International Journal of Life Sciences and Biotech
Plant Disease Resistance Proteins: R-gene Products as Immune Defense Molecules
Plant Disease Resistance Proteins: R-gene Products as Immune Defense Molecules
Author: Berna Baş
Abstract: Disease resistance-related R genes encoding NBS-LRR proteins are functional in systemic acquired immunity, briefly referred as ETI, in plant pathology. Upon infection of pathogens at first stage of plant immune responses, if plant pathogens subvert PTI immunity that activated by cell membrane surface receptor, then ETI immunity is initiated the relay to second step of defense. Also when pathogen effectors are directly translocatedinto cell cytoplasm across host membranes where can be faced with plant R proteins, ETI immunity develops faster and stronger than PTI efficacy. A great number of pathogen effectors are directly or indirectly reacted with R-gene proteins in similar to epitope-paratope structural interaction. With what kind of mechanisms do the plants that show similar immune responses to all known biotic agents recognize the effectors of a wide variety of pathogenic organisms? However, many approaches are available involved in molecular mechanisms of intracellular pattern-recognition receptors in plants, findings for each mechanism have been obtained from specific workings of personally researchers. It is not known how many different strategy models prevailing in molecular interaction of a wide variety of effector-receptor recognition are functional. So in the presented review article is focused just to molecular mechanism kinds of physical connection between many different effectors and intracellular receptors.
Keywords: Proteins related with plant disease resistance, R-gene
proteins, Effectorreceptor interactions, NBLRR proteins
Plant Disease Resistance Proteins: R-gene Products as Immune Defense Molecules
Plant Disease Resistance Proteins: R-gene Products as Immune Defense Molecules
Author: Berna Baş
Abstract: Disease resistance-related R genes encoding NBS-LRR proteins are functional in systemic acquired immunity, briefly referred as ETI, in plant pathology. Upon infection of pathogens at first stage of plant immune responses, if plant pathogens subvert PTI immunity that activated by cell membrane surface receptor, then ETI immunity is initiated the relay to second step of defense. Also when pathogen effectors are directly translocatedinto cell cytoplasm across host membranes where can be faced with plant R proteins, ETI immunity develops faster and stronger than PTI efficacy. A great number of pathogen effectors are directly or indirectly reacted with R-gene proteins in similar to epitope-paratope structural interaction. With what kind of mechanisms do the plants that show similar immune responses to all known biotic agents recognize the effectors of a wide variety of pathogenic organisms? However, many approaches are available involved in molecular mechanisms of intracellular pattern-recognition receptors in plants, findings for each mechanism have been obtained from specific workings of personally researchers. It is not known how many different strategy models prevailing in molecular interaction of a wide variety of effector-receptor recognition are functional. So in the presented review article is focused just to molecular mechanism kinds of physical connection between many different effectors and intracellular receptors.
Keywords: Proteins related with plant disease resistance, R-gene
proteins, Effectorreceptor interactions, NBLRR proteins