipation of future events. They can craft tools from twigs and branches to hook grubs from deep recesses, they can solve puzzles and intricate methods of gaining access to food set by humans., and have even bent pieces of wire into hooks to obtain food. They have been proven to have a higher cognitive ability level than seven year old humans.

  

Communications wise, their repertoire of wraw-wraw's is not fully understood, but the intensity, rhythm, and duration of caws seems to form the basis of a possible language. They also remember the faces of humans who have hindered or hurt them and pass that information on to their offspring.

  

Aesop's fable of 'The Crow and the Pitcher, tells of a thirsty crow which drops stones into a water pitcher to raise the water level and enable it to take a drink. Scientists have conducted tests to see whether crows really are this intelligent. They placed floating treats in a deep tube and observed the crows indeed dropping dense objects carefully selected into the water until the treat floated within reach. They had the intelligence to pick up, weigh and discount objects that would float in the water, they also did not select ones that were too large for the container.

  

Pet crows develop a unique call for their owners, in effect actually naming them. They also know to sunbathe for a dose of vitamin D, regularly settling on wooden garden fences, opening their mouths and wings and raising their heads to the sun. In groups they warn of danger and communicate vocally. They store a cache of food for later if in abundance and are clever enough to move it if they feel it has been discovered. They leave markers for their cache. They have even learned to place walnuts and similar hard food items under car tyres at traffic lights as a means of cracking them!

  

Crows regularly gather around a dead fellow corvid, almost like a funeral, and it is thought they somehow learn from each death. They can even remember human faces for decades. Crows group together to attack larger predators and even steal their food, and they have different dialects in different areas, with the ability to mimic the dialect of the alpha males when they enter their territory!

  

They have a twenty year life span, the oldest on record reaching the age of Fifty nine. Crows can leave gifts for those who feed them such as buttons or bright shiny objects as a thank you, and they even kiss and make up after an argument, having mated for life.

  

In mythology they are associated with good and bad luck, being the bringers of omens and even witchcraft and are generally reviled for their attacks on baby birds and small mammals. They have an attack method of to stunning smaller birds before consuming them, tearing violently at smaller, less aggressive birds, which is simply down to the fact that they are so highly intelligent, and also the top of the food chain. Their diet includes over a thousand different items: Dead animals (as their name suggests), invertebrates, grain, as well as stealing eggs and chicks from other birds' nests, worms, insects, fruit, seeds, kitchen scraps. They are highly adaptable when food sources grow scarce. I absolutely love them, they are magnificent, bold, beautiful and incredibly interesting to watch and though at times it is hard to witness attacks made by them, I cannot help but adore them for so many other and more important reasons.

  

OBSERVATIONS ON THE PAIR IN MY GARDEN

  

Crows have been in the area for a while, but rarely had strayed into my garden, leaving the Magpies to own the territory. Things changed towards the end of May when a beautiful female Carrion crow appeared and began to take some of the food that I put down for the other birds. Within a few days she began to appear regularly, on occasions stocking up on food, whilst other times placing pieces in the birdbath to soften them. She would stand on the birdbath and eat and drink and come back over the course of the day to eat the softened food.

  

Shortly afterwardsds she brought along her mate, a tall and handsome fella, much larger than her who was also very vocal if he felt she was getting a little too close to me. By now I had moved from a seated position from the patio as an observer, to laying on a mat just five feet from the birdbath with my Nikon so that I could photograph the pair as they landed, scavenged and fed. She was now confident enough to let me be very close, and she even tolerated and recognized the clicking of the camera. At first I used silent mode to reduce the noise but this only allowed two shooting frame rates of single frame or continuous low frame which meant I was missing shots. I reverted back to normal continuous high frames and she soon got used to the whirring of the frames as the mirror slapped back and forth.

  

The big fella would bark orders at her from the safety of the fence or the rear of the garden, whilst she rarely made a sound. That was until one day when in the sweltering heat she kept opening her beak and sunning on the grass, panting slightly in the heat. I placed the circular water sprayer nearby and had it rotating so that the birdbath and grass was bathed in gentle water droplets and she soon came back, landed and seemed to really like the cooling effect on offer. She then climbed onto the birdbath and opened her wings slightly and made some gentle purring, cooing noises....

