View allAll Photos Tagged extensor
After nearly a month of foot pain, I finally hobbled over to UHS and got diagnosed with extensor tendonitis.
The pain isn't so bad that it keeps me from getting around, obviously, but it's bad enough that it makes me limp sometimes, which of course makes my OTHER leg start to hurt (plus I look like Lurch or something staggering around campus, heh), and the whole thing was getting tedious enough that I was willing to brave the UHS waiting room.
I was strictly instructed to ice the offending foot 2-3 times a day for 20 minutes each time, take 2 Advil three times a day to reduce the swelling of the tendon sheath, and to at all costs AVOID WALKING AROUND OR STANDING AS MUCH AS POSSIBLE.
Since I had been walking on it for so long before getting it looked at, and because the pain, 'though bearable, was so sharp, the doctor was concerned that the tendon sheath had been damaged and might, worse case scenario, be on its way to rupture.
The icing I can handle, but I really, REALLY hate taking that much pain med. And of course the walking thing is practically impossible. I don't have a car. I walk EVERYWHERE. EVERY DAY. What am I supposed to do?
I have a follow-up appointment on Dec. 13, and if no improvement has been made I have been threatened with the possibility of a Boot, so I guess I'll be downing that Advil, like it or not. Can't do a bloody thing about the walking, though.
Sidenote the first: The doctor wanted to set me up with the University physical therapy system, but of course they're booked solid from now through the end of all time, so that's fairly impossible. The system works!
Sidenote the second: We have no idea how this happened. It's unlikely my shoes are the problem, and it's not as though I'm training for a marathon over here. I made the mistake of mentioning the fact that I stand for long periods of time at an easel to paint, and the doctor seized on that as an explanation, but I HIGHLY doubt this.
Paige was diagnosed with West syndrome or West's Syndrome (September 2010) it is an uncommon to rare epileptic disorder in infants.
The syndrome is age-related, generally occurring between the third and the twelfth month, generally manifesting around the fifth month. There are various causes ("polyetiology"). The syndrome is often caused by an organic brain dysfunction whose origins may be prenatal, perinatal (caused during birth) or postnatal.
It is either Cryptogenic or Idiopathic.
Cryptogenic
When a direct cause cannot be determined but the child has other neurological disorder, the case is referred to as cryptogenic West syndrome, where an underlying cause is most likely but, even with modern means, cannot be detected. Currently the cryptogenic group is often combined with idiopathic while referred to as "cryptogenic".
Sometimes multiple children within the same family develop West syndrome. In this case it is also referred to as cryptogenic, in which genetic and sometimes hereditary influences play a role. There are known cases in which West syndrome appears in successive generations in boys; this has to do with X-chromosomal heredity.
[edit] Idiopathic
Occasionally the syndrome is referred to as idiopathic West syndrome, when a cause cannot be determined. Important diagnostic criteria are:
Regular development until the onset of the attacks or before the beginning of the therapy no pathological findings in neurological or neuroradiological studies no evidence of a trigger for the spasms
Those are becoming rare due to modern medicine.
Clinical presentation
The epileptic seizures which can be observed in infants with West syndrome fall into three categories, collectively known as infantile spasms. Typically, the following triad of attack types appears; while the three types usually appear simultaneously, they also can occur independently of each other:
Lightning attacks: Sudden, severe myoclonic convulsions of the entire body or several parts of the body in split seconds, and the legs in particular are bent (flexor muscle convulsions here are generally more severe than extensor ones).
Nodding attacks: Convulsions of the throat and neck flexor muscles, during which the chin is fitfully jerked towards the breast or the head is drawn inward.
Salaam or jackknife attacks: a flexor spasm with rapid bending of the head and torso forward and simultaneous raising and bending of the arms while partially drawing the hands together in front of the chest and/or flailing. If one imagined this act in slow motion, it would appear similar to the oriental ceremonial greeting (Salaam), from which this type of attack derives its name.
Paige was first put on Vigabatrin which she outgrew a month later. She was admitted again into Kingston General Hospital to begin a treatment called ACTH.
Therapy
Compared with other forms of epilepsy, West syndrome is difficult to treat. To raise the chance of successful treatment and keep down the risk of longer-lasting effects, it is very important that the condition is diagnosed as early as possible and that treatment begins straight away. However, there is no guarantee that therapy will work even in this case.
Insufficient research has yet been carried out into whether the form of treatment has an effect upon the long-term prognosis. Based on what is known today, the prognosis depends mainly on the cause of the attacks and the length of time that hypsarrhythmia lasts. In general it can be said that the prognosis is worse when the patient does not react as well to therapy and the epileptic over-activity in the brain continues. Treatment differs in each individual case and depends on the cause of the West syndrome (etiological classification) and the state of brain development at the time of the damage.
