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Teflex is a revolutionary new type of disinfectant, capable of ensuring an environment free of pathogenic influence. The active ingredient involved in Teflex’s antimicrobial properties is a guanidine based copolymer. The germicidal function is attributed to the ability of the guanidine functional groups of the Teflex polymer bonding with the cellular membranes of pathogenic microbes. Upon bonding, the guanidine functional group infiltrates the cell’s cytoplasm and inhibits the cell’s regulatory functions. These functions include: enzymatic activity; the transfer of essential nutrients across the cell membrane; cellular respiration; and the multiplication of the microbe. The disruption of the cell’s regulatory tasks is such that the cells can no longer function the way that they need to in order to stay alive; ultimately resulting in cell death.

 

The following is an abbreviated description of the destruction process of the microorganisms:

- The absorption of the biocidal Teflex molecule through the microbes cell membrane.

- The diffusion of the absorbed Teflex molecules through cell walls.

- The destabilization and destruction of the cytoplasmic membrane.

- The cell contents’ disperse through the destabilized cell membrane, leading to,

- The death of the cell.

 

The ability of Teflex to connect to and interfere with the cellular membranes of microorganisms is attributed to the positively charged ions in the quarter-amino functional groups of the Teflex polymer. These positively charged sites of Teflex are attracted to the negatively charged sites located in the pathogen’s phospholipid bilayer. It is essential to note that these negatively charged sites of the microorganism’s membrane are required in the composition of the cellular membrane and its regulatory functions.

Thorough calculations indicate that each microbial cell reacts with 6x10^-8 (0.00000006) ml of the Teflex polymer.

Upon initial contact with the pathogen, there is an induction of electro-static interference between the negatively charged groups on the cellular membrane and the positively charged quarter-amino groups of the Teflex polymer. This leads to a change in orientation in the pathogen’s membrane. The Teflex macromolecule connects with a large number of phospholipids in the microbe’s membrane; this reaction takes place in a cooperative manner thereby evoking neutralization of the negative charge in the cell’s phospholipid bilayer. The resulting compound of Teflex stabilizes, leading to an irreversible change in electrostatic and hydrophobic interactions of the cell’s membrane; this results in further weakening of the membrane that is an essential element to the life of the microbe. Eventually, lipid-lipid interactions are weakened to a point where the membrane is so unstable that the microbe no longer functions properly. Another result of the Teflex fusion to the phospholipid bilayer is a break in the barrier and transportation functions of the microbe’s membrane.

 

As the Teflex polymer infiltrates even further, a hydrophobic fragment penetrates into the non-polar part of the cellular membrane, leading to a widening of the membrane and a disturbance of vanderwaal forces among phospholipid molecules. This leads to permeability changes, followed by a weakened membrane integrity, and eventually, the cell fragments and dies.

 

All of these effects are directed against viruses, gram-positive and gram-negative bacteria, fungi and protozoa; On the contrary, Teflex does not harm the cells of larger organisms (i.e human or animal cells). This is due to a difference in molecular structures and biochemical properties, (for example, our cells contain cholesterol which helps stabilize our membrane), and a difference in our cytoplasm and nucleus.

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