  

I swear she was expressing happiness, joy....

  

On another blisteringly hot day when the sprayer was on, she came down, walked towards it and opened her wings up running into the water spray. Not once, but many times.

  

A final observation came with the male and female on the rear garden fence. They sat together, locked beaks like a kiss and then the male took his time gently preening her head feathers and the back of her neck as she made tiny happy sounds. They stayed together like that for several minutes, showing a gentle, softer side to their nature and demonstrating the deep bond between them. Into July and the pair started to bring their three youngsters to my garden, the nippers learning to use the birdbath for bathing and dipping food, the parents attentive as ever.

  

I was privileged to be in close attendance as the last juvenile was brought down by the pair, taught to take food and then on a night in July, to soar and fly with it's mother in the evening sky as the light faded. She would swoop and twirl, and at regular intervals just touch the juvenile in flight with her wing tip feathers, as if to reassure it that she was close in attendance. What an amazing experience to view.

  

Corvus Corone.... magnificently misunderstood by some!

  

Paul Williams June 4th 2021

  

©All photographs on this site are copyright: ©DESPITE STRAIGHT LINES (Paul Williams) 2011 – 2021 & GETTY IMAGES ®

  

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Nikon D850 Focal length: 550mm Shutter speed: 1/1600s Aperture: f/7.1 iso640 Hand held with Tamron VC Vibration control set to ON (Position 1) 14 Bit uncompressed RAW NEF file size L (8256 x 5504 pixels) FX (36 x 24) Focus mode: AF-C AF-Area mode: 3D-tracking AF-C Priority Selection: Release. Nikon Back button focusing enabled 3D Tracking watch area: Normal 55 Tracking points Exposure mode: Manual exposure mode Metering mode: Matrix metering White balance on: Auto1 (4990k) Colour space: RGB Picture control: Neutral (Sharpening +2)

  

Tamron SP 150-600mm F/5-6.3 Di VC USD G2. Nikon GP-1 GPS module. Lee SW150 MKII filter holder. Lee SW150 95mm screw in adapter ring. Lee SW150 circular polariser glass filter.Lee SW150 Filters field pouch. Hoodman HEYENRG round eyepiece oversized eyecup.Mcoplus professional MB-D850 multi function battery grip 6960.Two Nikon EN-EL15a batteries (Priority to battery in Battery grip). Black Rapid Curve Breathe strap. My Memory 128GB Class 10 SDXC 80MB/s card. Lowepro Flipside 400 AW camera bag.

  

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LATITUDE: N 51d 28m 28.11s

LONGITUDE: E 0d 8m 10.63s

ALTITUDE: 58.00m

  

RAW (TIFF) FILE: 130.00MB NEF FILE: 90.5MB

PROCESSED (JPeg) FILE: 52.70MB

    

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PROCESSING POWER:

  

Nikon D850 Firmware versions C 1.10 (9/05/2019) LD Distortion Data 2.018 (18/02/20) LF 1.00

  

HP 110-352na Desktop PC with AMD Quad-Core A6-5200 APU 64Bit processor. Radeon HD8400 graphics. 8 GB DDR3 Memory with 1TB Data storage. 64-bit Windows 10. Verbatim USB 2.0 1TB desktop hard drive. WD My Passport Ultra 1tb USB3 Portable hard drive. Nikon ViewNX-1 64bit Version 1.4.1 (18/02/2020). Nikon Capture NX-D 64bit Version 1.6.2 (18/02/2020). Nikon Picture Control Utility 2 (Version 2.4.5 (18/02/2020). Nikon Transfer 2 Version 2.13.5. Adobe photoshop Elements 8 Version 8.0 64bit.