Due to their side-effects, two drugs are currently being used as the first-line treatment: ACTH and Vigabatrin.
[edit] ACTH
ACTH - Use primarily in United States
Side effects are: Weight gain, especially in the trunk and face, hypertension, metabolic abnormalities, severe irritability, osteoporosis, sepsis, and congestive heart failure.
[edit] Vigabatrin
Vigabatrin (Sabril) - Approved in several countries, including most of Europe, Canada, Mexico, and more recently the United States.
Side effects are: Somnolence, headache, dizziness, fatigue, weight gain, decreased vision or other vision changes
Vigabatrin is known for being effective, especially in children with tuberous sclerosis, with few and benign side effects. But due to some recent studies[4] showing visual field constriction (loss of peripheral vision), it was not approved in the United States until mid-2009. It is currently debated that a short use (6 months or less) of Vigabatrin will not affect vision. Also, considering the effect of frequent seizures on day to day life and mental development, some parents prefer to take the risk of some vision loss.
Other
When those two are proving ineffective, other drugs may be used in conjunction or alone. From those, corticosteroids (prednisone) are often used. In Japan, there is a good experience with pyridoxine therapy. Further, topiramate (Topamax), lamotrigine (Lamictal), levetiracetam (Keppra) and zonisamide (Zonegran) are amongst those drugs most widely used.
The ketogenic diet has been shown to be effective in treating infantile spams,[5] up to 70% of children having a 50% or more reduction in seizure.
You can read more on Infantile Spasms by going to : en.wikipedia.org/wiki/West_syndrome
You can also view my youtube channel:
www.youtube.com/user/Shaeree624
I have uploaded videos for those who wonder what the Syndrome is and looks like.
I posted pictures of my beautiful baby girl because people need to understand being chubby or overweight may not always be caused by overfeeding. My daughters was caused by the ACTH treatment which is a steroid.
Details best viewed in Original Size.
I photographed this squirrel raiding the food bowl of the Red Panda at the Denver Zoo. The resident Red Pandas seemed oblivious to the theft of their food. According to Wikipedia, The fox squirrel, also known as the eastern fox squirrel or Bryant's fox squirrel, is the largest species of tree squirrel native to North America. Despite the differences in size and coloration, they are sometimes mistaken for American Red Squirrels or Eastern Gray Squirrels in areas where the species co-exist. The squirrel's total body length measures 17.7 to 27.6 inches (45 to 70cm), tail length is 7.9 to 13.0 inches (20 to 33cm), and they range in weight from 1.1 to 2.2 pounds (500 to 1,000 grams. There is no sexual dimorphism in size or appearance. Individuals tend to be smaller in the west. There are three distinct geographical phases in coloration: In most areas the animal's upper body is brown-grey to brown-yellow with a typically brownish-orange underside, while in eastern regions such as the Appalachians there are more strikingly patterned dark brown and black squirrels with white bands on the face and tail. In the south can be found isolated communities with uniform black coats. To help with climbing, they have sharp claws, developed extensors of digits and flexors of forearms, and abdominal musculature. Fox squirrels have excellent vision and well-developed senses of hearing and smell. They use scent marking to communicate with other fox squirrels. Fox squirrels also have several sets of vibrissae, thick hairs or whiskers that are used as touch receptors to sense the environment. These are found above and below their eyes, on their chin and nose, and on each forearm.
I had erroneously identified this as a Douglas Squirrel but was corrected by Aquila-chrysaetos (Bryant Olsen). Thank you, Bryant.
Jocelyn Olivier
The NeuroMuscular Reprogramming Network
Scoliosis Priority Protocols
Step 1. Torsion Imbalances Rt/Left at Hips and Lumbar
Test QL side to side with client supine and knees and hips flexed.
Test QL side to side with client prone and knees bent and hips neutral.
Find the top of QL weak in the open position on the side that lacks lordosis. Check for weak psoas on that side also.
Release the QL on the opposite side while engaging the QL that tested weak. Client learns to breathe and relax the side of the back that normally overworks, and, with this “hold/release” technique to re-engage the side that lacks lumbar curve and is not functioning properly.
Step 2. Right/Left Imbalances and Reactives along Lateral lines.
First check Quadratus Lumborum side to side in all reciprocal coordination positions.
Then Bottom of QL on short side to top of QL on over developed side (lacking lumber lordosis).
Lats on overdeveloped side of thorax will be weak, reactive to same side QL and/or upper trapezius.
Step 3. Sequencing Back Extensors
Disparities in development are clearly seen between 2 sides of erector spinae.
