roger_barb
A617/0263
Herceptin drug and breast cancer cell. Molecular model of Herceptin (Trastuzumab), showing the molecule's secondary structure, seen as alpha helices (coils) and beta sheets (arrows). A breast cancer cell is at lower left (blue). Herceptin binds to HER2 (human epidermal growth factor receptor 2), a receptor that is over-expressed in some breast cancer cells, making them grow faster and more likely to spread. Herceptin blocks HER2, which kills the breast cancer cells. Around a fifth of breast cancers result from cells that over-express HER2, making them viable for Herceptin treatment.
TRASTUZUMAB
HERCEPTIN
DRUG
MOLECULE
PROTEIN
MONOCLONAL ANTIBODY
CELL
BIOCHEMISTRY
BIOLOGY
CHEMISTRY
MEDICINE
PHARMACOLOGY
IMMUNOLOGY
ONCOLOGY
ARTWORK
ILLUSTRATION
MOLECULAR MODEL
ABNORMAL
ALPHA HELIX
BALLS
BETA SHEET
BIOCHEMICAL
BIOLOGICAL
BREAST CANCER
CANCER DRUG
CANCER TREATMENT
CANCEROUS
CHEMICAL
CHEMOTHERAPY
COMPOUND
COMPOUNDS
CUT OUT
CUT-OUT
CUTOUT
DOTS
HELICES
HELIXES
HER2
HUMAN EPIDERMAL GROWTH FACTOR
MALIGNANCY
MALIGNANT
MEDICAL
MOLECULAR
MOLECULES
PHARMACOLOGICAL
PROTEINS
RECEPTOR 2
SCANNING ELECTRON MICROGRAPH
SECONDARY STRUCTURE
SEM
SHEETS
THERAPY
TREATMENT
TUMOUR
UNHEALTHY
A617/0263
Herceptin drug and breast cancer cell. Molecular model of Herceptin (Trastuzumab), showing the molecule's secondary structure, seen as alpha helices (coils) and beta sheets (arrows). A breast cancer cell is at lower left (blue). Herceptin binds to HER2 (human epidermal growth factor receptor 2), a receptor that is over-expressed in some breast cancer cells, making them grow faster and more likely to spread. Herceptin blocks HER2, which kills the breast cancer cells. Around a fifth of breast cancers result from cells that over-express HER2, making them viable for Herceptin treatment.
TRASTUZUMAB
HERCEPTIN
DRUG
MOLECULE
PROTEIN
MONOCLONAL ANTIBODY
CELL
BIOCHEMISTRY
BIOLOGY
CHEMISTRY
MEDICINE
PHARMACOLOGY
IMMUNOLOGY
ONCOLOGY
ARTWORK
ILLUSTRATION
MOLECULAR MODEL
ABNORMAL
ALPHA HELIX
BALLS
BETA SHEET
BIOCHEMICAL
BIOLOGICAL
BREAST CANCER
CANCER DRUG
CANCER TREATMENT
CANCEROUS
CHEMICAL
CHEMOTHERAPY
COMPOUND
COMPOUNDS
CUT OUT
CUT-OUT
CUTOUT
DOTS
HELICES
HELIXES
HER2
HUMAN EPIDERMAL GROWTH FACTOR
MALIGNANCY
MALIGNANT
MEDICAL
MOLECULAR
MOLECULES
PHARMACOLOGICAL
PROTEINS
RECEPTOR 2
SCANNING ELECTRON MICROGRAPH
SECONDARY STRUCTURE
SEM
SHEETS
THERAPY
TREATMENT
TUMOUR
UNHEALTHY