 

Nation of China systematically organizes itself to operate just like a brain, with each individual acting as a neuron (forming what has come to be called a "Blockhead")Chinese thought, including seemingly disparate fields such as geomancy or Feng shui, astrology, traditional Chinese medicin, as seen on "Floating Perspective," a technique which displaces the static eye of the viewer and highlights the differences between Chinese and Western modes of spatial representation. It summarizes these into three ways of minor, middle and great achievement, and illustrates each with charts, explanations and formulae..The 5-volumed Record of the Realization of Perfection by the Concourse of Immortals of West Mountain ( 《西山群仙會真記》 Xishan Qunxian Huizhen Ji ) was authored by Shi Jianwu and compiled by Li Song. Shi, whose title was "The Perfect Man of Huayang" ( 華陽真人 Huayang Zhenren ) lived during the Tang Dynasty. After his success in the national civil service examinations, Shi remained secluded as a Daoist on the West Mountain (present Jiangxi province) to cultivate the Dao. But some say that there were two men named "Shi Jianwu". the Comprehensive Annotated Bibliography of the Four Repositories ( 《四庫提要》 Siku Tiyao ) say some Daoists, under the name of Shi, wrote this book during the Jin and Yuan Dynasties. Still some hold that the book should have been completed no later than the Northern Song. The preface by the author states, the book, in secret accordance with earlier and later sages, imitating the number of five agents ( 五行 Wuxing ) and corresponding to the pure Yang one-breath ( 純陽一氣 Chunyang Yiqi ), expounds the mystery and supreme truth of the Zhong-Lu lineage in one five-volume book. The contents, similar to the Transmission of Dao by Zhongli Chuan and Lü Dongbin ( 《鍾呂傳道集》 Zhinglu Chuandao Ji ), explains the ways of inner alchemy, citing the Supreme Hidden Book ( 《太上隱書》 Taishang Yinshu ), Record of the Western Mountain ( 《西山記》 Xishan Ji ), Numinous Treasure Book of Inner Contemplation ( 《靈寶內觀經》 Lingbao Neiguan Jing ), the Book of Communion with the Mystery ( 《通玄經》 Tongxuan Jing ) and quotations of the immortals Ge, Yin and Lu. The theme, based on Three Ways Unified and Normalized ( 《參同契》 Cantong Qi ), expounds the theory about increasing and reducing fire ( 抽添 Choutian ) and the practice that leads to liberation.

  

en.daoinfo.org/wiki/Record_of_the_Realization_of_Perfecti...

getting some fresh air in Victoria Park

Edit: Hurray for the GFP Nobel Prize. Aside from just green, it is a wonderful rainbow of tools providing results...

 

20X magnification of the somatosensory cortex of a mouse brain slice. These little guys have green flourescent protein (GFP) from a jelly-fish expressed in a subset of their neurons. Layer V neuron cell bodies are the teardrop shaped things at the bottom and then the dendrite reaches up like a tree to then bifurcate near the top (pial surface).

Neuronal cells were cultured in a microfluidic device with channels to induce guidance of axons. We load the device with nanorods which penetrate into the cells and we study how those rods move inside the axons. Cells have been fixed and plastified with resin (thin layer plastification, procedure developed in our lab) such that both the rods and the cells are preserved and visible. In fact we can distinguish between the penetrating rods and those which just stay outside the cell.

 

Courtesy of Dr. Francesca Santoro , Stanford University - Chemistry Department

 

Image Details

Instrument used: Helios NanoLab

Voltage: 5kV

Detector: BSE

 

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En Biodiversidad virtual

 

y también Twiter

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De forma misteriosa y por ello todavía incomprendida, las amebas constructoras, que como Centropyxis modelan granito a grano su caparazón, sin planos, sin neuronas y sin manos, lo hacen siguiendo siempre el mismo patrón no escrito y ese patrón permite diferenciar a los numerosos géneros y a la gran variedad de especies que fabrican su casa siguiendo técnicas completamente distintas, siempre con un resultado sorprendente y hermoso del Arte de la Naturaleza.

 

Centropyxis es un género que reúne a poco más de una docena de especies, de contorno variado, pero casi siempre elíptico o circular, cuya cubierta está tapizada por diminutos granitos de arena o caparazones minúsculos de algunos organismos que como las diatomeas, tras vivir navegando un tiempo, han quedado depositados sobre el mullido fondo en una colección de joyas de naufragio que Centropyxis consigue recuperar para adornarse.