Test right to left side extensors—Release overdeveloped thoracic segment.
Re-do, refine test for superior segments : 1 Cervical 2 Thoracic
Test top to bottom for each side.
Test bottom to top. Then check diagonals.
Step 4. Torsion Imbalances Rt/Left at Shoulders:
All upper thoracic rotators (trapezius, rhomboids, latissimus dorsi and deep rotators and multifidi) will be weak on the side opposite the short side of the thorax, reactive to short side thoracic extensors.
Using arms braced together as a rudder to turn the thorax, notice the imbalances in twisting through the thoracic spine.
Step 5. Right/Left lateral flexion at neck, thorax and waist:
Next: Check Lateral Bending of thorax only (“the candle”)
Check Rt/Lt lateral cervical flexion for reactives.
Check cervicals to thorax and lumbar lateral flexion also.
Step 6. Right/left rotational imbalances in the neck
Check Rt/Lt rotation. Release the side that tests strong.
Check: scalenes to rotators and extensors. Release scalenes.
Check Upper thoracic extensors to rotation of the neck. Release thoracic extensors.
Step 4. Further explorations
All the extensor segments need to be checked with respect to the rotators. It will be found that some of the rotators are inhibiting the extensors and possibly the other way as well. (Eg., left rotation of upper thorax in preferred direction will inhibit right thoracic extension of the opposite side.
Other considerations with respect to the psoas:
Because the psoas has been disabled for so long it is predictable that thoracic flexion will be accomplished by the pec minor and internal obliques which pull the thorax forward thus inhibiting the mid and lower trapezius of the opposite side, creating the hump back look on one side of the thorax with a widening of the depth of the chest. Release right internal Obliques and left QL, along with the posterior inferior serratus.
Hip flexion on the side with the weak psoas will be compensated by tight rectus femoris creating the anterior tilt to pelvis on the same side and chronic pain in the same Sacro Iliac Joint. Test hip flexion to lateral flexion (QL to Psoas) on the flat side of the lumbar.
Once the psoas is firing reliably with respect to the QL it will be necessary to begin looking at the psoas function with the spine laterally flexed to one side or the other. In this “side bending” position, you may find the psoas once again inhibited.
Additional considerations
Thoracic Rotational stabilization while sitting
Problem: Right thorax weak rotationally.
Observation: thorax can’t rotate right without support from right inner thigh.
Therefore...
Test right upper thorax rotation to:
→Right adductors and flexors.
→Left post inf. Serratus
→Left Lower Trapezius and Latissimus Dorsi
Before right turning is functional and reliable these muscles will need to be released and re-programmed.
Canon EOS 60D - Sigma 70-300 en 300mm - Tubos extensores 32mm - Video RAW Magic Lantern - Apilado 72 cuadros.
Photoshop - Lightroom
motorchase.com/wp-content/uploads/2016/06/VLF-Destino-1-7...
Nós falamos recentemente que o antigo Fisker Karma vai voltar aos negócios quase como era, mas rebatizado como Karma Revero. Por “quase como era”, queremos dizer que ele será um carro elétrico com um extensor de autonomia, semelhante ao BMW i3. Mas há uma versão rebelde dele, também: ...
motorchase.com/pt/2016/06/vlf-destino-o-fisker-karma-com-...
Tokina 2,8 28mm Invertido , Extensores kenko 12mm y 20mm
Dos Flashes controlados inhalambricamente
A Pulso
-----
Lo que parece el tallo liso de una flor a simple vista, para una mariquita se convierte en un manto de espinas.
vapores en venta en bolsas de celofán,
las abre todas y nos sentamos los dos en el final.
El sol, sombrillas, sueños.
Extensores de agua y sal.
Fotos movidas para no olvidar.
No sólo los días que su sonrisa crece más
sino también las crecidas del río cuando ya no estás,
ya que el agua abre sus pantalones en el camino
y muy despacio el río le abre paso.
Aa alucinación a a a a alucinación alucinación.
Quieres ser natural,
planes de estar sentado en el sofá
sin pensar en la solución real
subiendo metros.
Millones de burbujas sin gas llegando al cielo,
llegando al cielo, llegando al hielo.
Pantalones flotando en brisas de celofán,
se mueven muy despacio parece que vayan a estallar.
A cámara lenta esas piernas duelen más.
Aa alucinación a a a a alucinación alucinación
aa alucinación a a a alucinación a a a alucinación alucinación.
Crépitation (Aperion). Letra y canción.
motorchase.com/wp-content/uploads/2016/03/Techrules-AT96-...