 

A veces Centropyxis se acicala rematando su casa con pequeñas torres de aguja que coronan sus contornos y cada una de las especies lo hace de una manera propia y singular. Sin embargo, todas tienen algo en común: la puerta por la que Centropyxis asoma su cuerpo al agua, como si fuese la puerta de una concha de caracol, suele ser circular como un ojo de buey y está situada en posición central.

 

...sin embargo, alguna de estas diminutas amebas, excéntrica y original, ha decidido probar suerte de una manera diferente y construir su casa con la puerta desplazada hacia un extremo y asomar así su cuerpo por ella como si lo hiciese desde la boca de un buzón y esa ameba excéntrica es la ameba de hoy Centropyxis platystoma.

  

Al igual que al resto de sus hermanas a Centropyxis platystoma también le encanta rebozarse de arena y si fuésemos de su tamaño, la confundiríamos con el fondo de las charcas en las que vive camuflada como si fuese el mismo lecho del agua.

 

Todo lo que encuentra en el fondo le sirve para ir construyendo, grano a grano, la casa que le dará cobijo y, así, de forma mágica, cristal a cristal de cuarzo fragmentos invisibles de limo van modelando la vasija que contiene su vida.

 

Centropyxis platystoma también es una ameba de gruesos brazos y cuerpo de agua, por eso necesita vivir protegida y es ella misma la que fabrica una casa con pared de doble muro: uno fino y suave, casi transparente, de materia orgánica en contacto con su cuerpo como una delicada camisa y otro sobre ella, armadura y abrigo de piedrecitas recogidas en el fondo.

 

Sin embargo, Centropyxis platystoma es excéntrica y construye a su manera especial la entrada de su casa, protegida además por encima por un arco en forma de abanico extendido, desde donde se asoma para ver la vida a su alrededor, ver sin vista...eso sí que es original y excéntrico y no sólo es habilidad de esta ameba excéntrica, todas ellas ven así.

 

Centropyxis platystoma vive entre los sedimentos alimentándose de pequeñas partículas de la materia orgánica contenida en ellos y se cita por vez primera desde esta galería para el Lago de Sanabria y quizá también para la Península Ibérica.

 

La imagen, tomada a 400 aumentos con la técnica de campo oscuro y polarización, procede de una muestra recogida a 4m de profundidad, en los fondos del Lago de Sanabria junto a la Isla de Moras el 7 de julio de 2015, por Laura, Mª José y Tomás desde el catamarán Helios Sanabria el primer catamarán del mundo propulsado por energía eólica y solar.

 

Más información.

 

Presentación ponencia congreso internacional de Limnología de la AIL

 

El Lago en Europa

 

Informes de contaminación en el Lago de Sanabria

 

Informe de evolución de la contaminación en el Lago de Sanabria

 

Reportaje en Tele5.

 

El Lago en TVE.

My Website : Twitter : Facebook : Instagram : Photocrowd

 

Another from last Thursdays visit to Canary Wharf Winter Lights Festival, this time it's 'Neuron' by Juan Fuentes.

 

This for me was one of the moer interesting installations but without my tripod I've not really been able to do it justice.

 

Click here to see more photos from Canary Wharf : www.flickr.com/photos/darrellg/albums/72177720298516472

 

From Canarywharf.com, "This newly commissioned installation is inspired by the intricate network of neural connections in the human brain. The vast three-dimensional structure composed of thousands of luminous filaments that interweave and intricately connect, aims to make the incredibly complex concept of the human brain into a tangible, and beautiful art object. Each filament represents a neuron, and their interconnection symbolizes neuronal synapses, where the transmission of electrical and chemical signals take place."

 

© D.Godliman

Close-up of neurons in a mouse with the neurodegenerative disease Niemann-Pick type C1. A major way to assess the impact of a potential therapeutic on the central nervous system is to assess cerebellar pathology in mice.

 

Credit: I. Williams, National Institute of Child Health and Human Development, NIH

nEUROn et Rafale M en vol au-dessus du porte-avions Charles de Gaulle.

Essais à la mer afin d'étudier l'utilisation d'un drone de combat dans un contexte naval.

 

© Dassault Aviation - A. Pecchi

2011/2/14.

Today's weather report said it was going to be sunny today.

But...

Another pareidolia found on a Mussel shell. Im amazed at what im finding on the surface of these shells which to the naked eye look fairly bland.

 

Taken on a set of bellows using my Nikon El-Nikkor 50mm 2.8 lens,Reversed.

  

Hand-drawn neurons edited on Photoshop

“I can never read all the books I want; I can never be all the people I want and live all the lives I want....I am horribly limited.”

― (Sylvia Plath) ―

 

When you spend the day trying to write, and the words don't come: the neurons start melting.

Neurons from a mouse spinal cord. S. Jeong, NICHD

LOOK KG 243 racer from late 1990's built with Columbus Neuron steel. Components are generally 2000's - but a mix of new and old.

 

Photo: Thomas Ohlsson Photography

 

www.thomasohlsson.com | 500px | Facebook | Flickr | Instagram

Pissenlit pris en macro

(c) 2014, Strutz et al, subject to a CC-BY 4.0 license (creativecommons.org/licenses/by/4.0/). Free to use and re-use, provided proper attribution is included.

 

Original research article can be found at dx.doi.org/10.7554/eLife.04147

Neuron e-scooter, for hire.

Feliz Día de San Valentín · Happy St Valentine's Day

Hormonal sentience, first described by Robert A. Freitas Jr., describes the information processing rate in plants, which are mostly based on hormones instead of neurons like in all major animals (except sponges). Plants can to some degree communicate with each other and there are even examples of one-way-communication with animals.

 

Acacia trees produce tannin to defend themselves when they are grazed upon by animals. The airborne scent of the tannin is picked up by other acacia trees, which then start to produce tannin themselves as a protection from the nearby animals. When attacked by caterpillars, some plants can release chemical signals to attract parasitic wasps that attack the caterpillars.

A similar phenomenon can be found not only between plants and animals, but also between fungus and animals. There exists some sort of communication between a fungus garden and workers of the leaf-cutting ant Atta sexdens rubropilosa. If the garden is fed with plants that are poisonous for the fungus, it signals this to the ants, which then will avoid fertilizing the fungus garden with any more of the poisonous plant.The Venus flytrap, during a 1- to 20-second sensitivity interval, counts two stimuli before snapping shut on its insect prey, a processing peak of 1 bit/s. Mass is 10-100 grams, so the flytrap's SQ is about +1. Plants generally take hours to respond to stimuli though, so vegetative SQs (Sentience Quotient) tend to cluster around -2.In theory even an organism with a hormonal system instead of a nervous system could be intelligent in some degree, but it would be an extremely slow brain, to say the least.And yet, at least higher plants are able to produce electrical signals, even if they do not use them in the same way animals do. František Baluška from the University of Bonn in Germany is one of the authorities on plant neurobiology.

en.wikipedia.org/wiki/Hormonal_sentience

 