A Jaguar teve uma chance de ouro de deixar o queixo do mundo caído produzindo o C-X75. Mas decidiu deixá-la passar. Não foi o caso de uma empresa chinesa que gostou da idéia de ter uma turbina como um extensor de autonomia para seu supercarro elétrico. Foi assim que o GT96 e o AT96 foram desenvo...
motorchase.com/pt/2016/03/techrules-mostra-modelos-eletri...
The extensor retinaculum (dorsal carpal ligament), a strong, fibrous band, extending obliquely downward and medialward across the back of the wrist can be observed. It holds the tendons of the extensor muscles in place. It is continuous with the palmar carpal ligament, which is located on the anterior side of the forearm.
Spinal border cells from a Cooper and Sherrington publication from 1940. Cooper, S. & Sherrington, C. S. On ‘Gower’s tract and spinal border cells’. Brain 68, 123–134 (1940). Border cells were originally described by Gaskell, who gave the name to the neurons scattered at the periphery of the lateral column in the spinal cord of the alligator. In Sherrington’s box there are several spinal cord slides with labels pointing to cells at the edge of the spinal cord (or spinal border cells) (FIG. 1c,d in Molnar and Brown, 2010). These are the sections that led Sherrington, while working at Cambridge and at St Thomas’ Hospital, to describe a group of large nerve cells in the ventrolateral grey matter of the lumbar spinal cord of monkeys and cats as ‘outlying nerve cells’. Later, in one of his last publications from the University of Oxford, he called these neurons ‘spinal border cells’ because they were located predominantly along the lateral border of the ventral horn. Sherrington was interested in these cells because he suspected that they caused the sustained tonic inhibition of extensor muscle α-motor neurons in the cervical enlargement. Only much later were these cells identified as spinocerebellar tract neurons. Acute spinal injuries caudal to the cervical enlargement and cranial to border-cell neurons result in sudden deprivation of tonic inhibition of cervical enlargement neurons and cause their excitation. This excitation results in the extensor hypertonia observed in the thoracic limbs. Because Schiff described this syndrome in amphibian spinal cord before Sherrington, it is usually referred to as the Schiff–Sherrington phenomenon. This work provides an excellent example of the way Sherrington combined anatomical and physiological approaches to understand the interactions among spinal circuits that regulate reflex action by inhibition.
Transcription: Spinal border cells; large outlying cells in [good?] set[illeg.] C.S.S
For more about CSlide, go to: history.medsci.ox.ac.uk/cslide.
This video illustrates mallet finger non-splint treatment with an exercise technique developed by Valdas Macionis, MD, PhD. The technique is based on frequent hold-relax tip-to-tip power pinch exercises. Splint and surgery related problems can be avoided. Complete or almost complete recovery of extension can be achieved. The technique does not preclude further splinting or surgical tendon repair.
Mallet finger is a deformity caused by traumatic loss of continuity between the extensor apparatus and the distal phalanx. The injury may involve just the terminal extensor tendon or phalangeal bone fracture with or without wound. Untreated mallet finger may result in stiffness and osteoarthritis of the distal interphalangeal (DIP) joint and secondary swan-neck deformity (hyper-extension at the proximal IP joint and flexion at the DIP joint). The standard conservative and surgical treatments, all involving immobilization of the DIP joint in extension, are associated with frequent complications including deficit in range of motion of the DIP joint and soft tissue problems. It has been shown that splinting is effective at a considerable time after closed non-fracture mallet injury. Therefore, it is clinically sound to attempt treatment by exercises before proceeding to the immobilization methods. Treatment by exercises should be especially useful for old mallet deformities with stiffness of the DIP joint. The possible mechanism of the treatment may involve elongation of the central extensor slip and proximal slide of the digital extensor apparatus.
References:
1. Akeson WH, Amiel D, Abel MF, Garfin SR, Woo SL. Effects of immobilization on joints. Clin Orthop Rel Res. 1987;219:28-37.
2. Alla SR, Deal ND, Dempsey IJ. Current concepts: mallet finger. Hand (NY). 2014;9:138-144.
3. Altan E, Alp NB, Baser R, Yalçın L. Soft-Tissue Mallet Injuries: A Comparison of Early and Delayed Treatment. J Hand Surg Am. 2014;39:1982-1985.
4. Bloom JM, Khouri JS, Hammert WC. Current concepts in the evaluation and treatment of mallet finger injury. Plast Reconstr Surg. 2013;132:560e-566e.
5. Chao JD, Sarwahi V, Da Silva YS, Rosenwasser MP, Strauch RJ. Central slip tenotomy for the treatment of chronic mallet finger: an anatomic study. J Hand Surg Am. 2004;29:216-219.