Plants do not have a brain or neuronal network, but reactions within signalling pathways may provide a biochemical basis for learning and memory in addition to computation and problem solving.Controversially, the brain is used as a metaphor in plant intelligence to provide an integrated view of signalling.Plants respond to environmental stimuli by movement and changes in morphology. They communicate while actively competing for resources. In addition, plants accurately compute their circumstances, use sophisticated cost–benefit analysis and take tightly controlled actions to mitigate and control diverse environmental stressors. Plants are also capable of discriminating positive and negative experiences and of "learning" (registering memories) from their past experiences. Plants use this information to update their behaviour in order to survive present and future challenges of their environment.Plant physiology studies the role of signalling, communication, and behaviour to integrate data obtained at the genetic, molecular, biochemical, and cellular levels with the physiology, development, and behaviour of individual organisms, plant ecosystems, and evolution. The neurobiological view sees plants as information-processing organisms with rather complex processes of communication occurring throughout the individual plant organism. It studies how environmental information is gathered, processed, integrated and shared (sensory plant biology) to enable these adaptive and coordinated responses (plant behaviour); and how sensory perceptions and behavioural events are 'remembered' in order to allow predictions of future activities upon the basis of past experiences. Plants, it is claimed by some plant physiologists, are as sophisticated in behaviour as animals but this sophistication has been masked by the time scales of plants' response to stimuli, many orders of magnitude slower than animals'.It has been argued that although plants are capable of adaptation, it should not be called intelligence, as plant neurobiologists are relying primarily on metaphors and analogies to argue that complex responses in plants can only be produced by intelligence.[32]"A bacterium can monitor its environment and instigate developmental processes appropriate to the prevailing circumstances, but is that intelligence? Such simple adaptation behaviour might be bacterial intelligence but is clearly not animal intelligence." However, plant intelligence fits a definition of intelligence proposed by David Stenhouse in a book about evolution and animal intelligence where he described it as "adaptively variable behaviour during the lifetime of the individual".Critics of the concept have also argued that a plant cannot have goals once it is past the development stage of plantlet because, as a modular organism, each module seeks its own survival goals and the resultant whole organism behavior is not centrally controlled.[33] This view, however, necessarily accommodates the possibility that a tree is a collection of individually intelligent modules cooperating with, competing with and influencing each other, thus determining organism level behavior from the base up. The development into a larger organism whose modules must deal with different environmental conditions and challenges is not universal across plant species either, as smaller organisms might be subject to the same conditions across their bodies, at least, when the below and above ground parts are considered separately. Moreover, the claim that central control of development is completely absent from plants is readily falsified by apical dominance.Charles Darwin studied the movement of plants and in 1880 published a book The Power of Movement in Plants. In the book he concludes:It is hardly an exaggeration to say that the tip of the radicle thus endowed [..] acts like the brain of one of the lower animals; the brain being situated within the anterior end of the body, receiving impressions from the sense-organs, and directing the several movements.In philosophy, there are few studies of the implications of plant perception. Michael Marder put forth a phenomenology of plant life based on the physiology of plant perception.Paco Calvo Garzon offers a philosophical take on plant perception based on the cognitive sciences and the computational modeling of consciousness.Comparison to neurobiology:.A plant's sensory and response system has been compared to the neurobiological processes of animals. Plant neurobiology, an unfamiliar misnomer, concerns mostly the sensory adaptive behaviour of plants and plant electrophysiology. Indian scientist J. C. Bose is credited as the first person to research and talk about neurobiology of plants. Many plant scientists and neuroscientists, however, view this as inaccurate, because plants do not have neurons.The ideas behind plant neurobiology were criticised in a 2007 article published in Trends in Plant Science by Amedeo Alpi and other scientists, including such eminent plant biologists as Gerd Jürgens, Ben Scheres, and Chris Sommerville. The breadth of fields of plant science represented by these researchers reflects the fact that the vast majority of the plant science research community reject plant neurobiology. Their main arguments are that:"Plant neurobiology does not add to our understanding of plant physiology, plant cell biology or signaling".

"There is no evidence for structures such as neurons, synapses or a brain in plants".The common occurrence of plasmodesmata in plants which "poses a problem for signaling from an electrophysiological point of view" since extensive electrical coupling would preclude the need for any cell-to-cell transport of a ‘neurotransmitter-like’ compounds.The authors call for an end to "superficial analogies and questionable extrapolations" if the concept of "plant neurobiology" is to benefit the research community.There were several responses to the criticism clarifying that the term "plant neurobiology" is a metaphor and metaphors have proved useful on several previous occasions.[37][38] Plant ecophysiology describes this phenomenon.Parallels in other taxa. As described above in the case of a plant, similar mechanisms exist in a bacterial cell, a choanoflagellate, a fungal hypha, or a sponge, among the many other examples. All of these individual organisms of the respective taxa, despite being devoid of a brain or nervous system, are capable of sensing their immediate and momentary environment and responding accordingly. In the case of single-celled life, the sensory pathways are even more primitive in the sense that they take place on the surface of a single cell, as opposed to a network of many cells.