6. Cheung JP, Fung B, Ip WY. Review on mallet finger treatment. Hand Surg. 2012;17:439-447.
7. Garberman SF, Diao E, Peimer CA. Mallet finger: results of early versus delayed closed treatment. J Hand Surg Am. 1994;19:850-852.
8. Geyman JP, Fink K, Sullivan SD. Conservative versus surgical treatment of mallet finger: a pooled quantitative literature evaluation. J Am Board Fam Pract. 1998;11:382-390.
9. Gruber JS, Bot AG, Ring D. A prospective randomized controlled trial comparing night splinting with no splinting after treatment of mallet finger. Hand (N Y ). 2014;9:145-150.
10. Grundberg AB, Reagan DS. Central slip tenotomy for chronic mallet finger deformity. J Hand Surg Am. 1987;12:545-547.
11. Handoll HH, Vaghela MV. Interventions for treating mallet finger injuries. Cochrane Database Syst Rev. 2004;:CD004574.
12. LaStayo PC, Cass R. Continuous passive motion for the upper extremity: why, when, and how. In: Mackin EJ, Callahan AD, Skirven TM, Schneider LH, Osterman AL, eds. Rehabilitation of the Hand and upper extremity. 5th ed. St Louis, MO: Mosby; 2002:1764-1778.
13. Macionis V. Self-regulated frequent power pinch exercises: a non-orthotic technique for the treatment of old mallet deformity. J Hand Ther. 2015;28(4):433-6.
14. Macionis V. Is Diagrammatic Goniometry Feasible for Finger ROM Evaluation and Self-evaluation? Clin Orthop Relat Res. 2013;471(6):1894-903.
15. Macionis V. A local adhesive finger splint. J Hand Surg Am. 2001; 26A(5):962-964.
'Muscles du Pied. Plan Dorsal Superficiel.' (Muscles of the Foot. Superficial.) This plate shows the superficial muscles of the foot of an adult. Tibia, peroneus tertius, tibialis anterior, extensor digitorum brevis muscle, flexor digitorum longus etc.
Esta foto mostra Ayrton concentrado no grid de largada para o que seria a sua ultima corrida diante da torcida Brasileira em 1994 em Interlagos.
Neste mesmo ano ocorreu sua trágica morte em Imola.
Perdemos o melhor piloto de todos os tempos, nunca superado até hoje.
Olimpus OM101 - Lente 300mm com tubo extensor - ISO 1600
Spiders (order Araneae) are air-breathing arthropods that have eight legs and chelicerae with fangs that inject venom. They are the largest order of arachnids and rank seventh in total species diversity among all other groups of organisms.Spiders are found worldwide on every continent except for Antarctica, and have become established in nearly every habitat with the exception of air and sea colonization. As of 2008, at least 43,678 spider species,and 109 families have been recorded by taxonomists; however, there has been confusion within the scientific community as to how all these families should be classified, as evidenced by the over 20 different classifications that have been proposed since 1900.
Anatomically, spiders differ from other arthropods in that the usual body segments are fused into two tagmata, the cephalothorax and abdomen, and joined by a small, cylindrical pedicel. Unlike insects, spiders do not have antennae. In all except the most primitive group, the Mesothelae, spiders have the most centralized nervous systems of all arthropods, as all their ganglia are fused into one mass in the cephalothorax. Unlike most arthropods, spiders have no extensor muscles in their limbs and instead extend them by hydraulic pressure.
Equipamentos que utilizo para fotografar em macro, composto por:
Canon Rebel XT;
Tubo extensor (somente anel 2); 15 reais (dealextreme)
Adaptador - anel inversor de lente; 12 reais (ebay)
Adaptador - Step-up 52/58mm; 19 reais (centro fotográfico)
Lente Sigma 35-70mm (montada invertida); 120 reais (Pedro Cine Foto)
Yongnuo OC-E3 Off-Camera Shoe Cord 60 reais
Flash bracket DIY
Flash Xing-Ling Digital Concepts;
Difusor - DIY;
Com esse setup montato, por de 230 reais(excluindo a câmera e o flash que são do meu irmão) eu consigo ampliação de um pouco menos de 1:2 (0,43x) até pouco mais que 3:1 (3,17x).
Para ver o equipamento desmontado clique no link abaixo:
www.flickr.com/photos/mega_vet/4967076661/in/set-72157625...
Vidro, tecido preto por baixo, sala escura. Flash Canon SB 580 EX II com cabo extensor. O tecido usado não é o ideal, pois reflete um pouco de luz.
Nada criativo, já vi isso em outras galerias e, para piorar não vou perguntar quem nasceu primeiro.