en.wikipedia.org/wiki/Plant_perception_(physiology)

  

Recent surprising similarities between plant cells and neuronsPlant cells and neurons share several similarities, including non-centrosomal microtubules, motile post-Golgi organelles, separated both spatially/structurally and functionally from the Golgi apparatus and involved in vesicular endocytic recycling, as well as cell-cell adhesion domains based on the actin/myosin cytoskeleton which serve for cell-cell communication. Tip-growing plant cells such as root hairs and pollen tubes also resemble neurons extending their axons. Recently, surprising discoveries have been made with respect to the molecular basis of neurodegenerative disorders known as Hereditary Spastic Paraplegias and tip-growth of root hairs. All these advances are briefly discussed in the context of other similarities between plant cells and neurons.There are very prominent similarities between tip-growing plant cells and the extending axons of neurons. However, recent advances reveal that these visible similarities stretch beyond the tip-growing plant cells and include plant tissue cells generating action potentials3 and accomplishing vesicle trafficking and recycling, typically at actin/myosin enriched cell-cell adhesion domains resembling neuronal synapses. Moreover, plant cells and neurons are similar from the cellular perspective, when most of their microtubules and Golgi apparatus organelles are not associated with the perinuclear centrosomes.In plant cells, Golgi stacks and Trans-Golgi Networks (TGNs) are motile organelles extending through the whole plant cells. Similarly in neurons centrosome-independent cortical microtubules are abundant in axons. They transport, among other cargo, so-called Golgi Outposts—which correspond to the TGNs of plant cells toward neuronal synapses. In both plant cells and neurons, TGNs act as independent organelles separated both spatially/structurally and functionally from the Golgi apparatus.Intriguingly, similarly as in neurons, also the TGN of plant cells is the inherent part of the endosomal/vesicular recycling pathways, supporting the dynamic and communicative nature of plant synapses.Plant action potentials (electric spikes) run in an axial direction, along the longitudinal axis of any plant organ, and the highest spike activity was scored in the transition zone of the root apex in maize.Hereditary spastic paraplegia (HSP) represents a heterogeneous group of genetic neurodegenerative disorders affecting the longest neurons of the human body, extending from the brain along the spinal cord /down to the legs.21 In the HSP disorders, axons of these long neurons degenerate causing problems in controlling leg muscles. One of the major genes in which mutation results in the HSP is Atlastin. Recent study has reported that Atlastin is homologous to the RHD3 protein of Arabidopsis.RHD3 protein is essential for proper growth and development of root hairs in Arabidopsis.Moreover, RHD3 is also important for the proper arrangement of root cell files which underlies the direction of root growth.In order to maintain their ordered cell files, root apex cross-walls (plant root synapses) perform active vesicle recycling. Both Arabidopsis RHD3 and Drosophila Atlastin are important for shaping tubular ER networks.RHD3 is also known to be required for the proper arrangement of the actin cytoskeleton and cell wall maintenance via vesicle trafficking.Moreover, similarly as Atlastin in neurons,RHD3 is important for the GA morphogenesis in plant cells too Importantly, both RHD3 and Atlastin are implicated in membrane tubulation and vesiculation whereas rhd3 mutant line emerges to be less active in endocytic internalization of FM endocytic tracer.Drosophila Atlastin regulates the stability of muscle microtubules and is required for both the axonal maintenance and synapse development. All this suggest that Arabidopsis emerges as an attractive and useful model object for investigations of mechanisms underlying HSP disorders in humans.Glutamate is one of the best understood and the most widespread excitatory .neurotransmitter which is perceived via glutamate receptors at brain synapses in animals and humans. These neuronal receptors have, in fact, deep evolutionary origin in prokaryotic bacteria, and are present also in plants., Importantly, the plant glutamate receptors have all the features of neuronal ones, and glutamate induces plant action potentials., All this strongly suggest that glutamate serves in neurotransmitter-like cell-cell communication in plants too. Interestingly in this respect, especially the root apices are target of the neuronal-like activity of glutamate in plants, with effects on cell development, root growth, morphogenesis, and behavior. The transition zone cells, localized between the apical meristem and basal cell elongation zone, respond to glutamate with rapid depolarization of the plasma membrane and this response is blocked by a specific antagonist of ionotropic glutamate receptors, 2-amino-5-phosphonopentanoate.Cells of the transition zone, also known as the distal elongation zone or the basal meristem, are crucial for root primordia priming,and exogenous glutamate is known to decrease primary root growth and increase lateral root proliferation.Beta-N-methylamino-L-alanine (BMAA) is a neurotoxic amino acid, derived from cycads, which is well-known to act as agonists and antagonists of mammalian glutamate receptors. BMAA inhibits root growth, cotyledon opening, and it stimulates elongation of light-grown hypocotyls in Arabidopsis.BMAA affects growth of Arabidopsis organs at very low concentrations, and these BMAA-induced effects are reversed by the addition of glutamate.This is consistent with a scenario wherein BMAA acts to block plant-specific glutamate receptors.Similarly to glutamate, aluminium also induces very rapid plasma membrane depolarization specifically in cells of the root apex transition zone. Moreover, glutamate and aluminium both induce rapid and strong calcium spikes with unique signatures in cells of the transition zone.These root cells represent the primary target for the aluminium toxicity in plants, whereas aluminium is not toxic to root cells which have already entered the rapid elongation region.Similarly, although aluminium is not so toxic in most plant cells, neuronal-like tip-growing root hairs and pollen tubes1,2 are sensitive to aluminium similarly as are the transition zone cells. In these latter cells, aluminium is specifically internalized via endocytosis. Internalized endocytic aluminium interferes with vesicle trafficking/recycling and endocytosis, inhibiting the PIN2-driven basipetal auxin transport in the transition zone of root apices.Aluminium targets specifically the auxinsecreting plant synapses and affects the polar auxin-transport-based root cell patterning. Moreover, aluminium affects also nitric oxide (NO) production which is highest in cells of the the distal portion of the transition zone. Importantly, the rapidly elongating root cells are not sensitive towards aluminium and neither is there internalization of aluminium into rapidly elongating root cells. In support of the endocytosis of aluminium being the primary process linked to the aluminium toxicity in root cells, endocytosis of aluminium and its toxicity is lowered in the Arabidopsis mutant over-expressing the DnaJ domain protein auxillin which regulates the clathrin-based endocytosis.In animals and humans, neuronal cells are extremely sensitive towards aluminium which is internalized via endocytosis specifically in these cells. Aluminium was found to be enriched in lysosomes, similarly like Alzheimer’s amyloid β-peptide plaque depositions. These are also internalized from cell surface and aluminium was reported to inhibit their degradation.In conclusion, in both transition zone root cells and neurons, endocytosis of aluminium emerges as relevant to its high biotoxicity. In plants, the aluminium toxicity is the most important limiting factor for crop production in acid soil environments worldwide. Further studies on these cells might give us crucial clues not only for plant biology and agriculture but also for our still limited understanding of the Alzheimer disease. In line with the original proposal of Charles and Francis Darwin, root apices of plants represent neuronal/anterior pole of plant bodies

www.ncbi.nlm.nih.gov/pmc/articles/PMC2884105/

Graphical representation of a neuron.

"Neuronale Strukturen" in einem gefrorenen See bei Rottweil / Germany

This striking Vivid installation illustrates the complexity and connectivity of the human brain. It's the creation of industrial design firm Amigo and Amigo and technology company S1T2, and explores the effects of Alzheimer's disease on electrochemical activity in the brain (not to mention the camera's colour sensors).

 

Touching the sculpture triggers a colour response. Purple light represents the emergence of Alzheimer’s disease. When the purple path reaches a neuron, it disconnects, signifying that memories will be lost.

 

For the technically minded, "Affinity" features Onled LED's lighting and 19 Intel Galileo boards in an interactive display using over 56 colours, 114 capacitive sensors and 4200 meters of LED strip lighting. There are 70 balls each with six sensors, and about 48 arms.

lichen, jedediah smith redwoods state park, ca

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